Paeds Vivas · respiratory-sleep-and-airway
Behavioural insomnia and circadian rhythm disorders — branching viva
Branching viva from the classification of behavioural insomnia of childhood and the circadian rhythm disorders, through the two-process and melatonin physiology, the sleep history and diary, behavioural treatment of the toddler, the delayed-phase adolescent with school refusal, and the child with autism and chronic insomnia.
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Target exams
Station opening
Examiner: "Define behavioural insomnia of childhood and distinguish it from a circadian rhythm disorder." [1]
Strong candidate (must-hit)
- Defines behavioural insomnia of childhood as difficulty initiating or maintaining sleep that is learned and behavioural, present despite adequate sleep opportunity, with daytime consequences; names the sleep-onset association and limit-setting subtypes; and contrasts this with a circadian rhythm disorder, in which sleep quality is normal but the clock is set to the wrong time, delayed sleep-wake phase disorder being commonest in adolescents. [1] [8]
Weak candidate
- "It is just a child who won't sleep; I would give melatonin or a sedative to fix it." [2]
Branch A — The 15-month-old who wakes repeatedly
Examiner: "A 15-month-old wakes three or four times a night and can only be resettled by rocking. How do you treat her?" [1] [2]
Strong
- Recognises sleep-onset association type behavioural insomnia; explains that a learned cue (rocking) is required to bridge normal night arousals; treats behaviourally first-line with a consistent bedtime routine, graduated extinction (controlled comforting) and bedtime fading to teach independent settling; and warns parents that a brief increase in protest precedes improvement and that consistency is key. [1] [2]
Weak
- "Prescribe melatonin at bedtime and review in a few months." [6]
Branch B — The 15-year-old who cannot wake for school
Examiner: "A 15-year-old cannot sleep before 2am and cannot wake for school, but sleeps well and late at weekends. What is going on and what do you do?" [8] [9]
Strong
- Diagnoses delayed sleep-wake phase disorder from the stable late sleep onset, difficulty waking, and normal refreshing sleep on the delayed weekend schedule; explains the biological adolescent phase delay (the perfect storm); actively screens for depression and suicidal ideation; and treats with a fixed daily wake time, reduced evening light and screens, timed morning bright light, and low-dose evening melatonin timed hours before habitual sleep onset as a chronobiotic, with school liaison. [8] [9]
Weak
- "Tell him to stop being lazy and go to bed earlier." [9]
Branch C — The 6-year-old with autism and chronic insomnia
Examiner: "A 6-year-old with autism has chronic difficulty settling despite a good routine. What is your approach and where does melatonin fit?" [7] [6]
Strong
- Keeps behavioural strategies first-line but adapts them to the child's communication and sensory needs; treats coexisting anxiety and reviews the effect of any stimulant; and adds melatonin, noting its strongest evidence is in children with neurodevelopmental disorders, citing Gringras and colleagues' randomised trial showing improved sleep onset, and Bruni's recommendations for its use. [7] [6]
Weak
- "Autistic children just don't sleep; there is nothing to be done." [7]
Branch D — The remote family with limited sleep services
Examiner: "A family from a remote community cannot easily access a sleep clinic or actigraphy. How do you manage their child's sleep problem?" [8] [6]
Strong
- Relies on a structured sleep history and a one to two week sleep diary to make the diagnosis without laboratory tests; provides a clear written behavioural plan and parent education; uses melatonin as an adjunct where indicated; follows up by telehealth; and gives realistic, culturally appropriate advice that fits the family's housing and circumstances, acknowledging equity of access as an issue. [8] [6]
Weak
- "Do nothing until they can travel to a sleep laboratory." [8]
Close
Examiner: "Summarise your approach to the child who will not sleep in one sentence." [1] [8]
Strong
- "I take a sleep history and a sleep diary, confirm adequate sleep opportunity and exclude organic disorders, treat behavioural insomnia with behavioural techniques first-line, treat delayed sleep-wake phase disorder with a fixed wake time, morning bright light and correctly timed low-dose evening melatonin, and use melatonin as an adjunct — with its strongest evidence in children with neurodevelopmental disorders — rather than reaching first for a sedative." [1] [8]
References
- [1]Mindell JA; Kuhn B; Lewin DS; Meltzer LJ; Sadeh A Behavioral treatment of bedtime problems and night wakings in infants and young children. Sleep, 2006.PMID 17068979
- [2]Morgenthaler TI; Owens J; Alessi C; Boehlecke B; Brown TM; Coleman J; et al Practice parameters for behavioral treatment of bedtime problems and night wakings in infants and young children. Sleep, 2006.PMID 17068980
- [6]Bruni O; Alonso-Alconada D; Besag F; Biran V; Braam W; Cortese S; et al Current role of melatonin in pediatric neurology: clinical recommendations. Eur J Paediatr Neurol, 2015.PMID 25553845
- [7]Gringras P; Gamble C; Jones AP; Wiggs L; Williamson PR; Sutcliffe A; et al Melatonin for sleep problems in children with neurodevelopmental disorders: randomised double masked placebo controlled trial. BMJ, 2012.PMID 23129488
- [8]Auger RR; Burgess HJ; Emens JS; Deriy LV; Thomas SM; Sharkey KM Clinical Practice Guideline for the Treatment of Intrinsic Circadian Rhythm Sleep-Wake Disorders: Advanced Sleep-Wake Phase Disorder (ASWPD), Delayed Sleep-Wake Phase Disorder (DSWPD), Non-24-Hour Sleep-Wake Rhythm Disorder (N24SWD), and Irregular Sleep-Wake Rhythm Disorder (ISWRD). An Update for 2015: An American Academy of Sleep Medicine Clinical Practice Guideline. J Clin Sleep Med, 2015.PMID 26414986
- [9]Crowley SJ; Wolfson AR; Tarokh L; Carskadon MA An update on adolescent sleep: New evidence informing the perfect storm model. J Adolesc, 2018.PMID 29908393