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Clinical Atlas Prestige · Evidence-first

Psych CASC / OSCEAddiction psychiatry

Psych CASC / OSCE · Addiction psychiatry

Explaining Wernicke, Korsakoff, and long-term supports — CASC communication station

MRCPsych/FRANZCP-style communication station: explain WE vs KS vs ARBI, thiamine rationale and doses in plain language, imaging limits, prognosis with abstinence, capacity-sensitive disposition, and ARBD supports without stigma.

communication
On this page & tools

Target exams

FRANZCPMRCPsychABPNMD-DNB

Target exams

FRANZCPMRCPsychABPNMD-DNB
Prompt
A 52-year-old man is day five after admission for confusion and heavy alcohol use. He received high-dose IV thiamine and is more alert, but still cannot retain new information and confabulates about having discharged home yesterday. His sister is angry: 'They said he has dementia like Mum — is that true? Why the huge vitamin drips? Will he ever live alone again?' She wants plain language about Wernicke, Korsakoff, MRI, and what supports he needs.

Station brief

Format. Communication station, approximately 7–10 minutes after reading time. You are the psychiatry registrar. [3][4]

Candidate instructions. Explain in plain language: what Wernicke encephalopathy is and why high-dose vitamin B1 drips were given; what Korsakoff/ARBI mean and how this differs from typical late-life Alzheimer disease; role of MRI; realistic recovery with abstinence and supports; capacity and why major decisions may need delay/support; involve sister respectfully; agree next steps. Check understanding. [1][2][5]

Candidate scenario

Sister: “Is this the same dementia our mother had? Why so many drips if it’s just alcohol? Can he sign a new lease tomorrow?” Patient smiles and says he is “fine to go home to my own flat” (he has none). [3][5]

Marking domains

  • Empathy; non-stigmatising language about alcohol-related brain injury
  • Accurate plain-language WE (thiamine/B1 emergency; treat early)
  • Explains high-dose injections/drips vs tablets for acute risk
  • Distinguishes KS/ARBI memory problems from inevitable Alzheimer narrative
  • Honest prognosis: some recovery over months with abstinence; amnesia may persist
  • Capacity: memory gaps mean big legal/housing decisions need assessment and support
  • Shared plan: addiction care, rehab strategies, community/ARBD supports, follow-up
  • Teach-back
[2] [3] [4] [5] [6]
Reveal assessor key

Open. Introduce role; acknowledge fear and anger; thank sister for advocacy. [5]

Wernicke. Heavy drinking plus poor nutrition can empty the body’s stores of vitamin B1 (thiamine). The brain’s energy systems fail in specific areas — people can become confused, develop eye-movement problems, and become unsteady. That is a medical emergency called Wernicke’s encephalopathy. We do not wait for every classic sign. High-dose B1 into the vein for several days protects and treats the brain better than ordinary tablets in this acute phase.[1][2]

Korsakoff / ARBI. After that injury, some people are left with severe difficulty laying down new memories (Korsakoff). They may fill gaps with confident wrong stories — not lying on purpose. Broader alcohol-related brain injury can also affect planning and judgement. This pathway is different from typical late-life Alzheimer disease, though mixed problems can coexist in older people.[3][4]

Scans. MRI can support the diagnosis but treatment starts on clinical grounds; a normal scan does not always rule out early Wernicke.[2]

Hope and honesty. Stopping alcohol and good nutrition give the best chance of improvement over months; memory problems may still need long-term strategies (diaries, routines, supported housing if needed).[5][6]

Lease tomorrow. Because he cannot reliably remember new information, he may not currently have capacity for that decision — we will assess carefully and involve appropriate supports rather than rush a binding agreement.[3][5]

Close. Teach-back; written info; addiction and ARBD follow-up; crisis contacts; sister involved with consent. [5]

References

  1. [1]Caine D, Halliday GM, Kril JJ, et al. Operational criteria for the classification of chronic alcoholics: identification of Wernicke's encephalopathy. J Neurol Neurosurg Psychiatry, 1997.PMID 9010400
  2. [2]Galvin R, Bråthen G, Ivashynka A, et al. EFNS guidelines for diagnosis, therapy and prevention of Wernicke encephalopathy. Eur J Neurol, 2010.PMID 20642790
  3. [3]Kopelman MD, Thomson AD, Guerrini I, et al. The Korsakoff syndrome: clinical aspects, psychology and treatment. Alcohol Alcohol, 2009.PMID 19151162
  4. [4]Zahr NM, Kaufman KL, Harper CG Clinical and pathological features of alcohol-related brain damage. Nat Rev Neurol, 2011.PMID 21487421
  5. [5]Wilson K, Halsey A, Macpherson H, et al. The psycho-social rehabilitation of patients with alcohol-related brain damage in the community. Alcohol Alcohol, 2012.PMID 22278316
  6. [6]Svanberg J, Evans JJ Neuropsychological rehabilitation in alcohol-related brain damage: a systematic review. Alcohol Alcohol, 2013.PMID 23955833