Psych CASC / OSCE · Addiction psychiatry
Explaining alcohol withdrawal, seizures, DT, and thiamine — CASC communication station
MRCPsych/FRANZCP-style communication station: explain AWS timeline, seizure and DT risk, CIWA-guided benzodiazepine detox as short-term medical treatment, parenteral thiamine/Wernicke prevention, inpatient criteria next time, non-stigmatising recovery framing.
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Target exams
Station brief
Format. Communication station, approximately 7–10 minutes after reading time. You are the psychiatry registrar. [6]
Candidate instructions. Acknowledge anger and fear; explain in plain language why seizures and delirium can occur after stopping heavy drinking; explain short-term benzodiazepines guided by symptom scores (CIWA); explain thiamine injections for brain protection; discuss why future detox should be supervised inpatient given complicated withdrawal; offer hope and follow-up without stigma. Check understanding. [1][2][5]
Candidate scenario
Partner: “If alcohol damages him, why give more drugs?” Patient: “I want to stop at home next week with just willpower.” Observations now stable; CIWA low. [3][5]
Marking domains
- Empathy, de-escalation, non-stigmatising language
- Accurate timeline: early symptoms, seizure window, DT window
- Benzodiazepines as short-term medical detox, not lifelong “replacement addiction” when protocolised
- Thiamine / Wernicke prevention in plain language
- Why prior seizure/DT makes unsupervised home detox unsafe
- Shared plan: addiction follow-up, crisis contacts, teach-back
Reveal assessor key
Open. Introduce role; validate that a fit and confusion are terrifying; apologise for communication gaps without defensiveness. [6]
Explain withdrawal. After long heavy use the brain adapts; stopping suddenly can cause shaking, sweating, high pulse, fits (often within about one to two days), and sometimes severe confusion with hallucinations (delirium tremens, often around day two to three). That is a medical emergency, not “going mad forever.”[1]
Benzodiazepines. Short-term medicines from the same calming-system family as alcohol’s effect, given in hospital and adjusted to symptoms (CIWA-type scores) so he is safe — not meant as a permanent substitute when used in a detox protocol.[2][3]
Thiamine. Heavy drinking and poor nutrition can empty vitamin B1 stores. Low B1 risks Wernicke brain injury (eye movement problems, unsteadiness, confusion). Injections deliver high doses quickly; tablets alone are not enough when risk is high.[4]
Next time. Because he already had a withdrawal seizure (and severe features), stopping alone at home is unsafe — he needs a planned inpatient or highly supervised detox. After stabilisation, offer counselling and optional recovery medicines for alcohol use disorder.[5][6]
Close. Teach-back; written plan; addiction service follow-up; when to return (confusion, fits, severe shaking, chest pain). [5]
References
- [1]Schuckit MA Recognition and management of withdrawal delirium (delirium tremens). N Engl J Med, 2014.PMID 25427113
- [2]Mayo-Smith MF Pharmacological management of alcohol withdrawal. A meta-analysis and evidence-based practice guideline. JAMA, 1997.PMID 9214531
- [3]Sullivan JT, Sykora K, Schneiderman J, et al. Assessment of alcohol withdrawal: the revised clinical institute withdrawal assessment for alcohol scale (CIWA-Ar). Br J Addict, 1989.PMID 2597811
- [4]Galvin R, Bråthen G, Ivashynka A, et al. EFNS guidelines for diagnosis, therapy and prevention of Wernicke encephalopathy. Eur J Neurol, 2010.PMID 20642790
- [5]ASAM The ASAM Clinical Practice Guideline on Alcohol Withdrawal Management. J Addict Med, 2020.PMID 32511109
- [6]Connor JP, Haber PS, Hall WD Alcohol use disorders. Lancet, 2016.PMID 26343838