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Orthopaedics
Emergency Medicine

Achilles Tendon Rupture

High EvidenceUpdated: 2025-12-22

On This Page

Red Flags

  • Missed diagnosis (patient sent home with 'ankle sprain')
  • Skin compromise in chronic/neglected rupture
  • Re-rupture after treatment
  • Deep wound infection post-surgery
Overview

Achilles Tendon Rupture

1. Clinical Overview

Summary

Achilles tendon rupture is a common injury affecting the strongest tendon in the human body. It typically occurs in middle-aged "weekend warriors" during activities involving sudden push-off or landing (e.g., tennis, squash, basketball). The rupture usually occurs at the "watershed zone" 2-6cm above the calcaneal insertion where blood supply is poorest. Most ruptures occur on a background of chronic tendinopathy. Diagnosis is clinical using Simmonds-Thompson test. Modern evidence supports functional rehabilitation (non-operative) for most patients, with surgery reserved for elite athletes, delayed presentations, or re-ruptures.

Key Facts

  • Incidence: 18-37 per 100,000 per year (increasing)
  • Peak age: 30-50 years ("weekend warrior")
  • Sex ratio: Male:Female 10:1
  • Rupture site: 2-6cm above calcaneal insertion (watershed zone)
  • Missed diagnosis rate: 20-25% initially misdiagnosed as "ankle sprain"
  • Key test: Simmonds-Thompson (calf squeeze) test
  • Treatment trend: Functional rehabilitation now first-line for most patients

Clinical Pearls

"Someone Kicked Me": Patients classically describe feeling like they were kicked or struck in the back of the leg. This is pathognomonic history for Achilles rupture.

They CAN Still Walk: Do not be fooled! Patients can walk and even weakly plantarflex (using tibialis posterior and peronei). The Simmonds test is mandatory for all posterior ankle pain.

Equinus Position: Initial immobilization MUST be in full plantarflexion (equinus) to approximate tendon ends. Casting at 90 degrees leaves a gap and prevents healing.

Why This Matters Clinically

Achilles rupture is commonly missed in emergency departments. Delayed diagnosis leads to worse outcomes despite treatment. Understanding the clinical examination and avoiding the trap of preserved ambulation prevents missed injuries. The shift towards non-operative management has reduced surgical complications while maintaining comparable outcomes.


2. Epidemiology

Incidence & Prevalence

  • Incidence: 18-37 per 100,000 per year
  • Trend: Increasing (more participation in recreational sport)
  • Lifetime risk: Estimated 1-2%

Demographics

FactorDetails
Age30-50 years (peak); bimodal with smaller elderly peak
SexMale:Female 10:1
ActivityRecreational athletes ("weekend warriors")
LateralityLeft slightly more common (pushing leg in right-handed)

Risk Factors

Non-Modifiable:

  • Age 30-50 (tendon degeneration begins)
  • Male sex
  • Previous Achilles tendinopathy

Modifiable:

Risk FactorRelative Risk
Quinolone antibiotics2-6x (FDA black box warning)
Corticosteroid use2-3x (oral or injected)
Sudden increase in activityVariable
Poor conditioning/warm-upVariable

3. Pathophysiology

Mechanism

Step 1: The "Degenerative Prelude" (Tendinopathy)

  • Normal Tendon: Type I Collagen (Strong, linear). Few cells.
  • Pathology: Age + Overuse -> Microtrauma.
  • Healing attempt: Angiofibroblastic hyperplasia (Neo-vessels + Type III Collagen).
  • Weakness: Type III collagen is disorganized and weak. The "rope" is fraying from the inside.

Step 2: The "Watershed" Vulnerability

  • Location: 2-6cm proximal to calcaneal insertion.
  • Vascularity: This zone has the poorest blood supply (supplied by recurrent vessels from calcaneus and descending vessels from muscle), making it prone to hypoxia and poor repair.

Step 3: The Mechanical Failure

  • Trigger: Eccentric contraction (Lengthening while contracting).
    • Scenario: Pushing off to sprint (Gastroc contracts, Foot plantarflexed -> Dorsiflexed).
  • Force: Forces can exceed 10x body weight.
  • Failure: The weakened collagen snaps.

Step 4: The "Gap" and Retraction

  • Retraction: The Gastrocnemius-Soleus muscle belly pulls the proximal stump proximally.
  • Gap: A void fills with hematoma.
  • Consequence: If healed in a "long" position (gap not closed), the muscle-tendon unit is too long -> Loss of push-off power (End-stage failure).

Classification

TypeDescription
Acute ruptureLess than 2 weeks from injury; fresh ends
Chronic/neglected ruptureGreater than 4-6 weeks; retracted, scarred ends
Partial ruptureIncomplete tear; controversial entity
Re-ruptureRupture after previous treatment

Anatomical Considerations

  • Achilles tendon is the strongest tendon in the body (12-17cm long)
  • Formed by confluence of gastrocnemius and soleus tendons
  • Inserts on posterior calcaneal tuberosity
  • Sural nerve runs lateral to tendon (at risk in surgery)
  • Paratenon (not synovium) surrounds tendon; poor healing capacity

4. Clinical Presentation

Symptoms

Typical Presentation:

Important Caveat:

Signs

Red Flags

[!CAUTION] Red Flags — High suspicion if:

  • "Pop" + weakness in back of ankle (assume rupture until proven otherwise)
  • Posterior ankle pain unable to single-leg heel raise
  • Elderly patient on quinolones with sudden calf pain
  • Patient previously diagnosed with "ankle sprain" but not improving (missed rupture)

Sudden "pop" or "snap" felt/heard (80%)
Common presentation.
Feeling of being "kicked" or "struck" in back of leg (classic)
Common presentation.
Immediate sharp pain, often subsiding quickly
Common presentation.
Weakness pushing off
Common presentation.
Difficulty walking on toes
Common presentation.
5. Clinical Examination

Structured Approach

General:

  • Gait assessment (limping, difficulty pushing off)
  • Compare both sides

Inspection:

  • Swelling posterior ankle
  • Bruising (may be extensive)
  • Gap visible in tendon (if swelling permits)
  • Altered resting ankle position

Palpation:

  • Palpable defect/gap 2-6cm above insertion
  • Tenderness at rupture site

Special Tests

TestTechniquePositive FindingSensitivity/Specificity
Simmonds-Thompson (Squeeze) TestPatient prone, feet over edge; squeeze calfNo foot movement = POSITIVE (rupture)96% / 93%
Matles Test (Angle of Dangle)Patient prone, knees 90°; observe anklesAffected side more dorsiflexed88% / 85%
O'Brien Needle TestNeedle inserted proximal to suspected tear; observe movement with ankle motionNeedle moves with dorsiflexion if tendon intactUsed if examination equivocal
Single Leg Heel RaiseAsk patient to stand on one leg and rise onto toesUnable to perform = POSITIVEHigh sensitivity

6. Investigations

First-Line (Bedside)

  • Clinical examination — Simmonds test is diagnostic in most cases
  • No imaging required if clinical diagnosis clear

Laboratory Tests

TestExpected FindingPurpose
Not routinely indicated——

Imaging

ModalityFindingsIndication
UltrasoundGap visualization; tendon discontinuity; dynamic assessmentEquivocal clinical picture; assess gap size for surgical planning
MRIDetailed tendon morphology; partial vs completeChronic/neglected ruptures; pre-operative planning
X-rayAvulsion fracture at calcaneal insertion (rare)High-energy trauma

Diagnostic Criteria

  • Clinical diagnosis: Positive Simmonds-Thompson test + appropriate history is sufficient
  • Imaging is adjunctive, not essential for acute presentation with clear clinical findings

7. Management

Management Algorithm

           Sudden Calf Pain ("Snap")
                      ↓
┌───────────────────────────────────────────────┐
│              CLINICAL DIAGNOSIS               │
│  - Simmonds-Thompson Test (Squeeze Calf)      │
│  - Matles Test (Resting Dangle)               │
│  - Palpable Gap                               │
└───────────────────────────────────────────────┘
                      ↓
┌───────────────────────────────────────────────┐
│            IMMEDIATE ED MANAGEMENT            │
│  1. Analgesia                                 │
│  2. EQUINUS CAST/BOOT (Toes pointed down)     │
│  3. Non-Weight Bearing                        │
│  4. VTE Prophylaxis (Risk Assess)             │
└───────────────────────────────────────────────┘
                      ↓
┌───────────────────────────────────────────────┐
│            DEFINITIVE DECISION                │
│           (Ortho Clinic within 1w)            │
├───────────────────────┬───────────────────────┤
│    NON-OPERATIVE      │      OPERATIVE        │
│ (Standard of Care)    │     (Select Cases)    │
├───────────────────────┼───────────────────────┤
│ Indicates:            │ Indicates:            │
│ - Most patients       │ - Elite Athletes      │
│ - Gap under 1cm (US)  │ - Re-ruptures         │
│ - Closed injury       │ - Delayed >4w         │
│                       │ - Open Avulsion       │
└───────────┬───────────┴───────────┬───────────┘
            ↓                       ↓
   Functional Rehab            Surgical Repair
   (VacoCast Protocol)        (Open vs Minimally Invasive)

Acute/Emergency Management

Initial ED/Clinic Management:

  1. Analgesia (paracetamol ± NSAIDs if no contraindication)
  2. Immobilize in EQUINUS position (full plantarflexion)
  3. Backslab or VacoPed boot with maximal wedges
  4. Non-weight bearing with crutches
  5. Elevate limb
  6. Urgent orthopedic/fracture clinic referral (within 1 week)
  7. Consider VTE prophylaxis

Conservative Management: Functional Rehabilitation (e.g., VacoPed)

The modern gold standard involves early weight-bearing in a specialized boot.

PhaseTimeBoot Setting (Equinus)Weight BearingPhysio Goals
10-2w30° Plantarflexion (Max Equinus)Non-Weight Bearing (Crutches)Rest, Ice, Elevation. Core exercises.
22-4w30° PlantarflexionWeight Bear as ToleratedLoad the tendon (Micro-motion aids healing).
34-6w15-30° PlantarflexionFull Weight BearingActive inversion/eversion. Knee extension.
46-8w0-15° PlantarflexionFull Weight BearingFlat foot walking. Proprioception.
58-10wNeutral (0°)Full Weight BearingWean from boot. Double leg heel raise.
6>12wRegular ShoesFullSingle leg heel raise. Return to jog (16w). Sport (6-9m).

Why Functional Rehab?

  • Controlled "loading" stimulates collagen alignment (Wolff's Law).
  • Reduces DVT risk (calf pump active).
  • Reduces muscle atrophy compared to casting.

Surgical Management

Indications for Surgery:

  • Elite/professional athletes (earlier return to maximal power)
  • Delayed presentation (greater than 2-4 weeks)
  • Re-rupture after non-operative treatment
  • Large gap (greater than 10mm on ultrasound — relative indication)

Techniques:

  1. Open Repair: Posteromedial incision. Sural nerve protected. Tendon ends debrided and sutured (Krackow or Kessler suture technique with heavy non-absorbable suture). Paratenon closed separately (gliding layer).
  2. Percutaneous (Minimally Invasive): "Achillon" or similar jig. Suture passed blindly through skin. Pros: Small cosmetic scar. Cons: Higher nerve injury risk.

Complications of Surgery:

  • Infection: The skin over the Achilles is thin with poor vascularity. Necrosis can lead to tendon exposure -> Plastic Surgery Flaps. This is why diabetic/smokers are non-op.
  • Sural Nerve: Numbness 5-10%.

Surgical Risks:

  • Wound infection/breakdown (5-10%) — devastating in this area
  • Sural nerve injury (numbness lateral foot; 3-5%)
  • Re-rupture (2-5%)
  • DVT

Disposition

  • Outpatient management: Standard for acute ruptures
  • Admit if: Major trauma, polytrauma, unable to manage at home
  • Follow-up: Orthopedic clinic within 1-2 weeks for definitive plan

8. Complications

Immediate (Minutes-Hours)

ComplicationIncidencePresentationManagement
Severe painVariableAcute painAnalgesia; immobilization

Early (Days-Weeks)

  • DVT: High risk with immobilization; consider LMWH prophylaxis
  • Skin pressure injury: From casting/boot; ensure proper fitting
  • Muscle atrophy: Early physiotherapy when appropriate

Late (Months-Years)

  • Re-rupture: 2-5% with functional rehab; similar with surgery
  • Tendon lengthening: Heals "long" leading to reduced push-off power
  • Calf atrophy: Often permanent; smaller calf on affected side
  • Post-operative infection: Skin over Achilles is thin; poor blood supply
  • Sural nerve injury: After surgery; lateral foot numbness
  • Stiffness: Ankle dorsiflexion restriction

9. Prognosis & Outcomes

Natural History

  • Untreated complete rupture will not heal spontaneously with functional recovery
  • Chronic neglected ruptures develop muscle shortening and scarring

Outcomes with Treatment

VariableFunctional RehabSurgical Repair
Re-rupture rate2-5%2-3%
Wound complications0%5-10%
Return to previous activity80-90%80-90%
Time to return to sport6-9 months6-9 months
Push-off strength85-90% contralateral90-95% contralateral

Prognostic Factors

Good Prognosis:

  • Early diagnosis (less than 2 weeks)
  • Appropriate initial immobilization (equinus)
  • Compliance with rehabilitation
  • Younger age
  • No prior tendinopathy

Poor Prognosis:

  • Delayed diagnosis (greater than 4 weeks)
  • Re-rupture
  • Poor compliance
  • Continued quinolone/steroid use
  • Significant comorbidities

10. Evidence & Guidelines

Key Guidelines

  1. BOAST 13 (2019) — British Orthopaedic Association Standards for Trauma. Supports functional rehabilitation as first-line; emphasizes VTE prophylaxis. BOA BOAST
  2. AAOS Clinical Practice Guideline (2010) — American Academy of Orthopaedic Surgeons. Non-operative management acceptable for most patients.

Landmark Trials

Willits et al. (2010) — Surgery vs Non-Operative with Functional Rehabilitation

  • 144 patients randomised
  • Key finding: No significant difference in re-rupture rate (3.5% surgical vs 4.6% non-op)
  • Key finding: Higher complication rate in surgical group (16% vs 1%)
  • Clinical Impact: Shifted practice globally towards non-operative management

Lantto et al. (2016) — Long-term Outcomes

  • Prospective randomised trial
  • Key finding: At 18 months, no difference in functional outcomes
  • Clinical Impact: Confirmed durability of non-operative approach

Metz et al. (2008) — Minimally Invasive vs Open Surgery

  • Key finding: Lower wound complication rate with minimally invasive techniques
  • Clinical Impact: If surgery chosen, consider minimally invasive approach

Evidence Strength

InterventionLevelKey Evidence
Functional rehabilitation as first-line1bWillits et al. (Meta-analysis): Similar re-rupture rates to surgery if functional rehab used.
Simmonds test for diagnosis2aHigh Specificity (>90%). The "Gold Standard" Clinical Test.
VTE prophylaxis2aBOAST Guidelines: Recommend risk assessment for all immobilized lower limb trauma.
Early Weight Bearing1bStimulates collagen alignment and reduces atrophy.

11. Patient/Layperson Explanation

What has happened?

Your Achilles tendon — the strong cord at the back of your ankle connecting your calf muscle to your heel — has snapped (ruptured). This is like a rope that has frayed over time finally breaking when put under sudden strain. It is a common injury, especially in active people in their 30s-50s.

Why did it happen?

In most cases, the tendon had already weakened over time (a process called tendinopathy) without causing any symptoms. When you suddenly pushed off or changed direction, the tendon could not cope and snapped. Certain medications (like ciprofloxacin) and steroids can weaken tendons and increase this risk.

How is it treated?

Good news: in the past, everyone had surgery. We now know that for most people, healing in a special boot works just as well and avoids surgical risks.

  1. Boot treatment (functional rehabilitation): You will wear a boot with your foot pointed down (like a ballerina). We gradually flatten your foot over 8-10 weeks while the tendon heals. You will work with a physiotherapist.
  2. Surgery: This may be recommended if you are a professional athlete, if the diagnosis was delayed, or if the tendon re-ruptures. Surgery stitches the tendon together but carries risks of wound problems.

What to expect

  • You will be in a boot for about 10 weeks
  • You can start putting weight on the leg after 2-4 weeks (in the boot)
  • It takes 6-9 months before you can return to running or sports
  • Your calf muscle may remain slightly smaller on that side permanently

When to seek help

Contact your doctor or return to hospital if:

  • Increasing pain or swelling in the leg (DVT risk)
  • Problems with the boot or cast (rubbing, pressure sores)
  • You hear or feel another "pop" (re-rupture)
  • Signs of infection if you had surgery (redness, pus, fever)

12. References

Primary Guidelines

  1. British Orthopaedic Association Standards for Trauma (BOAST 13). Acute Achilles Tendon Rupture. 2019. BOA BOAST
  2. American Academy of Orthopaedic Surgeons. The Diagnosis and Treatment of Acute Achilles Tendon Rupture. 2010.

Key Trials

  1. Willits K, et al. Operative versus nonoperative treatment of acute Achilles tendon ruptures: a multicenter randomized trial using accelerated functional rehabilitation. J Bone Joint Surg Am. 2010;92(17):2767-75. PMID: 21037028
  2. Lantto I, et al. A prospective randomized trial comparing surgical and nonsurgical treatments of acute Achilles tendon ruptures. Am J Sports Med. 2016;44(9):2406-14. PMID: 27307296
  3. Maffulli N, et al. Minimally invasive surgery for Achilles tendon ruptures: current evidence. Foot Ankle Clin. 2017;22(3):567-576. PMID: 28779822

Further Resources

  • Versus Arthritis: versusarthritis.org
  • Chartered Society of Physiotherapy: csp.org.uk


13. Examination Focus

The "Simmonds" Station

1. Inspect (Prone Position)

  • Ask patient to kneel on chair or lie prone on couch with feet hanging off edge.
  • Look:
    • Swelling: Loss of definition of the tendon.
    • The Angle of Dangle (Matles Test): The normal foot rests in slight plantarflexion (gravity). The ruptured foot hangs vertically (neutral/dorsiflexed) because the "guy rope" is cut.
    • Gap: Visible defect?

2. Palpate

  • Feel the tendon defect. It is usually 2-6cm proximal to the calcaneus.
  • Tenderness.

3. The Special Test (Simmonds-Thompson)

  • Action: Squeeze the calf muscle belly (Gastrocnemius).
  • Normal Response: The foot plantarflexes (points down). (The tendon pulls the heel up).
  • Abnormal Response: The foot stays still. (The connection is broken).
  • Note: This is the ONLY test that matters.

Viva Questions:

  • Q: Can a patient walk with a ruptured Achilles?
    • A: YES. They can use Tibialis Posterior and Peroneals towards the floor. Do not rule out rupture based on ability to walk.
  • Q: Why do we treat in Equinus (Plantarflexion)?
    • A: To approximate the tendon ends. Treating in neutral leaves a gap -> lengthened tendon -> functional failure.
  • Q: What is the main risk of surgery?
    • A: Wound Breakdown. The skin is tenuous. Infection here often leads to disastrous consequences (exposed tendon).
  • Q: What nerve is at risk?
    • A: Sural Nerve (runs lateral to the tendon).

Medical Disclaimer: MedVellum content is for educational purposes and clinical reference. Clinical decisions should account for individual patient circumstances. Consult an orthopaedic surgeon for Achilles tendon rupture management.

Last updated: 2025-12-22

At a Glance

EvidenceHigh
Last Updated2025-12-22

Red Flags

  • Missed diagnosis (patient sent home with 'ankle sprain')
  • Skin compromise in chronic/neglected rupture
  • Re-rupture after treatment
  • Deep wound infection post-surgery

Clinical Pearls

  • **"Someone Kicked Me"**: Patients classically describe feeling like they were kicked or struck in the back of the leg. This is pathognomonic history for Achilles rupture.
  • **They CAN Still Walk**: Do not be fooled! Patients can walk and even weakly plantarflex (using tibialis posterior and peronei). The Simmonds test is mandatory for all posterior ankle pain.
  • **Equinus Position**: Initial immobilization MUST be in full plantarflexion (equinus) to approximate tendon ends. Casting at 90 degrees leaves a gap and prevents healing.
  • Loss of push-off power (End-stage failure).
  • **Red Flags — High suspicion if:**

Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines