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Haematology
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Gastroenterology

Anaemia

High EvidenceUpdated: 2026-01-01

On This Page

Red Flags

  • Severe symptomatic anaemia (Hb less than 70)
  • Rapid drop in haemoglobin
  • Signs of active bleeding
  • Haemodynamic instability
  • Neurological symptoms (B12 deficiency)
  • Suspected malignancy
Overview

Anaemia

1. Clinical Overview

Summary

Anaemia is defined as haemoglobin concentration below the normal range for age and sex. It is not a diagnosis but a sign of underlying disease. Classification by mean corpuscular volume (MCV) provides a practical diagnostic framework: microcytic (MCV less than 80 fL), normocytic (MCV 80-100 fL), and macrocytic (MCV greater than 100 fL). Iron deficiency is the most common cause worldwide. Investigation should identify the cause - in older patients with iron deficiency, GI malignancy must be excluded. Treatment is directed at the underlying cause plus replacement therapy where appropriate.

Key Facts

  • Definition: Hb less than 130 g/L (men), less than 120 g/L (women) (WHO criteria)
  • Prevalence: 25% of world population; most common haematological abnormality
  • Demographics: More common in women (menstruation, pregnancy), elderly, developing countries
  • Classification: Microcytic, normocytic, macrocytic (by MCV)
  • Gold Standard Investigation: FBC, blood film, reticulocytes, iron studies, B12/folate
  • First-line Treatment: Treat underlying cause + replacement if deficient
  • Prognosis: Depends on underlying cause

Clinical Pearls

MCV Pearl: MCV is your first branch point. Microcytic = think iron. Macrocytic = think B12/folate/liver/alcohol/drugs.

Iron Deficiency Pearl: In men and postmenopausal women with iron deficiency, GI investigation (OGD + colonoscopy) is MANDATORY to exclude malignancy.

B12 Pearl: Neurological damage from B12 deficiency can be irreversible. Subacute combined degeneration affects dorsal columns and corticospinal tracts.

Reticulocyte Pearl: Reticulocytes are high if marrow is responding (haemolysis, bleeding, treatment response). Low reticulocytes = production problem.

Ferritin Pearl: Ferritin is an acute phase reactant. May be "normal" in iron deficiency with concurrent inflammation. Check transferrin saturation.

Why This Matters Clinically

Anaemia is extremely common and the underlying cause must be found. Missing GI cancer as a cause of iron deficiency is a serious diagnostic failure. Systematic investigation using MCV and indices identifies the cause in most cases.


2. Epidemiology

Prevalence

PopulationPrevalence
Global25% (1.6 billion people)
Pregnant women40%
Children under 540%
Elderly20%

Causes by Frequency

CauseContribution
Iron deficiency50% (most common globally)
Anaemia of chronic disease30% (in hospitalised patients)
ThalassaemiaCommon in Mediterranean, Asian, African populations
B12/folate deficiency5-10%
Haemolytic anaemias5%

3. Pathophysiology

Normal Erythropoiesis

  • Erythropoietin (kidney) → bone marrow → red cell production
  • Requires: iron, B12, folate, functioning marrow
  • RBC lifespan: 120 days

Classification by MCV

Microcytic (MCV less than 80 fL):

  • Iron deficiency (most common)
  • Thalassaemia
  • Anaemia of chronic disease (may be normocytic)
  • Sideroblastic anaemia
  • Lead poisoning

Normocytic (MCV 80-100 fL):

  • Anaemia of chronic disease
  • Acute blood loss
  • Chronic kidney disease
  • Bone marrow failure/infiltration
  • Mixed deficiencies (iron + B12)
  • Haemolysis

Macrocytic (MCV greater than 100 fL):

  • B12 deficiency (megaloblastic)
  • Folate deficiency (megaloblastic)
  • Alcohol/liver disease
  • Hypothyroidism
  • Myelodysplasia
  • Drugs (methotrexate, azathioprine, hydroxyurea)
  • Reticulocytosis (larger cells)

Iron Deficiency Anaemia

Causes:

  • Blood loss (GI, menstrual) - most common
  • Reduced absorption (coeliac, gastrectomy)
  • Increased requirements (pregnancy, growth)
  • Poor dietary intake (rare in developed countries alone)

Pathophysiology:

  • Depleted iron stores → impaired haem synthesis → reduced Hb

B12 Deficiency

Causes:

  • Pernicious anaemia (autoimmune gastritis, anti-IF antibodies)
  • Gastric surgery
  • Ileal disease/resection
  • Dietary (strict vegans)
  • Medications (metformin, PPIs)

Pathophysiology:

  • B12 needed for DNA synthesis → megaloblastic erythropoiesis
  • Also causes neurological damage (subacute combined degeneration)

4. Clinical Presentation

General Symptoms of Anaemia

SymptomMechanism
FatigueReduced oxygen delivery
Dyspnoea on exertionCompensatory response
PalpitationsTachycardia
Dizziness/light-headednessCerebral hypoperfusion
HeadacheHypoxia
PallorReduced haemoglobin

Cause-Specific Features

CauseFeatures
Iron deficiencyPica, koilonychia, angular stomatitis, glossitis
B12 deficiencyGlossitis, neurological (paraesthesia, ataxia, dementia)
Folate deficiencySimilar to B12 but NO neurological signs
HaemolysisJaundice, dark urine, splenomegaly
Chronic diseaseFeatures of underlying condition

Red Flags

[!CAUTION]

  • Hb less than 70 g/L or symptomatic at rest
  • Signs of active bleeding (melaena, haematemesis)
  • Haemodynamic instability
  • Neurological symptoms (B12)
  • Suspicious features for malignancy (weight loss, mass)

5. Clinical Examination

General

  • Pallor (conjunctivae, palms, nail beds)
  • Tachycardia
  • Systolic flow murmur (hyperdynamic circulation)
  • Signs of cardiac failure (if severe)

Specific Signs

SignSuggests
Koilonychia (spoon nails)Iron deficiency
Angular stomatitisIron or B12 deficiency
GlossitisIron or B12 deficiency
Jaundice + pallorHaemolysis
SplenomegalyHaemolysis, haematological malignancy
Peripheral neuropathyB12 deficiency
Petechiae/bruisingBone marrow failure

6. Investigations

First-Line

TestValue
FBCHb, MCV, MCH, MCHC, RDW, WCC, platelets
Blood filmMorphology (microcytic, macrocytic, hypersegmented neutrophils)
Reticulocyte countProduction vs destruction
Iron studiesFerritin, iron, TIBC, transferrin saturation
B12 and folate

Iron Studies Interpretation

ConditionFerritinIronTIBCTransferrin Sat
Iron deficiencyLowLowHighLow (less than 16%)
Chronic diseaseNormal/HighLowLow/NormalLow/Normal
Thalassaemia traitNormalNormalNormalNormal

Additional Tests (as indicated)

TestIndication
Haemolysis screenSuspected haemolysis (LDH, bilirubin, haptoglobin, Coombs)
Haemoglobin electrophoresisSuspected thalassaemia/haemoglobinopathy
Bone marrow biopsyUnexplained cytopenias, suspected marrow pathology
Parietal cell/IF antibodiesSuspected pernicious anaemia
TFTsMacrocytosis
LFTsLiver disease
GI investigationsIron deficiency in men/postmenopausal women
Coeliac serologyUnexplained iron deficiency

7. Management

Management Algorithm

           ANAEMIA CONFIRMED (Hb LOW)
                      ↓
┌────────────────────────────────────────────────────────────┐
│              CLASSIFY BY MCV                               │
├──────────────────┬─────────────────┬──────────────────────┤
│ MICROCYTIC       │ NORMOCYTIC      │ MACROCYTIC           │
│ (MCV less than   │ (MCV 80-100)    │ (MCV greater than    │
│ 80)              │                 │ 100)                 │
│ Iron studies     │ Reticulocytes   │ B12, folate          │
│ Ferritin first   │ Renal function  │ LFTs, TFTs           │
│                  │ Haemolysis      │ Blood film           │
│                  │ screen          │                      │
└──────────────────┴─────────────────┴──────────────────────┘
                      ↓
┌────────────────────────────────────────────────────────────┐
│               IRON DEFICIENCY                              │
│  1. Find the cause (GI investigation if male/postmeno)     │
│  2. Oral iron (ferrous sulfate 200mg TDS) x 3 months       │
│  3. Recheck Hb at 2-4 weeks (should rise 10-20 g/L/month)  │
│  4. IV iron if intolerant, non-compliant, malabsorption    │
└────────────────────────────────────────────────────────────┘
                      ↓
┌────────────────────────────────────────────────────────────┐
│          B12 DEFICIENCY                                    │
│  1. Identify cause (pernicious anaemia, dietary, ileal)    │
│  2. IM hydroxocobalamin: 1mg on alternate days x 2 weeks   │
│     Then 1mg every 3 months for life (if pernicious)       │
│  3. Oral B12 may be effective for dietary deficiency       │
│  4. Urgent treatment if neurological symptoms              │
└────────────────────────────────────────────────────────────┘
                      ↓
┌────────────────────────────────────────────────────────────┐
│          FOLATE DEFICIENCY                                 │
│  1. Rule out B12 deficiency first (can mask/worsen neuro)  │
│  2. Folic acid 5mg daily for 4 months                      │
│  3. Address cause (diet, malabsorption, drugs)             │
└────────────────────────────────────────────────────────────┘

Iron Replacement

RouteRegimenNotes
OralFerrous sulfate 200mg TDSTake on empty stomach if tolerated; vitamin C helps absorption
IVFerric carboxymaltose (Ferinject)If oral fails, malabsorption, or rapid correction needed

B12 Replacement

CauseTreatment
Pernicious anaemiaIM hydroxocobalamin lifelong
DietaryOral cyanocobalamin 50-150mcg daily OR IM
Neurological involvementIM loading (alternate days x 3 weeks), then maintenance

Blood Transfusion

  • Consider if Hb less than 70 g/L or symptomatic at higher levels
  • In elderly/cardiac patients, transfuse slowly (diuretic cover)
  • Aim to correct symptoms, not normalise Hb acutely

8. Complications
ComplicationFeatures
Cardiac failureHigh-output failure in severe anaemia
AnginaMay precipitate in CAD
Subacute combined degenerationB12 deficiency; irreversible if delayed
Growth retardationChronic childhood anaemia
Pregnancy complicationsIron deficiency; low birth weight, prematurity

9. Prognosis and Outcomes
  • Iron deficiency: excellent with replacement and cause treated
  • B12 deficiency: haematological recovery good; neurological may be incomplete if delayed
  • Depends on underlying cause (malignancy, haematological disease)

10. Evidence and Guidelines

Key Guidelines

  1. BSH Guidelines on Iron Deficiency Anaemia — Goddard AF et al. Gut. 2011

  2. BSH Guidelines on Cobalamin and Folate — Devalia V et al. Br J Haematol. 2014

  3. NICE Guideline NG24. Blood transfusion — 2015

Key Evidence

  • Oral iron: raises Hb 10-20 g/L per month
  • IV iron superior to oral for CKD, IBD, post-surgical
  • PMID: 21054451

11. Patient Explanation

What is anaemia?

Anaemia means you don't have enough red blood cells or haemoglobin to carry oxygen around your body. This makes you feel tired and breathless.

What causes it?

The most common cause is iron deficiency, often from blood loss or poor diet. Other causes include vitamin deficiencies (B12, folate) or chronic diseases.

Treatment

Depends on the cause:

  • Iron tablets (may cause dark stools and constipation)
  • B12 injections
  • Treating the underlying condition

When to seek help

  • Very tired, short of breath at rest
  • Black stools or blood in stools
  • Numbness or tingling (B12)

12. References
  1. Goddard AF et al. Guidelines for the management of iron deficiency anaemia. Gut. 2011;60(10):1309-1316. PMID: 21561874

  2. Devalia V et al. Guidelines for the diagnosis and treatment of cobalamin and folate disorders. Br J Haematol. 2014;166(4):496-513. PMID: 24942828

  3. Camaschella C. Iron-Deficiency Anemia. N Engl J Med. 2015;372(19):1832-1843. PMID: 25946282

  4. Green R. Vitamin B12 deficiency from the perspective of a practicing hematologist. Blood. 2017;129(19):2603-2611. PMID: 28360040

  5. NICE Guideline NG24. Blood transfusion. 2015.


13. Examination Focus

Viva Points

"Anaemia is classified by MCV: microcytic (iron, thalassaemia), normocytic (chronic disease, CKD, haemolysis), macrocytic (B12, folate, liver, drugs). Iron deficiency in men/postmenopausal women needs GI investigation for malignancy. Treat with oral iron for 3 months. B12 deficiency can cause irreversible neurological damage - treat urgently."

Common Mistakes

  • ❌ Not investigating cause of iron deficiency (missing GI cancer)
  • ❌ Treating folate before excluding B12 deficiency
  • ❌ Forgetting ferritin is acute phase reactant
  • ❌ Not recognising subacute combined degeneration

Last Reviewed: 2026-01-01 | MedVellum Editorial Team

Last updated: 2026-01-01

At a Glance

EvidenceHigh
Last Updated2026-01-01

Red Flags

  • Severe symptomatic anaemia (Hb less than 70)
  • Rapid drop in haemoglobin
  • Signs of active bleeding
  • Haemodynamic instability
  • Neurological symptoms (B12 deficiency)
  • Suspected malignancy

Clinical Pearls

  • **MCV Pearl**: MCV is your first branch point. Microcytic = think iron. Macrocytic = think B12/folate/liver/alcohol/drugs.
  • **Iron Deficiency Pearl**: In men and postmenopausal women with iron deficiency, GI investigation (OGD + colonoscopy) is MANDATORY to exclude malignancy.
  • **B12 Pearl**: Neurological damage from B12 deficiency can be irreversible. Subacute combined degeneration affects dorsal columns and corticospinal tracts.
  • **Reticulocyte Pearl**: Reticulocytes are high if marrow is responding (haemolysis, bleeding, treatment response). Low reticulocytes = production problem.
  • **Ferritin Pearl**: Ferritin is an acute phase reactant. May be "normal" in iron deficiency with concurrent inflammation. Check transferrin saturation.

Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines