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Clinical Atlas Prestige · Evidence-first

Psych VivasIntellectual disability — neurodevelopmental

Psych Vivas · Intellectual disability — neurodevelopmental

Fragile X syndrome — structured clinical viva

Fellowship viva on FMR1 allele classes, Sherman paradox, full-mutation phenotype, FXTAS/FXPOI, molecular diagnosis, comorbidity care and cascade counselling.

clinical
On this page & tools

Target exams

FRANZCPMRCPsychABPNMD-DNB

Target exams

FRANZCPMRCPsychABPNMD-DNB
Prompt
You are the dual-diagnosis psychiatry registrar. Parents of a boy with probable fragile X syndrome ask: (1) What is fragile X and how is it inherited? (2) What do the CGG numbers mean? (3) Why does mum's early menopause matter? (4) Is there a medicine that cures the gene problem? (5) What psychiatric problems should we watch for? (6) Who else in the family needs testing? Discuss genetics, phenotype, premutation risks, management and counselling.

Interpretation

Reveal interpretation

What is FXS / inheritance? Most common inherited monogenic ID. X-linked FMR1 CGG expansion. Full mutation typically greater than 200 repeats with methylation and FMRP loss. Boys usually affected; girls variable. Full mutations almost always come via mother.[1][2]

CGG numbers. Normal ~5–44; intermediate ~45–54; premutation ~55–200; full mutation greater than 200. Premutation ≠ mild FXS. Sherman paradox: expanding alleles across generations, especially maternal transmission.[2][3]

Mother's early menopause. Suggests possible premutation with FXPOI risk — established association with premature ovarian failure/insufficiency. Needs genetic counselling and reproductive discussion, not dismissal as coincidence.[6][2]

Cure medicine? No approved routine disease-modifying core cure restoring FMRP. Supports + treat anxiety/ADHD/seizures/behaviour risk. Experimental mGluR-targeted ideas exist as theory, not standard cure claims.[1][2]

Psychiatric watch-list. Anxiety (very high), ADHD symptoms, ASD features, sensory hyperarousal, sleep, mood; seizures in a minority. Adapt MSE for sensory load.[4][1]

Who else to test? Cascade testing/counselling for mother and other at-risk maternal-line relatives; adult premutation carriers need awareness of FXTAS with ageing (tremor/ataxia/executive decline) and FXPOI in women.[2][5][6]

Name evidence. Hagerman 2017 primer; GeneReviews FMR1 disorders; Fu 1991 Sherman; Cordeiro 2011 anxiety; Jacquemont 2004 FXTAS penetrance; Allingham-Hawkins 1999 POF.[1][2][3][4][5][6]

Key points

Full mutation vs premutation

FMRP loss versus RNA toxicity — different counselling and different adult risks.

No core cure tablet

Scaffold development; treat comorbidities; monitor medicines.

Cascade is mandatory

Every index diagnosis is a family diagnosis (FXPOI/FXTAS/expansion risk).
[1] [2] [6]

References

  1. [1]Hagerman RJ, Berry-Kravis E, Hazlett HC, et al. Fragile X syndrome Nat Rev Dis Primers, 2017.PMID 28960184
  2. [2]Hunter JE, Berry-Kravis E, Hipp H, Todd PK FMR1 Disorders GeneReviews, 1993.PMID 20301558
  3. [3]Fu YH, Kuhl DP, Pizzuti A, et al. Variation of the CGG repeat at the fragile X site results in genetic instability: resolution of the Sherman paradox Cell, 1991.PMID 1760838
  4. [4]Cordeiro L, Ballinger E, Hagerman R, Hessl D Clinical assessment of DSM-IV anxiety disorders in fragile X syndrome: prevalence and characterization J Neurodev Disord, 2011.PMID 21475730
  5. [5]Jacquemont S, Hagerman RJ, Leehey MA, et al. Penetrance of the fragile X-associated tremor/ataxia syndrome in a premutation carrier population JAMA, 2004.PMID 14747503
  6. [6]Allingham-Hawkins DJ, Babul-Hirji R, Chitayat D, et al. Fragile X premutation is a significant risk factor for premature ovarian failure: the International Collaborative POF in Fragile X study Am J Med Genet, 1999.PMID 10208170