Psych CASC / OSCE · Addiction psychiatry — substance use disorders
Explain opioid agonist treatment and naloxone — CASC communication station
MRCPsych/FRANZCP-style communication station: explain OUD as a medical disorder, OAT rationale and induction, precipitated withdrawal, take-home naloxone, and negotiate against unsafe ultra-rapid detox pressure.
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Target exams
Station brief
Format. Communication station, approximately 7–10 minutes active time after reading. You are the psychiatry/addiction registrar. The examiner may play the patient and/or mother. [3]
Candidate instructions. Explain opioid use disorder and why medication treatment is recommended after overdose. Compare methadone and buprenorphine in plain language. Explain how buprenorphine is started (waiting for withdrawal), side-effects, take-home naloxone, and why a 5-day forced detox is risky. Check understanding and agree a collaborative plan. [1][3]
Candidate scenario
He meets criteria for severe OUD, survived an overdose yesterday, and is now in moderate withdrawal. You propose buprenorphine–naloxone induction today with clinic follow-up, plus take-home naloxone. Mother insists medication is “another drug.” [4][3]
Marking domains
- Empathy, structure, non-stigmatising language
- Accurate plain-language model of OUD and OAT
- Explains precipitated withdrawal timing simply
- Methadone vs buprenorphine differences at lay level
- Naloxone education and mortality/retention rationale
- Negotiates with family without colluding in unsafe detox
- Checks understanding / teach-back [2][3]
Reveal assessor key
Open. Name time; ask patient and mother top concerns (control, stigma, “addiction to methadone”). [3]
Explain OUD. “This is a medical brain-and-behaviour condition where opioids take priority despite harm — not a simple willpower gap. After overdose, the risk of dying remains high if we only send him home.” [1]
Explain OAT. “Medications like buprenorphine or methadone stabilise receptors, cut craving and illicit use, and people on treatment are less likely to die than people who stop treatment.” Use lay mortality framing from retention evidence without drowning in statistics.[2]
Buprenorphine plan. “We start when he is already withdrawing so the medicine does not knock other opioids off the receptor too fast — that sudden jump is precipitated withdrawal. Typical start is a small under-the-tongue dose (e.g. 2–4 mg), then build up, aiming for a daily dose that stops withdrawal and craving (often 8–24 mg).” Link to follow-up clinic.[3][4]
Methadone contrast. Full agonist, daily supervised dosing early, very effective, needs careful slow start because it accumulates; cardiac monitoring in some patients. Choice can be revisited. [3]
Naloxone. Train both on recognising slow breathing and using take-home naloxone; call emergency services. [3]
Five-day detox. Acknowledge wish for quick fix; explain high relapse and overdose risk when tolerance falls. Offer supports for the relationship crisis without abandoning MOUD. [2]
Close. Summarise plan, written info, crisis contacts, early review, teach-back. [3]
References
- [1]Volkow ND, Frieden TR, Hyde PS, Cha SS Medication-assisted therapies--tackling the opioid-overdose epidemic N Engl J Med, 2014.PMID 24758595
- [2]Sordo L, Barrio G, Bravo MJ, et al. Mortality risk during and after opioid substitution treatment: systematic review and meta-analysis of cohort studies BMJ, 2017.PMID 28446428
- [3]American Society of Addiction Medicine The ASAM National Practice Guideline for the Treatment of Opioid Use Disorder: 2020 Focused Update J Addict Med, 2020.PMID 32511106
- [4]D'Onofrio G, O'Connor PG, Pantalon MV, et al. Emergency department-initiated buprenorphine/naloxone treatment for opioid dependence: a randomized clinical trial JAMA, 2015.PMID 25919527