Paeds Cases · adolescent-and-young-adult-medicine
Eating disorders: refeeding and multidisciplinary-care OSCE — risk stratification, refeeding-syndrome prevention and family engagement
Observed structured encounter testing a safe refeeding plan for a malnourished adolescent: risk stratification, micronutrient and electrolyte management, recognition of refeeding syndrome, and family engagement in family-based treatment.
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Target exams
Station objectives
- Classify refeeding risk and complete the safe-preparation checklist before the first feed. [1]
- Design a refeeding regimen with thiamine and a phosphate–magnesium–potassium monitoring plan, and justify the calorie-start choice from the evidence. [3]
- Recognise and immediately manage evolving refeeding syndrome. [1]
- Engage anxious parents in family-based treatment with a non-blaming, structured frame. [5]
Candidate brief
You are the paediatric doctor on the adolescent ward. You have 10 minutes for Station A (preparing to refeed a newly admitted patient and engaging her parents) and 8 minutes for Station B (day-two deterioration with a falling phosphate). Examiners score safety, evidence, communication and partnership language. [1] [6]
Station A — Preparing to refeed and engaging the family
Setup: A 14-year-old with restricting-type anorexia nervosa, 71% median BMI, minimal intake for a week, 6 kg lost in two months. Baseline phosphate 0.80 mmol/L, potassium 3.3 mmol/L, magnesium 0.70 mmol/L; heart rate 44 supine to 78 standing; QTc 470 ms. Her parents are frightened and worried they caused the illness. [1] [6]
Expected actions:
- Classify her as high refeeding risk and state the stacked risk factors. [1]
- Complete the safe-preparation checklist: baseline biochemistry done; give thiamine before or with the first feed; correct the baseline phosphate, potassium and magnesium deficits; document the prolonged QTc and plan to repeat the ECG; set a calorie start matched to her risk. [1] [2]
- Set up serial monitoring: phosphate, potassium and magnesium daily (consider twice-daily in the first 48–72 hours), daily weight and orthostatic vitals. [3]
- Brief the nursing team on supervised meals, post-meal observation, exercise restriction and bathroom monitoring. [6]
- Explain to the parents, without blame, that they did not cause the illness and that family-based treatment makes them central to recovery: in phase 1 they take full, supported charge of her nutrition. Reassure them that phase-1 resistance is expected, not failure. [5]
Station B — Day-two falling phosphate
Setup: On day two of refeeding her phosphate has fallen from 0.80 to 0.45 mmol/L; potassium 3.1 mmol/L. She reports mild palpitations. [1]
Expected actions:
- Recognise evolving refeeding syndrome: phosphate is the sentinel marker and it is falling; the palpitations raise arrhythmia concern. [1] [2]
- Slow or briefly pause the feed; give intravenous phosphate per protocol; correct potassium and magnesium (magnesium must be adequate for potassium to correct); confirm thiamine has been given. [1]
- Monitor cardiac rhythm, repeat the ECG and QTc; escalate to high-dependency if there is haemodynamic instability, respiratory compromise or dangerous arrhythmia. [1]
- Resume feeding at a rate the biochemistry tolerates once corrected; do not abandon nutritional rehabilitation. [3]
Marking anchors
Clear pass: correctly classifies high refeeding risk; completes thiamine, baseline-electrolyte correction and matched calorie start; sets up daily biochemistry; recognises the falling phosphate as refeeding syndrome and acts immediately with IV phosphate and feed modulation; engages parents without blame and frames FBT phase 1; names the evidence (STRONG) and a guideline. [1] [3] [5] Borderline: good rapport but vague on monitoring frequency, reactive rather than proactive phosphate, or uncertain on the calorie evidence. Fail: starts feeding without thiamine or baseline-electrolyte correction; misses the falling phosphate; over-resuscitates with fluids; blames the parents; discharges intent without a monitoring or FBT plan. [1] [6]
Debrief pearls
- The first sign of refeeding syndrome is on the blood form, not the monitor — act on the phosphate trend before bedside decompensation. [2]
- Higher-calorie refeeding is evidence-supported only when paired with intensive monitoring and proactive replacement — the safety precondition, not the calorie number, is the point. [3] [4]
- Parents are the treatment, not the cause: FBT makes the family central to recovery from day one. [5]
References
- [1]Mehanna HM; Moledina J; Travis J Refeeding syndrome: what it is, and how to prevent and treat it. BMJ, 2008.PMID 18583681
- [2]O'Connor G; Nicholls D Refeeding hypophosphatemia in adolescents with anorexia nervosa: a systematic review. Nutrition in Clinical Practice, 2013.PMID 23459608
- [3]Garber AK; Cheng J; Accurso EC; Adams SH Short-term Outcomes of the Study of Refeeding to Optimize Inpatient Gains for Patients With Anorexia Nervosa: A Multicenter Randomized Clinical Trial. JAMA Pediatrics, 2021.PMID 33074282
- [4]Golden NH; Cheng J; Kapphahn CJ; Buckelew SM Higher-Calorie Refeeding in Anorexia Nervosa: 1-Year Outcomes From a Randomized Controlled Trial. Pediatrics, 2021.PMID 33753542
- [5]Lock J; Le Grange D; Agras WS; Moye A Randomized clinical trial comparing family-based treatment with adolescent-focused individual therapy for adolescents with anorexia nervosa. Archives of General Psychiatry, 2010.PMID 20921118
- [6]Rosen DS; American Academy of Pediatrics Committee on Adolescence Identification and management of eating disorders in children and adolescents. Pediatrics, 2010.PMID 21115584