Psych CASC / OSCE · Addiction psychiatry
Explaining Wernicke, Korsakoff, and long-term supports — CASC communication station
MRCPsych/FRANZCP-style communication station: explain WE vs KS vs ARBI, thiamine rationale and doses in plain language, imaging limits, prognosis with abstinence, capacity-sensitive disposition, and ARBD supports without stigma.
On this page & tools
Target exams
Station brief
Format. Communication station, approximately 7–10 minutes after reading time. You are the psychiatry registrar. [3][4]
Candidate instructions. Explain in plain language: what Wernicke encephalopathy is and why high-dose vitamin B1 drips were given; what Korsakoff/ARBI mean and how this differs from typical late-life Alzheimer disease; role of MRI; realistic recovery with abstinence and supports; capacity and why major decisions may need delay/support; involve sister respectfully; agree next steps. Check understanding. [1][2][5]
Candidate scenario
Sister: “Is this the same dementia our mother had? Why so many drips if it’s just alcohol? Can he sign a new lease tomorrow?” Patient smiles and says he is “fine to go home to my own flat” (he has none). [3][5]
Marking domains
- Empathy; non-stigmatising language about alcohol-related brain injury
- Accurate plain-language WE (thiamine/B1 emergency; treat early)
- Explains high-dose injections/drips vs tablets for acute risk
- Distinguishes KS/ARBI memory problems from inevitable Alzheimer narrative
- Honest prognosis: some recovery over months with abstinence; amnesia may persist
- Capacity: memory gaps mean big legal/housing decisions need assessment and support
- Shared plan: addiction care, rehab strategies, community/ARBD supports, follow-up
- Teach-back
Reveal assessor key
Open. Introduce role; acknowledge fear and anger; thank sister for advocacy. [5]
Wernicke. Heavy drinking plus poor nutrition can empty the body’s stores of vitamin B1 (thiamine). The brain’s energy systems fail in specific areas — people can become confused, develop eye-movement problems, and become unsteady. That is a medical emergency called Wernicke’s encephalopathy. We do not wait for every classic sign. High-dose B1 into the vein for several days protects and treats the brain better than ordinary tablets in this acute phase.[1][2]
Korsakoff / ARBI. After that injury, some people are left with severe difficulty laying down new memories (Korsakoff). They may fill gaps with confident wrong stories — not lying on purpose. Broader alcohol-related brain injury can also affect planning and judgement. This pathway is different from typical late-life Alzheimer disease, though mixed problems can coexist in older people.[3][4]
Scans. MRI can support the diagnosis but treatment starts on clinical grounds; a normal scan does not always rule out early Wernicke.[2]
Hope and honesty. Stopping alcohol and good nutrition give the best chance of improvement over months; memory problems may still need long-term strategies (diaries, routines, supported housing if needed).[5][6]
Lease tomorrow. Because he cannot reliably remember new information, he may not currently have capacity for that decision — we will assess carefully and involve appropriate supports rather than rush a binding agreement.[3][5]
Close. Teach-back; written info; addiction and ARBD follow-up; crisis contacts; sister involved with consent. [5]
References
- [1]Caine D, Halliday GM, Kril JJ, et al. Operational criteria for the classification of chronic alcoholics: identification of Wernicke's encephalopathy. J Neurol Neurosurg Psychiatry, 1997.PMID 9010400
- [2]Galvin R, Bråthen G, Ivashynka A, et al. EFNS guidelines for diagnosis, therapy and prevention of Wernicke encephalopathy. Eur J Neurol, 2010.PMID 20642790
- [3]Kopelman MD, Thomson AD, Guerrini I, et al. The Korsakoff syndrome: clinical aspects, psychology and treatment. Alcohol Alcohol, 2009.PMID 19151162
- [4]Zahr NM, Kaufman KL, Harper CG Clinical and pathological features of alcohol-related brain damage. Nat Rev Neurol, 2011.PMID 21487421
- [5]Wilson K, Halsey A, Macpherson H, et al. The psycho-social rehabilitation of patients with alcohol-related brain damage in the community. Alcohol Alcohol, 2012.PMID 22278316
- [6]Svanberg J, Evans JJ Neuropsychological rehabilitation in alcohol-related brain damage: a systematic review. Alcohol Alcohol, 2013.PMID 23955833