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Nephrology

Hypertension

High EvidenceUpdated: 2026-01-01

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Red Flags

  • Hypertensive emergency (greater than 180/120 with end-organ damage)
  • Accelerated/malignant hypertension (papilloedema)
  • Secondary hypertension features
  • Resistant hypertension
  • Pregnancy (pre-eclampsia)
Overview

Hypertension

1. Clinical Overview

Summary

Hypertension is sustained elevation of blood pressure, defined as clinic BP 140/90 mmHg or higher (or ABPM/HBPM 135/85 mmHg or higher). It is a major modifiable cardiovascular risk factor and the leading risk factor for mortality globally. Most cases are primary (essential) hypertension with no identifiable cause. Secondary causes should be considered in young patients, resistant hypertension, or those with suggestive features. NICE recommends confirming diagnosis with ambulatory BP monitoring (ABPM). Treatment follows a stepwise approach: ACEi/ARB, then calcium channel blocker, then thiazide-like diuretic. Target BP is less than 140/90 (or less than 130/80 if high CV risk, diabetes, or CKD).

Key Facts

  • Definition: Clinic BP 140/90+ OR ABPM/HBPM 135/85+
  • Prevalence: 30% of adults; increases with age
  • Demographics: More common in older adults, African-Caribbean descent
  • Classification: Primary (90-95%) vs Secondary (5-10%)
  • Gold Standard Diagnosis: ABPM (or HBPM if ABPM declined/not tolerated)
  • First-line Treatment: ACEi/ARB OR CCB (based on age/ethnicity)
  • Prognosis: Treatment reduces MI, stroke, HF, CKD progression

Clinical Pearls

ABPM Pearl: NICE requires ABPM to confirm hypertension and stage it. Clinic BP alone is not sufficient for diagnosis.

A+C+D Pearl: NICE stepwise: Step 1: A (ACEi/ARB) or C (CCB); Step 2: A+C; Step 3: A+C+D (thiazide-like); Step 4: Add spironolactone if K+ permits.

African-Caribbean Pearl: CCB is first-line for Black African/Caribbean patients of any age (reduced RAA system response).

White Coat Pearl: White coat hypertension (high clinic, normal ABPM) increases CV risk slightly. Monitor annually.

Secondary Pearl: Consider secondary causes in: age less than 40, resistant HTN, hypokalaemia, renal bruits, cushingoid features.

Why This Matters Clinically

Hypertension is the most important modifiable CV risk factor. Treatment prevents stroke, MI, HF, and CKD progression. Most patients are asymptomatic - screening and opportunistic BP measurement are essential.


2. Epidemiology

Prevalence

Age GroupPrevalence
18-3910%
40-5935%
60+60%+
Overall adults30%

Demographics

  • Increases with age
  • Higher in African-Caribbean populations
  • More common in men until 45; equalises after menopause

Risk Factors

CategoryFactors
Non-modifiableAge, family history, ethnicity, male sex
ModifiableObesity, high sodium diet, low potassium, sedentary lifestyle, excess alcohol, smoking
ComorbiditiesDiabetes, CKD, OSA

3. Pathophysiology

Primary (Essential) Hypertension (90-95%)

Multifactorial:

  • Genetic predisposition
  • Sympathetic nervous system overactivity
  • Renin-angiotensin-aldosterone system (RAAS) activation
  • Endothelial dysfunction
  • Arterial stiffness
  • Sodium retention

Secondary Hypertension (5-10%)

CauseFeatures
Renal parenchymal diseaseCKD, raised creatinine
Renovascular diseaseRenal artery stenosis, abdominal bruit
Primary aldosteronismHypokalaemia without diuretics
PhaeochromocytomaEpisodic HTN, sweating, palpitations
Cushing's syndromeCushingoid features, hypokalaemia
Coarctation of aortaArm-leg BP difference, young patient
OSASnoring, daytime somnolence
Drug-inducedNSAIDs, steroids, OCP, cocaine

4. Clinical Presentation

Usually Asymptomatic

Symptoms of Severe/Complicated HTN

Secondary HTN Clues

FindingSuggests
Young age (less than 40)Secondary cause
Resistant HTNSecondary, non-adherence, white coat
HypokalaemiaPrimary aldosteronism
Abdominal bruitRenovascular disease
Radio-femoral delayCoarctation
CushingoidCushing's syndrome
Episodic symptomsPhaeochromocytoma

Red Flags - Hypertensive Emergency

[!CAUTION] BP greater than 180/120 with:

  • Papilloedema (malignant HTN)
  • Acute coronary syndrome
  • Heart failure
  • Aortic dissection
  • Stroke/encephalopathy
  • Acute kidney injury
  • Eclampsia

Most patients have no symptoms
Common presentation.
Discovered on routine screening
Common presentation.
5. Clinical Examination

Blood Pressure Measurement

  • Correct cuff size (bladder 80% of arm circumference)
  • Seated, arm supported at heart level
  • After 5 minutes rest
  • Measure both arms (use higher reading)
  • Record as average of last 2 readings

Examination for End-Organ Damage

SystemFindings
CardiovascularApex displacement (LVH), S4, murmurs
FundoscopyArteriolar narrowing, AV nipping, haemorrhages, exudates, papilloedema
RenalBruits (renovascular disease)
NeurologicalFocal signs (stroke)

Features of Secondary Causes

  • Cushingoid features
  • Thyroid signs
  • Radio-femoral delay

6. Investigations

Confirming Diagnosis

MethodThreshold
Clinic BP140/90 mmHg or higher
ABPM (preferred)Daytime mean 135/85 or higher
HBPMMean 135/85 or higher

Staging (NICE)

StageClinic BPABPM/HBPM
Stage 1140-159/90-99135-149/85-94
Stage 2160-179/100-109150-174/95-109
Stage 3 (severe)180+/110+175+/110+

Baseline Investigations (All Patients)

TestPurpose
U&ERenal function, hypokalaemia
Glucose/HbA1cDiabetes screening
Lipid profileCV risk
UrinalysisProteinuria, haematuria
12-lead ECGLVH, ischaemia

Additional (if secondary cause suspected)

  • Renal ultrasound, renal artery Doppler
  • Aldosterone:renin ratio
  • 24-hour urinary catecholamines/metanephrines
  • Dexamethasone suppression test
  • MRI adrenals

QRISK3

  • 10-year CV risk calculation
  • Used to guide statin therapy

7. Management

Management Algorithm (NICE)

         CONFIRMED HYPERTENSION (ABPM/HBPM)
                       ↓
┌────────────────────────────────────────────────────────────┐
│           LIFESTYLE MODIFICATION (ALL)                     │
│  - Salt restriction (less than 6g/day)                     │
│  - Weight loss if overweight                               │
│  - Regular exercise                                        │
│  - Limit alcohol                                           │
│  - Stop smoking                                            │
│  - DASH diet                                               │
└────────────────────────────────────────────────────────────┘
                       ↓
┌────────────────────────────────────────────────────────────┐
│                STEP 1                                      │
│  Age less than 55 or diabetes:     A (ACEi or ARB)         │
│  Age 55+ or Black African/Caribbean: C (CCB - amlodipine)  │
└────────────────────────────────────────────────────────────┘
                       ↓
┌────────────────────────────────────────────────────────────┐
│                STEP 2                                      │
│                   A + C                                    │
└────────────────────────────────────────────────────────────┘
                       ↓
┌────────────────────────────────────────────────────────────┐
│                STEP 3                                      │
│               A + C + D (Thiazide-like)                    │
│  (Indapamide 1.5mg or chlortalidone 12.5-25mg)             │
└────────────────────────────────────────────────────────────┘
                       ↓
┌────────────────────────────────────────────────────────────┐
│           STEP 4 (Resistant HTN)                           │
│  Check K+:                                                 │
│  - If K+ 4.5 or less → Add spironolactone 25mg             │
│  - If K+ greater than 4.5 → Alpha-blocker or beta-blocker  │
│  Specialist referral if still uncontrolled                 │
└────────────────────────────────────────────────────────────┘

Drug Classes

ClassExamplesNotes
ACEiRamipril, lisinoprilFirst-line if less than 55, DM, CKD. Cough SE
ARBLosartan, candesartanIf ACEi intolerant
CCBAmlodipineFirst-line if 55+ or Black. Ankle oedema SE
Thiazide-likeIndapamide, chlortalidoneStep 3. Check U&E
MRASpironolactoneStep 4. Monitor K+
Alpha-blockerDoxazosinStep 4 alternative
Beta-blockerBisoprololLess preferred; use if compelling indication

Treatment Targets

PopulationClinic BP TargetABPM/HBPM Target
Under 80 yearsLess than 140/90Less than 135/85
80 years and overLess than 150/90Less than 145/85
Diabetes/CKD/high CV riskLess than 130/80Less than 125/75

Hypertensive Emergency

  • Controlled BP reduction (not precipitous - risk of stroke)
  • IV labetalol, sodium nitroprusside, or GTN infusion
  • Specialist/HDU management

8. Complications
ComplicationMechanism
StrokeIschaemic and haemorrhagic
Coronary artery diseaseAccelerated atherosclerosis
Heart failureLVH → diastolic then systolic dysfunction
Chronic kidney diseaseNephrosclerosis
Peripheral arterial diseaseAtherosclerosis
RetinopathyMicrovascular damage
Aortic dissectionIntimal tear

9. Prognosis and Outcomes

Benefits of Treatment

  • 40% reduction in stroke
  • 20-25% reduction in MI
  • 50% reduction in heart failure

BP Control Rates

  • Less than 50% of treated patients achieve target BP
  • Adherence is a major challenge

10. Evidence and Guidelines

Key Guidelines

  1. NICE Guideline NG136. Hypertension in adults — 2019 (updated 2022)

  2. ESC/ESH Guidelines for Management of Arterial Hypertension — 2018

  3. AHA/ACC Guideline for High Blood Pressure — 2017

Landmark Trials

SPRINT

  • Intensive BP control (target less than 120) reduced CV events
  • PMID: 26551272

ALLHAT

  • Thiazides as effective as CCBs and ACEi for outcomes
  • PMID: 12479763

PATHWAY-2

  • Spironolactone most effective add-on for resistant HTN
  • PMID: 26559744

11. Patient Explanation

What is high blood pressure?

High blood pressure means the force of blood pushing against your artery walls is too high. Over time, this can damage your heart, brain, kidneys, and eyes.

Why does it matter?

High blood pressure usually has no symptoms, but if untreated, it significantly increases your risk of heart attack, stroke, and kidney disease.

Treatment

  • Lifestyle changes: reduce salt, lose weight, exercise, limit alcohol
  • Medication: usually one or more tablets taken daily
  • The goal is to get your BP below 140/90 (or lower if you have diabetes or kidney disease)

Taking your medication

  • Take tablets at the same time each day
  • Don't stop without discussing with your doctor
  • Some medications may cause side effects - discuss alternatives if needed

12. References
  1. NICE Guideline NG136. Hypertension in adults: diagnosis and management. 2019.

  2. Williams B et al. 2018 ESC/ESH Guidelines for the management of arterial hypertension. Eur Heart J. 2018;39(33):3021-3104. PMID: 30165516

  3. SPRINT Research Group. A Randomized Trial of Intensive versus Standard Blood-Pressure Control. N Engl J Med. 2015;373(22):2103-2116. PMID: 26551272

  4. Williams B et al. Spironolactone versus placebo, bisoprolol, and doxazosin to determine optimal treatment for resistant hypertension (PATHWAY-2). Lancet. 2015;386(10008):2059-2068. PMID: 26414968

  5. ALLHAT Officers. Major outcomes in high-risk hypertensive patients randomized to ACE inhibitor or calcium channel blocker vs diuretic. JAMA. 2002;288(23):2981-2997. PMID: 12479763

  6. Whelton PK et al. 2017 ACC/AHA Guideline for High Blood Pressure. Hypertension. 2018;71(6):e13-e115. PMID: 29133356


13. Examination Focus

Viva Points

"Hypertension is diagnosed with ABPM (135/85+). NICE stepwise: A or C first (A if under 55/DM; C if 55+/Black), then A+C, then A+C+D, then spironolactone. Target less than 140/90. Secondary causes: renovascular, primary aldosteronism, phaeo, Cushing's. Treatment reduces stroke 40%, MI 25%."

Common Mistakes

  • ❌ Diagnosing without ABPM
  • ❌ Wrong drug for age/ethnicity
  • ❌ Not checking for secondary causes in young/resistant HTN
  • ❌ ACEi + ARB together (avoid combination)
  • ❌ Not monitoring U&E with ACEi/ARB/diuretics

Last Reviewed: 2026-01-01 | MedVellum Editorial Team

Last updated: 2026-01-01

At a Glance

EvidenceHigh
Last Updated2026-01-01

Red Flags

  • Hypertensive emergency (greater than 180/120 with end-organ damage)
  • Accelerated/malignant hypertension (papilloedema)
  • Secondary hypertension features
  • Resistant hypertension
  • Pregnancy (pre-eclampsia)

Clinical Pearls

  • **ABPM Pearl**: NICE requires ABPM to confirm hypertension and stage it. Clinic BP alone is not sufficient for diagnosis.
  • **A+C+D Pearl**: NICE stepwise: Step 1: A (ACEi/ARB) or C (CCB); Step 2: A+C; Step 3: A+C+D (thiazide-like); Step 4: Add spironolactone if K+ permits.
  • **African-Caribbean Pearl**: CCB is first-line for Black African/Caribbean patients of any age (reduced RAA system response).
  • **White Coat Pearl**: White coat hypertension (high clinic, normal ABPM) increases CV risk slightly. Monitor annually.
  • **Secondary Pearl**: Consider secondary causes in: age less than 40, resistant HTN, hypokalaemia, renal bruits, cushingoid features.

Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines