Achilles Tendon Rupture
Summary
Rupture of the Achilles tendon (Tendo-Achilles) is the most common major tendon rupture in the lower limb. It typically occurs in "Weekend Warriors" (middle-aged men playing explosive sports) with a sensation of being "kicked in the back of the leg". The rupture usually occurs in the Watershed Zone (2-6cm proximal to insertion) where blood supply is poorest. Diagnosis is purely clinical using the Simmonds-Thompson Squeeze Test. Management has shifted towards Functional Rehabilitation (early weight-bearing in an equinus brace) which yields results comparable to surgery without the wound complications, though surgery is still preferred for high-level athletes to minimize re-rupture risk. [1,2]
Key Facts
- Mechanism: Explosive push-off (Tennis, Squash).
- Sensation: "Bang" / "I was kicked".
- Site: 2-6cm proximal to calcaneal insertion (Watershed).
- Key Sign: Simmonds-Thompson Test (Squeeze calf -> Foot does NOT plantarflex).
- Common Pitfall: Missed diagnosis (20%). The patient can still plantarflex using secondary muscles (FHL, FDL, Tib Post), tricking the examiner.
- Management: Functional Bracing (Vacoped) vs Surgery.
- Re-rupture Rate: Conservative 8-12%, Surgical 2-4%.
Clinical Pearls
"The Gap is Real": Within the first 24 hours, you can often feel a palpable gap (delle) in the tendon. Mark it with a pen. Once swelling sets in (edema/haematoma), the gap disappears.
"Don't Trust the Toe Raise": A patient with an Achilles rupture can sometimes perform a toe raise (weakly) using their accessory flexors (Tibialis Posterior, Peroneals, Flexor Hallucis Longus). Do not rely on active movement. Trust the Squeeze Test.
"The Fluoroquinolone Factor": Ask about antibiotics. Ciprofloxacin can cause spontaneous tendon rupture, often bilaterally. Steroids are also a multiplier.
"Treat the Soft Tissues": The skin over the Achilles is thin and precarious. Whether surgical or conservative, handle the skin like gold dust. A necrotic wound here is a plastic surgery disaster involving free flaps.
Demographics
- Incidence: 18 per 100,000. Rising due to active elderly population.
- Age: Peak 30-50 years.
- Gender: Male > Female (10:1). The classical "Weekend Warrior".
- Side: Left > Right (Left leg is the "pusher" leg for right-handed people).
Risk Factors
- Intrinsic: Previous tendinopathy (degeneration), Age, Male gender.
- Medical: Systemic Steroids, Renal Failure, Gout, Rheumatoid Arthritis.
- Medication: Fluoroquinolones (Ciprofloxacin/Levofloxacin) - Black box warning.
- Activity: Stop-start sports (Squash, Badminton, Tennis).
Anatomy
- Triceps Surae: Gastrocnemius (fast twitch, crosses knee + ankle) + Soleus (slow twitch, ankle only).
- Tendon: The strongest/thickest in the body. It spirals 90 degrees as it inserts into the calcaneus.
- Blood Supply:
- Proximally: From muscle belly.
- Distally: From bone insertion.
- Watershed Zone: 2-6cm proximal to insertion. This area relies on diffusion from the paratenon and is prone to hypovascularity, degeneration and rupture.
- Paratenon: Unlike other tendons, the Achilles lacks a true synovial sheath. It has a vascular paratenon.
Mechanism
- Eccentric Loading: The muscle is maximally contracting while being forcibly lengthened (e.g., landing from a jump or pushing off to sprint).
- Failure: The load exceeds the tensile strength of the (often degenerated) tendon fibers.
- Histology: Usually shows prior Myxoid Degeneration (Angiofibroblastic dysplasia). A healthy tendon rarely snaps; a degenerated one does.
Symptoms
Physical Examination
1. Ultrasound (Dynamic) - The Workhorse
- Role: Confirms full thickness rupture. Diagnoses "missed" ruptures.
- Dynamic Gap Evaluation: This determines treatment.
- The sonographer plantarflexes the foot (full equinus).
- Ends Appose: If the tendon ends touch, Conservative Management is highly likely to succeed.
- Gap remains: If >5-10mm gap persists in full equinus, surgery may be indicated to bridge the defect.
2. MRI
- Role: Usually unnecessary for acute rupture.
- Indications:
- Chronic/Missed ruptures (to assess gap size and muscle atrophy/fatty infiltration).
- Diagnostic uncertainty (partial tears).
- Pre-operative planning for reconstruction (V-Y plasty).
3. X-Ray
- Role: Only to exclude avulsion fracture of the calcaneus (rare). Usually normal.
ACHILLES RUPTURE CONFIRMED
↓
ASSESS GAP ON ULTRASOUND (DYNAMIC)
DO ENDS MEET IN PLANTARFLEXION?
┌────────────┴────────────┐
YES NO (or >1cm Gap)
↓ ↓
FUNCTIONAL CONSIDER SURGERY
BRACING (Gap too big to heal)
(Standard) ↓
↓ PATIENT FACTORS?
(Vacoped Boot) (Athlete vs Sedentary)
↓ │
PROTOCOL ┌────┴────┐
(Full WB in Equinus) SURG CONSERVATIVE
↓
REHABILITATION
1. Functional Rehabilitation (Conservative) - "Accelerated Rehab"
- Concept: Bring the tendon ends together by pointing the foot down (Equinus) and holding it there. Use early weight bearing (proprioception) to stimulate collagen alignment.
- Device: Vacoped Boot (Gold standard - adjustable range) or Wedged Moon Boot.
- Protocol (Example):
- 0-2 weeks: Full Equinus (30 deg). Non-weight bearing (or heel touch).
- 2-4 weeks: Full Equinus. Weight bearing as tolerated.
- 4-8 weeks: Gradually remove wedges/reduce angle (5 deg per week).
- 8-10 weeks: Into shoes with heel lift.
- Success: Re-rupture rates are now similar to surgery (in specialized centers with strict protocols).
- Pros: No wound complications (0% infection).
2. Surgical Repair
- Indication:
- Gap >1cm in full equinus (ends don't meet).
- High-level athletes (need maximal push-off power).
- Delayed presentation (>2 weeks).
- Re-rupture.
- Technique:
- Open Repair: Large incision. Strong Krackow sutures. High wound risk.
- Percutaneous (Mini-Open): Jig systems (Achillon / PARS). Smaller scar, lower infection risk, higher Sural nerve injury risk.
- Pros: Lowest re-rupture rate (2-3%). Slightly better strength?
- Cons: Infection (Deep infection is disastrous). Sural nerve injury. DVT.
Non-Surgical
- Re-rupture: Risk 8-12% (Traditional) -> 4-6% (Functional Rehab).
- Elongation: Tendon heals "long". Result is weak push-off power. Patient walks with a limp.
- DVT/PE: High risk due to immobilisation. All patients need chemical thromboprophylaxis (LMWH/Rivaroxaban).
Surgical
- Wound Breakdown: The skin is thin. Necrosis exposes the tendon. Can require Free Flap reconstruction.
- Deep Infection: 1-2%. Can require excision of the entire tendon -> devastating.
- Sural Nerve Injury: Numbness on lateral foot. Painful neuroma. Common in percutaneous repair.
- Adhesions: Scarring of tendon to skin.
- Healing Time: 10-12 weeks for clinical union. 6 months for remodeling.
- Return to Sport:
- Jogging: 3-4 months.
- Sprinting/Sport: 6-9 months (often 9-12 for elite level).
- Performance: Most athletes lose ~10-15% of peak power permanently.
The Krackow Suture
- A "locking loop" stitch used for tendon repair.
- It grips the tendon fibers tightly and prevents pull-out.
- Usually 2-4 strands of heavy non-absorbable suture (Ethibond/Fiberwire/UltraBraid).
The Percutaneous Jig (Achillon / PARS)
- Instrument passed inside the paratenon.
- Needles passed blindly through skin/jig/tendon.
- Risk: Catching the Sural Nerve (which runs laterally).
- Prevention: Open the lateral side to see the nerve. Or ultrasound guidance.
Key Studies
- Willits et al. (2010): Landmark RCT. Surgery vs Functional Rehab. Result: NO significant difference in re-rupture rate if functional rehab used. Revolutionised non-operative care.
- Keating et al. (2011): Operative repair reduces re-rupture but increases complications significantly.
- Olsson et al. (2013): Early weight bearing improves healing.
Guidelines
- AAOS: Moderate evidence to support non-operative treatment in general population.
- BOA (British Orthopaedic Association): Conservative management is the standard of care for most patients, provided ends appose.
Did my tendon snap?
Yes. It has pulled apart like two ends of a mop.
Can it heal without surgery?
Amazingly, yes. The tendon ends are sticky. If we point your toes down in a special boot, the ends touch. Nature will glue them back together over 8-10 weeks. This avoids the risks of surgery (wound infection).
Why would anyone have surgery?
Surgery stitches them tighter. For Olympic athletes, that 1% extra power might matter. Or if the gap is too big for the ends to touch. But for most people, the boot gives a result just as good without the risk of a nasty infection.
Can I drive?
No. Not while in the boot (Right leg especially). Insurance is void. Usually 10-12 weeks.
- Willits K, et al. Operative versus nonoperative treatment of acute Achilles tendon ruptures: a multicenter randomized trial using accelerated functional rehabilitation. J Bone Joint Surg Am. 2010.
- Soroceanu A, et al. Surgical versus nonsurgical treatment of acute Achilles tendon rupture: a meta-analysis of randomized trials. J Bone Joint Surg Am. 2012.
- Maffulli N, et al. Types and epidemiology of tendinopathy. Clin Sports Med. 2003.
- Khan RJ, et al. Treatment of acute achilles tendon ruptures. a meta-analysis of randomized, controlled trials. J Bone Joint Surg Am. 2005.
Rivaroxaban (Xarelto)
- Class: Direct Factor Xa Inhibitor (DOAC).
- Indication: VTE Prophylaxis in lower limb immobilisation.
- Dose: 10mg OD.
- Duration: Usually until fully weight bearing (6-8 weeks).
- Contraindications: Active bleeding, severe renal impairment.
- Why?: The combination of trauma + immobilisation makes Achilles rupture a VERY high risk for DVT/PE.
Enoxaparin (Clexane)
- Class: Low Molecular Weight Heparin (LMWH).
- Indication: Alternative to Rivaroxaban.
- Dose: 40mg SC OD.
- Downside: Daily injections (patients hate it).
Case 1: The Sedentary Diabetic
Scenario: 50-year-old male, diabetic, smoker. Steps off curb, feels pop. Gap palpable. Management: Conservative (Functional Rehab). Reasoning: High risk of wound infection/healing problems with surgery. Diabetes + Smoking = Surgical Disaster waiting to happen.
Case 2: The Professional Sprinter
Scenario: 24-year-old athlete. Rupture confirmed. Management: Surgical Repair. Reasoning: Needs guaranteed maximal tensioning for power. Accepts wound risk for performance gain. Low re-rupture risk priority.
Case 3: The Missed Diagnosis
Scenario: 40-year-old male. Injury 6 weeks ago. "Calf strain". Now complains of weakness walking up stairs. Diagnosis: Chronic Achilles Rupture. Management: Surgical Reconstruction (V-Y Plasty or FHL Transfer). Primary repair impossible as ends have retracted and scarred.
Phase 1: 0-2 Weeks
- Vacoped Boot locked at 30 degrees plantarflexion.
- Touch weight bearing with crutches.
- Rivaroxaban 10mg OD.
- Elevation.
Phase 2: 2-4 Weeks
- boot locked at 30 degrees.
- Full weight bearing as tolerated.
- Active plantarflexion allowed (protected). NO dorsiflexion past neutral.
Phase 3: 4-8 Weeks
- Reduce boot angle by 5 degrees per week (or remove wedges).
- Aim for neutral (0 degrees) by week 8.
- Proprioception exercises.
Phase 4: 8+ Weeks
- Wean out of boot into shoes with heel lift.
- Heel raises (Double leg -> Single leg).
- Plyometrics start at 3-4 months.
- Achilles: Greek hero. Dipped in the River Styx by his mother Thetis, held by his heel. His only vulnerable spot. Killed by an arrow from Paris to the heel.
- Simmonds (1957): Described the squeeze test.
- Thompson (1962): Independent description of the same test. Hence "Simmonds-Thompson".
- Hunter (1700s): John Hunter (famous surgeon) ruptured his own Achilles while dancing. He treated it conservatively by binding it up. Proof that conservative care has a long history!
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