Orthopaedics
Emergency Medicine
Sports Medicine
High Evidence
Peer reviewed

Joint Dislocation in Adults

A joint dislocation occurs when the articular surfaces of a joint lose complete contact with one another, resulting in j... FRCS (Tr&Orth) exam preparation.

Updated 10 Jan 2026
Reviewed 17 Jan 2026
48 min read
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MedVellum Editorial Team
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MedVellum Medical Education Platform

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Urgent signals

Safety-critical features pulled from the topic metadata.

  • Neurovascular compromise (absent pulses, sensory/motor deficit)
  • Open dislocation with exposed joint
  • Compartment syndrome (pain out of proportion, tense compartment)
  • Irreducible dislocation (interposed soft tissue)

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  • FRCS (Tr&Orth)

Linked comparisons

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  • Fracture-Dislocation
  • Ligamentous Injury Without Dislocation

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FRCS (Tr&Orth)
Clinical reference article

Joint Dislocation in Adults

1. Clinical Overview

Summary

A joint dislocation occurs when the articular surfaces of a joint lose complete contact with one another, resulting in joint incongruity. Unlike subluxation (partial loss of contact), dislocation represents complete displacement of joint surfaces. Joint dislocations are common orthopaedic emergencies that typically result from high-energy trauma in younger adults or lower-energy mechanisms in older individuals or those with pre-existing joint laxity. The glenohumeral (shoulder) joint is the most frequently dislocated major joint in the body, accounting for approximately 45-50% of all dislocations. [1,2]

Dislocations require prompt recognition and reduction to restore joint congruity, minimize neurovascular complications, and optimize functional recovery. The key clinical challenge lies in: (1) accurate diagnosis including identification of associated fractures; (2) systematic neurovascular assessment before and after reduction; (3) appropriate reduction technique selection; (4) recognition of complications including nerve injury (10-25%), vascular injury (1-5%), and recurrent instability (15-50% depending on age and joint). [1,3] Early reduction (ideally within 24 hours) significantly reduces the risk of complications and improves long-term outcomes.

Key Facts

  • Definition: Complete loss of contact between articular surfaces of a joint
  • Incidence: Shoulder dislocation 23.9 per 100,000 person-years; overall joint dislocations represent 2-3% of emergency department orthopaedic presentations [1]
  • Mortality: less than 0.1% unless severe polytrauma or vascular injury
  • Peak age: Bimodal distribution—young adults (20-30 years, trauma/sports) and elderly (> 65 years, falls)
  • Critical feature: Obvious joint deformity, severe pain, complete loss of active and passive range of motion
  • Key investigation: Radiographs (AP + lateral minimum) before and after reduction; CT for complex patterns
  • First-line treatment: Prompt closed reduction under procedural sedation, neurovascular reassessment, post-reduction immobilization

Clinical Pearls

"Posterior shoulder dislocations are easily missed" — Posterior dislocations account for 2-4% of shoulder dislocations but are missed in up to 50-79% of initial presentations. Look for the "lightbulb sign" on AP radiograph and always obtain axillary or scapular-Y views. [4]

"Document neurovascular status before and after reduction" — Axillary nerve injury occurs in 10-25% of anterior shoulder dislocations, axillary artery injury in 1-2%. Always document pulses, sensation (axillary nerve regimental badge area), and motor function (deltoid) before reduction attempts, and repeat after successful reduction. [5]

"The younger the patient, the higher the recurrence risk" — Recurrence rates after first-time anterior shoulder dislocation approach 50-90% in patients less than 20 years old, compared to 10-15% in patients > 40 years old. Age less than 20 years is the strongest predictor of recurrence (OR 4.24, 95% CI 2.8-6.23). [6]

"Reduce early to prevent complications" — Delayed reduction beyond 24-48 hours increases difficulty, procedural sedation requirements, and risk of iatrogenic fracture. Muscle spasm and soft tissue interposition make late reductions progressively more challenging.

"Always X-ray before and after reduction" — Pre-reduction radiographs identify associated fractures (Hill-Sachs, Bankart, greater tuberosity) that may influence reduction technique and post-reduction management. Post-reduction films confirm successful reduction and detect iatrogenic fractures.

Why This Matters Clinically

Joint dislocations are among the most common orthopaedic emergencies managed in emergency departments worldwide. Prompt recognition and appropriate management prevent neurovascular complications, reduce the risk of recurrent instability, and optimize long-term functional outcomes. The glenohumeral joint, with its inherent anatomical instability (shallow glenoid, large humeral head), is particularly prone to dislocation and subsequent recurrence. Understanding the pathoanatomy of specific dislocation patterns—particularly the Bankart lesion (anteroinferior glenoid labral tear) and Hill-Sachs deformity (posterosuperolateral humeral head impaction fracture)—is essential for appropriate treatment selection and counseling patients about recurrence risk and surgical indications.


2. Epidemiology

Incidence & Prevalence

Overall Incidence:

  • Shoulder dislocation: 23.9 per 100,000 person-years (most common major joint dislocation) [1]
  • All joint dislocations: Estimated 50-70 per 100,000 person-years
  • Emergency department presentations: 2-3% of all orthopaedic trauma cases

Joint-Specific Distribution:

JointFrequencyTypical MechanismPeak Age
Shoulder (glenohumeral)45-50%Abduction + external rotation; fall on outstretched hand20-30 years
Finger (interphalangeal)15-20%Direct blow to flexed finger (sports)15-40 years
Elbow10-15%Fall on outstretched hand; hyperextensionChildren (nursemaid's elbow); adults 20-40 years
Patella5-10%Twisting injury, direct blow; lateral displacement15-25 years (females > males)
Ankle3-5%High-energy trauma (usually fracture-dislocation)20-50 years
Hip2-5%High-energy trauma (dashboard injury, MVA)20-40 years

Demographics

FactorDetails
AgeBimodal: 20-30 years (sports/trauma) and > 65 years (falls)
SexMale predominance 2-3:1 for traumatic dislocations; female predominance for patellar dislocation
EthnicityNo significant variation for traumatic dislocations
GeographyHigher incidence in regions with increased sports participation and motor vehicle trauma
SettingEmergency departments, sports fields, trauma centers

Risk Factors

Non-Modifiable:

Risk FactorRelative RiskMechanism
Age less than 20 years4-5x higher recurrenceJoint capsule elasticity, activity level, healing capacity
Generalized joint hyperlaxity4.55 (95% CI 2.19-9.44) [6]Ligamentous laxity, reduced static stability
Previous dislocation3-6xCapsulolabral injury, bone loss, proprioceptive deficit
Bone loss (glenoid > 20%, Hill-Sachs off-track)2.8-5.5x [6]Reduced osseous stability, engaging Hill-Sachs lesion

Modifiable:

Risk FactorRelative RiskMechanism
Contact/collision sports3-5xHigh-force trauma, repetitive microtrauma
High-energy trauma exposure5-10xMotor vehicle accidents, falls from height
Inadequate rehabilitation2-3xPersistent muscle weakness, proprioceptive deficit
Early return to sport2-4xIncomplete soft tissue healing

Shoulder Dislocation Epidemiology (Detailed)

Direction:

  • Anterior: 95-97% (subcoracoid most common, then subglenoid, subclavicular, intrathoracic) [2]
  • Posterior: 2-4% (often missed; seizures, electrocution, high-energy trauma)
  • Inferior (luxatio erecta): less than 1%
  • Superior: less than 1% (requires acromion/clavicle fracture)

Associated Injuries:

InjuryIncidenceClinical Significance
Bankart lesion (anteroinferior labral tear)65-90% of first-time dislocations [2]Primary pathology; surgical target
Hill-Sachs deformity (posterolateral humeral head impaction)40-90% depending on imaging modality [7]Risk factor for recurrence if "off-track"
Glenoid bone loss20-40% after recurrent instabilityCritical > 20%; requires bone augmentation
Greater tuberosity fracture10-15% (higher in elderly)May require surgical fixation
Axillary nerve injury10-25% (usually neurapraxia) [5]Test regimental badge sensation + deltoid
Rotator cuff tear10-35% (increases with age > 40 years)Higher risk in elderly; requires MRI assessment

3. Pathophysiology & Anatomical Considerations

Glenohumeral Joint Stability Mechanisms

The shoulder joint sacrifices stability for mobility, making it the most frequently dislocated major joint. Stability depends on static and dynamic stabilizers:

Static Stabilizers:

  1. Glenoid labrum: Deepens glenoid socket by 50%; provides attachment for glenohumeral ligaments
  2. Glenohumeral ligaments (superior, middle, inferior): Capsular thickenings that resist translation
    • Inferior glenohumeral ligament (IGHL): Primary restraint to anterior translation in abduction/external rotation
  3. Negative intra-articular pressure: Creates suction effect
  4. Bony congruity: Glenoid version and humeral head retroversion

Dynamic Stabilizers:

  1. Rotator cuff muscles: Compress humeral head into glenoid (concavity-compression)
  2. Scapular stabilizers: Maintain optimal glenoid position
  3. Long head of biceps: Superior stability

Mechanism of Anterior Shoulder Dislocation

Step 1: Mechanism of Injury

  • Force application: Abduction + external rotation + extension (classic: fall on outstretched hand)
  • Critical position: 90° abduction, > 90° external rotation
  • Force vector: Anterior-directed force on posterior humeral head

Step 2: Capsuloligamentous Failure

  • IGHL failure: Tensile failure or avulsion from glenoid insertion
  • Anteroinferior labral tear (Bankart lesion): Labrum detaches from glenoid rim
  • Capsular stretching/tearing: Loss of static restraint

Step 3: Humeral Head Displacement

  • Direction: Anteroinferior (subcoracoid 60%, subglenoid 30%, subclavicular/intrathoracic rare)
  • Hill-Sachs lesion formation: Posterolateral humeral head impacts anterior glenoid rim (compression fracture)
  • On-track vs. Off-track: Critical concept for recurrence risk [8]

Step 4: Neurovascular Injury (If Occurs)

  • Axillary nerve: Most common (10-25%); travels anteroinferior to glenohumeral joint
  • Musculocutaneous nerve: Less common (1-2%)
  • Axillary artery: Rare (1-2%); higher risk in elderly and with displaced fractures

Step 5: Post-Reduction Healing

  • Optimal scenario: Anatomical reduction, immobilization, labral/capsular healing
  • Suboptimal healing: Capsular redundancy, persistent Bankart lesion, engaging Hill-Sachs → recurrent instability

Classification Systems

Direction-Based Classification:

TypeDirectionFrequencyTypical Mechanism
AnteriorSubcoracoid, subglenoid95-97%Abduction + external rotation
PosteriorSubacromial, subglenoid2-4%Seizure, electrocution, direct posterior blow
Inferior (Luxatio erecta)Inferior with arm locked overheadless than 1%Hyperabduction
SuperiorSuperiorless than 1%Requires acromial/clavicular fracture

Bankart Lesion Classification:

TypeDescriptionManagement Implications
Soft tissue BankartLabral tear without boneArthroscopic Bankart repair
Bony BankartGlenoid rim fracture + labral tearMay require open repair or bone grafting if > 20%
ALPSA (Anterior Labral Periosteal Sleeve Avulsion)Labrum displaced medially on scapular neckHigher failure rate with standard repair
GLAD (Glenolabral Articular Disruption)Chondral injury + labral tearRisk of early arthritis
Perthes lesionLabral tear with intact periosteumMay appear normal on non-arthrographic imaging

Hill-Sachs Lesion: On-Track vs. Off-Track Concept [8]

Critical concept developed by Yamamoto et al. (2007) and refined by Di Giacomo et al.:

  • On-track lesion: Hill-Sachs lesion medial to glenoid track (contact zone between glenoid and humerus)

    • Lower recurrence risk after arthroscopic Bankart repair alone
  • Off-track lesion: Hill-Sachs lesion lateral to glenoid track

    • Hill-Sachs defect engages anterior glenoid during abduction/external rotation
    • 5-fold higher recurrence risk (OR 5.53, 95% CI 2.21-13.86) [6]
    • Requires additional procedure (remplissage, Latarjet)

Glenoid Bone Loss Quantification:

Bone LossClassificationManagement
less than 10%MinimalArthroscopic Bankart repair
10-20%SubcriticalBankart repair ± remplissage; individualize
> 20%CriticalBone augmentation (Latarjet, iliac crest graft) [9]

4. Clinical Presentation

Symptoms: The Patient's Story

Acute Presentation (First-Time Dislocation):

  • Severe pain: Immediate onset, inability to move shoulder
  • Obvious deformity: "Shoulder looks wrong" or "out of place"
  • Complete loss of function: Cannot actively move arm
  • Mechanical symptoms: Sensation of joint "popping out" ± clunk with reduction (if spontaneous)
  • Mechanism: Usually high-energy (sports, fall, MVA) or low-energy in elderly

History:

  • Age at first dislocation: Critical prognostic factor (younger = higher recurrence)
  • Mechanism: Abduction + external rotation (anterior); seizure/electrocution (posterior)
  • Previous episodes: Recurrent dislocations suggest chronic instability
  • Sport/occupation: Contact sports, overhead activities (swimming, volleyball)
  • Associated injuries: Fractures, neurovascular symptoms

Recurrent Dislocation:

  • Progressively lower force: Eventually dislocates with minimal trauma or activities of daily living
  • Self-reduction: Patients may learn to reduce their own dislocations
  • Apprehension: Fear of dislocation with certain positions (abduction + external rotation)
  • Functional limitation: Avoidance of provocative activities

Signs: Physical Examination

General Appearance:

  • Patient supports affected arm with contralateral hand
  • Reluctance to move shoulder
  • Visible distress

Inspection:

FindingAnterior DislocationPosterior Dislocation
Shoulder contourLoss of normal deltoid contour; "squared-off" appearanceProminent coracoid; flattened anterior shoulder
Humeral head positionPalpable in subcoracoid regionPalpable posteriorly
Arm positionSlight abduction, external rotationAdduction, internal rotation
SymmetryAsymmetric compared to contralateralAsymmetric; inability to externally rotate

Palpation:

  • Humeral head: Palpable in abnormal position (anterior or posterior)
  • Glenoid fossa: Empty on palpation ("sulcus sign")
  • Tenderness: Diffuse shoulder tenderness

Range of Motion:

  • Active ROM: Absent or severely limited
  • Passive ROM: Extremely painful; patient resists examination
  • Locked position: Arm held in fixed position; patient unable to rotate internally (anterior) or externally (posterior)

Neurovascular Examination (CRITICAL):

Document before and after reduction:

StructureAssessmentPositive FindingIncidence
Axillary nerveSensation over lateral deltoid ("regimental badge" area)Numbness, hypoesthesia10-25% [5]
Deltoid muscle contraction (attempt abduction)Weakness, inability to contract
Musculocutaneous nerveSensation over lateral forearmNumbness1-2%
Elbow flexion strengthWeakness
Radial nerveWrist/thumb extensionWeaknessless than 1%
Axillary arteryRadial pulse, brachial pulseDiminished, absent1-2% (higher in elderly)
Capillary refillDelayed > 2 seconds
Hand color/temperaturePale, cold

Special Tests:

TestTechniquePositive FindingClinical Significance
Apprehension test (chronic instability)Patient supine; shoulder abducted 90°, externally rotatePatient apprehension, fear of dislocationSuggests anterior instability
Relocation testPosterior force on proximal humerus during apprehensionRelief of apprehensionConfirms anterior instability
Sulcus signInferior traction on arm in neutral positionSulcus > 2 cm inferior to acromionInferior laxity, multidirectional instability

Red Flags

[!CAUTION] Red Flags — Immediate Escalation Required:

  • Neurovascular compromise: Absent pulses, pallor, paralysis, paresthesias, pain out of proportion (6 Ps) — requires immediate vascular surgery consultation
  • Open dislocation: Skin breach with joint exposure — requires urgent operative debridement and reduction
  • Irreducible dislocation: Failed closed reduction attempts — may indicate soft tissue interposition (long head of biceps, capsule); requires operative reduction
  • Compartment syndrome: Pain out of proportion, tense compartment, progressive neurological deficit — requires urgent fasciotomy
  • Posterior shoulder dislocation: Easily missed (50-79% initial miss rate) [4]; requires high index of suspicion
  • Associated displaced fractures: Greater tuberosity > 5 mm displacement, glenoid rim fractures > 20% — may require operative fixation
  • Bilateral shoulder dislocations: Suggests seizure or electrocution; often posterior; high risk of recurrence

5. Investigations

First-Line Investigations — Do Immediately

1. Clinical Assessment

  • History and examination: Usually sufficient for diagnosis
  • Neurovascular assessment: Mandatory before any intervention
  • Documentation: Record neurovascular status in detail

2. Radiographs (Before Reduction) — MANDATORY

Standard Views:

ViewWhat It ShowsKey Findings
AP (anteroposterior)Humeral head position relative to glenoidAnterior: humeral head inferior/medial; Posterior: "lightbulb sign" (internally rotated humerus) [4]
Axillary or Scapular-YDirection of dislocationAxillary: humeral head anterior or posterior to glenoid; Y-view: humeral head off glenoid "Y"
AP in internal rotation (if posterior suspected)Hill-Sachs or reverse Hill-SachsTrough sign, rim sign

Associated Fractures to Identify:

FractureIncidenceRadiographic FindingClinical Significance
Hill-Sachs40-90% [7]Posterolateral humeral head impaction (AP in internal rotation)Off-track lesions → higher recurrence
Bankart (bony)10-20%Anteroinferior glenoid rim fracture> 20% → bone grafting required
Greater tuberosity10-15%Fracture fragment displaced > 5 mmMay require ORIF
Reverse Hill-SachsPosterior dislocationsAnteromedial humeral head impactionLarge lesions → remplissage or arthroplasty

3. Point-of-Care Ultrasound (POCUS) — Emerging Modality

Recent meta-analysis (Gottlieb et al., 2022): [10]

  • Sensitivity: 100% (95% CI 85.6-100%)
  • Specificity: 100% (95% CI 79.4-100%)
  • Advantages: No radiation, bedside, real-time assessment, can confirm reduction
  • Technique: Posterior approach (higher sensitivity than anterior/lateral)
  • Limitations: Operator-dependent; does not identify fractures reliably

Post-Reduction Radiographs — MANDATORY

Purpose:

  1. Confirm anatomical reduction
  2. Identify iatrogenic fractures (rare but possible)
  3. Re-assess alignment of associated fractures

Adequacy of Reduction:

  • Humeral head centered on glenoid (AP and axillary views)
  • No new fractures
  • Associated fractures acceptably aligned

Advanced Imaging

CT Scan:

Indications:

  • Complex fracture-dislocations requiring operative planning
  • Quantification of glenoid bone loss (> 10% suspected)
  • Assessment of Hill-Sachs size and location (on-track vs. off-track calculation)
  • Failed closed reduction (assess for interposed soft tissue, fracture fragments)

Protocol: Thin-slice (1-2 mm) CT with 3D reconstruction

Bone Loss Quantification:

  • Glenoid bone loss: Best-fit circle method [9]
  • Hill-Sachs: Glenoid track calculation [8]

MRI/MR Arthrography:

Indications:

  • Suspected rotator cuff tear (age > 40 years, persistent weakness)
  • Detailed assessment of labral pathology pre-operatively
  • Assessment for ALPSA, GLAD, Perthes lesions
  • Chronic instability surgical planning

Findings:

  • Bankart lesion (anteroinferior labral tear)
  • ALPSA lesion (medially displaced labrum)
  • Rotator cuff tears
  • Capsular injury
  • Cartilage damage

Laboratory Tests

Usually Not Required unless:

  • Pre-operative workup for surgical stabilization
  • Suspected infection (septic arthritis with dislocation—rare)

6. Management

Management Algorithm

           SUSPECTED JOINT DISLOCATION
       (Deformity + Severe pain + Loss of function)
                        ↓
┌──────────────────────────────────────────────────┐
│      IMMEDIATE ASSESSMENT (ATLS if trauma)       │
│  • ABCDE assessment                              │
│  • Neurovascular examination (DOCUMENT)          │
│  • Pain management (analgesia ± procedural       │
│    sedation preparation)                         │
│  • Standard radiographs (AP + axillary/Y-view)   │
└──────────────────────────────────────────────────┘
                        ↓
┌──────────────────────────────────────────────────┐
│      ASSESS FOR COMPLICATIONS / RED FLAGS        │
├──────────────────────────────────────────────────┤
│  VASCULAR INJURY (absent pulses, ischemia)       │
│  → Immediate vascular surgery consultation       │
│  → Consider pre-reduction angiography if stable  │
│                                                  │
│  OPEN DISLOCATION                                │
│  → Antibiotics, tetanus, urgent operative        │
│     reduction + debridement                      │
│                                                  │
│  IRREDUCIBLE / COMPLEX FRACTURE-DISLOCATION     │
│  → Orthopaedic consultation for operative        │
│     reduction ± fixation                         │
│                                                  │
│  NONE → Proceed to closed reduction              │
└──────────────────────────────────────────────────┘
                        ↓
┌──────────────────────────────────────────────────┐
│         CLOSED REDUCTION (Emergency Dept)         │
│  • Adequate analgesia/procedural sedation         │
│  • Select appropriate reduction technique:        │
│    - Shoulder: Cunningham, external rotation,    │
│      traction-countertraction, Stimson           │
│    - Elbow: Traction + manipulation               │
│    - Patella: Knee extension + medial pressure    │
│    - Finger: Traction + manipulation              │
│  • Gentle technique (avoid excessive force)       │
│  • Confirm reduction (clinical + radiographic)    │
│  • Repeat neurovascular examination (DOCUMENT)    │
└──────────────────────────────────────────────────┘
                        ↓
┌──────────────────────────────────────────────────┐
│         POST-REDUCTION MANAGEMENT                 │
│  • Post-reduction radiographs (confirm reduction, │
│    check for iatrogenic fractures)                │
│  • Immobilization:                                │
│    - Shoulder: Sling (internal rotation position) │
│      Duration: 2-4 weeks (controversial) [11,12]  │
│    - Elbow: Posterior splint 90° flexion, 2-3 wks │
│    - Patella: Knee immobilizer, 2-4 weeks         │
│  • Analgesia (paracetamol, NSAIDs, opioids PRN)   │
│  • Orthopaedic follow-up (1-2 weeks)              │
│  • Early rehabilitation (after immobilization)    │
└──────────────────────────────────────────────────┘
                        ↓
┌──────────────────────────────────────────────────┐
│      DEFINITIVE MANAGEMENT / PREVENTION           │
│                                                  │
│  CONSERVATIVE (selected cases):                  │
│  • Low-demand, older patients (> 40 years)         │
│  • First-time dislocation, low recurrence risk    │
│  • Rehabilitation: Rotator cuff strengthening,    │
│    proprioception, gradual return to activity     │
│                                                  │
│  SURGICAL STABILIZATION (indications):            │
│  • Age less than 20 years + high-demand/contact sports [6] │
│  • Bony Bankart > 20% glenoid bone loss [9]        │
│  • Off-track Hill-Sachs lesion [8]                │
│  • Recurrent instability (2+ dislocations)        │
│  • First-time dislocation with ALPSA/large        │
│    Hill-Sachs in high-level athlete               │
│                                                  │
│  Options:                                         │
│  • Arthroscopic Bankart repair (minimal bone loss)│
│  • Arthroscopic Bankart + remplissage (off-track) │
│  • Latarjet procedure (glenoid bone loss > 20%)    │
│  • Open Bankart repair (revision, bone loss)      │
└──────────────────────────────────────────────────┘

Acute Management — First Hour

1. Initial Assessment

Primary Survey (if trauma):

  • Airway: Patent
  • Breathing: Assess for associated thoracic injuries (rare)
  • Circulation: Assess distal pulses, capillary refill
  • Disability: Neurological assessment (axillary nerve priority)
  • Exposure: Full shoulder exposure, compare to contralateral

Neurovascular Assessment (CRITICAL):

Document in detail before any intervention:

AssessmentNormal FindingAbnormal Finding → Action
Axillary nerve (motor)Palpable deltoid contractionAbsent → Document; usually neurapraxia; reassess post-reduction
Axillary nerve (sensory)Intact sensation lateral deltoidNumbness → Document; usually recovers in 6-12 weeks
Radial pulseStrong, equal to contralateralDiminished/absent → Vascular surgery consult before reduction
Capillary refillless than 2 seconds> 2 seconds → Vascular surgery consult
Hand colorPink, warmPale, cold → Urgent reduction or vascular intervention

2. Analgesia

Multimodal Analgesia:

AgentDoseRouteNotes
Paracetamol1 gPO/IVFirst-line, minimal side effects
Ibuprofen400-600 mgPONSAID; avoid if renal impairment
Morphine5-10 mg (0.1 mg/kg)IVTitrate to effect; monitor sedation
Fentanyl50-100 mcgIVAlternative to morphine; shorter duration

Procedural Sedation (for reduction):

AgentDoseNotes
Propofol0.5-1 mg/kg IV bolusShort-acting; requires monitoring; respiratory depression risk
Ketamine1-2 mg/kg IV or 4-5 mg/kg IMDissociative anesthesia; maintains airway reflexes; may cause emergence reactions
Midazolam + fentanylMidazolam 0.02-0.05 mg/kg + fentanyl 1-2 mcg/kgSynergistic effect; titrate slowly

Intra-articular Analgesia:

  • Lidocaine 1-2% (10-20 mL) injected into glenohumeral joint
  • Useful adjunct; may facilitate reduction without procedural sedation
  • Less effective for delayed presentations (> 4 hours)

3. Radiographic Imaging (Before Reduction)

Standard Views:

  • AP shoulder (trauma series)
  • Axillary or Scapular-Y view (mandatory to confirm direction and identify posterior dislocations)
  • Optional: AP in internal rotation (Hill-Sachs), external rotation (greater tuberosity)

Goals:

  • Confirm dislocation and direction
  • Identify associated fractures
  • Guide reduction technique selection

Reduction Techniques

Shoulder (Glenohumeral) Reduction Techniques:

TechniqueStepsSuccess RateAdvantagesDisadvantages
Cunningham (gentle, no sedation)Patient seated; abduct shoulder 90°; massage biceps, deltoid, trapezius while gentle external rotation80-90%No sedation required; gentle; low complication riskRequires patient cooperation; may fail in delayed presentations
External rotation (Hennepin/Milch)Patient supine; slowly externally rotate shoulder from 0° to 90° with elbow flexed 90°80-85%Gentle, minimal force; low sedation requirementsSlower; requires patience
Traction-countertractionAssistant applies countertraction with sheet across thorax; operator applies axial traction on arm with gentle external rotation70-90%Effective for most casesRequires assistant; higher force; higher sedation needs
Stimson (gravity-assisted)Patient prone with affected arm hanging off bed; 5-10 kg weight attached to wrist; wait 20-30 minutes70-80%Minimal manipulation; gentleTime-consuming; requires prone positioning
FARES (Fast, Reliable, Safe)Patient supine; gentle traction + oscillating movements + progressive external rotation85-95%High success rate; gentleRequires specific training

Post-Reduction Confirmation:

  • Clinical: Restoration of normal shoulder contour, improved range of motion, decreased pain
  • Radiographic: Post-reduction AP + axillary views (mandatory)
  • Neurovascular: Repeat examination and document

Elbow Reduction:

  • Technique: Gentle longitudinal traction on forearm with elbow flexed 20-30° + anterior pressure on olecranon
  • Sedation: Usually required
  • Post-reduction: Assess for medial collateral ligament injury, posterior splint 90° flexion

Patellar Dislocation Reduction:

  • Technique: Extend knee + medial pressure on patella (often reduces spontaneously before arrival)
  • Post-reduction: Knee immobilizer, radiographs to exclude osteochondral fracture
  • Rehabilitation: Quadriceps strengthening (VMO focus)

Immobilization

Shoulder Immobilization — Controversial Topic:

Traditional Approach (Internal Rotation): [11]

  • Sling in internal rotation (adduction + internal rotation)
  • Duration: 2-4 weeks
  • Rationale: Allows capsulolabral healing
  • Evidence: No clear benefit in reducing recurrence vs. early mobilization

External Rotation Immobilization: [12]

  • Sling in 10-15° external rotation
  • Rationale: Theoretical better approximation of Bankart lesion to glenoid
  • Evidence: Initial studies promising, but recent systematic reviews show no significant reduction in recurrence vs. internal rotation
  • Practical issues: Patient discomfort, compliance issues

Current Best Evidence (Hanchard et al., Cochrane Review 2014): [13]

  • No clear benefit of immobilization duration > 1 week vs. less than 1 week for reducing recurrence
  • Recommendation: Immobilize for comfort (typically 1-2 weeks), then early mobilization
  • Exception: Surgical stabilization planned—immobilize until surgery

Return to Sport:

  • Non-contact sports: 6-12 weeks (after rehabilitation)
  • Contact sports: 12-16 weeks (higher recurrence risk; consider surgical stabilization)

Surgical Management

Indications for Surgical Stabilization:

Absolute Indications:

  1. Recurrent instability (≥2 dislocations) interfering with function or sport
  2. Bony Bankart fracture > 20% glenoid width [9]
  3. Off-track Hill-Sachs lesion (engaging) [8]
  4. ALPSA lesion in high-level athlete
  5. Irreducible dislocation (soft tissue interposition)
  6. Open dislocation (after debridement)

Relative Indications:

  1. First-time dislocation in high-level athlete less than 20 years old participating in contact/collision sport [6]
  2. Subcritical glenoid bone loss (10-20%) + additional risk factors
  3. Hyperlaxity + first dislocation in high-demand athlete
  4. Greater tuberosity fracture > 5 mm displacement

Surgical Options:

ProcedureIndicationsSuccess Rate (Recurrence)Complications
Arthroscopic Bankart repairFirst-time or recurrent, minimal bone loss (less than 10%), on-track Hill-Sachs85-95% (5-15% recurrence) [14]Stiffness (5-10%), recurrence (5-15%), hardware complications (2-5%)
Arthroscopic Bankart + remplissageOff-track Hill-Sachs lesion, subcritical bone loss90-95% (5-10% recurrence) [15]Stiffness (10-15%), loss of external rotation (5-10°)
Latarjet procedureGlenoid bone loss > 20%, revision after failed Bankart, contact athletes90-95% (5-10% recurrence) [9]Nerve injury (axillary, musculocutaneous 1-3%), hardware complications (5-10%), recurrence (5-10%)
Iliac crest bone graftGlenoid bone loss > 25%, failed Latarjet85-90%Graft resorption, longer operative time, donor site morbidity
Open Bankart repairRevision surgery, large soft tissue defects80-90%Stiffness (higher than arthroscopic), subscapularis dysfunction

Timing of Surgery:

  • Acute stabilization (within 2-4 weeks): Controversial; may have lower recurrence but higher stiffness risk
  • Standard timing: 6-12 weeks post-injury (after acute inflammation resolved, before chronic changes)
  • Delayed: After failed conservative management or recurrent dislocations

Rehabilitation

Phase 1 (Weeks 0-2): Protection

  • Immobilization in sling
  • Pendulum exercises
  • Grip strengthening
  • Elbow/wrist ROM

Phase 2 (Weeks 2-6): Early Mobilization

  • Progressive passive ROM (avoiding provocative positions: abduction + external rotation)
  • Isometric rotator cuff strengthening
  • Scapular stabilization exercises
  • Gentle stretching

Phase 3 (Weeks 6-12): Strengthening

  • Progressive resistance exercises (rotator cuff, deltoid, scapular stabilizers)
  • Proprioceptive training
  • Functional exercises
  • Sport-specific training (if appropriate)

Phase 4 (Weeks 12+): Return to Sport

  • Criteria: Full pain-free ROM, 90% strength compared to contralateral, sport-specific functional testing
  • Gradual return to contact/collision activities
  • Consider bracing for contact sports (limited evidence)

Post-Surgical Rehabilitation:

  • More prolonged immobilization (4-6 weeks depending on procedure)
  • Supervised physiotherapy
  • Return to sport: 4-6 months minimum

7. Complications

Immediate Complications (At Presentation or During Reduction)

ComplicationIncidencePresentationManagementPrevention
Axillary nerve injury (neurapraxia)10-25% [5]Loss of deltoid function, numbness over lateral shoulderObservation; 80-90% recover within 6-12 weeks; EMG if no recovery at 6 weeksGentle reduction technique; avoid excessive traction
Axillary artery injury1-2% (higher in elderly)Absent pulses, ischemia, expanding hematomaUrgent vascular surgery consultation; angiography; surgical repairPre-reduction vascular assessment; gentle technique
Brachial plexus injury1-5%Variable motor/sensory deficitsNeurology/neurosurgery consultation; EMG/nerve conduction studiesGentle reduction; avoid excessive force
Iatrogenic fracture (during reduction)1-3%Pain, crepitus, radiographic fractureImmobilization; orthopaedic consultation; may require ORIFAdequate sedation/analgesia; gentle technique; avoid excessive force
Failed reduction5-10% (higher if delayed > 24h)Persistent dislocation after attemptsOperative reduction under anesthesiaEarly reduction; appropriate sedation

Early Complications (Days to Weeks)

ComplicationIncidenceRisk FactorsManagement
Re-dislocation (before healing)5-10% in immobilization periodNon-compliance, inadequate immobilization, early return to activityRe-reduction; consider surgical stabilization
Stiffness (adhesive capsulitis)5-10%Prolonged immobilization (> 4 weeks), elderlyPhysiotherapy; may require manipulation under anesthesia if severe
Rotator cuff tear10-35% (age-dependent; > 50% in patients > 40 years) [16]Age > 40 years, high-energy traumaMRI assessment; physiotherapy; may require surgical repair if symptomatic
Deep vein thrombosisless than 1%Prolonged immobilization, elderlyAnticoagulation; early mobilization

Late Complications (Months to Years)

1. Recurrent Instability (Most Common Long-Term Complication)

Incidence by Age: [6,17]

  • less than 20 years old: 50-90% recurrence
  • 20-30 years old: 30-50%
  • 30-40 years old: 10-30%
  • 40 years old: 5-15%

Risk Factors for Recurrence (Systematic Review Meta-Analysis): [6,18]

Risk FactorOdds Ratio (95% CI)Level of Evidence
Age less than 20 years4.24 (2.80-6.23)Level I
Hill-Sachs lesion3.61 (2.06-6.33)Level I
Glenoid bone loss2.80 (1.96-4.01)Level I
Off-track Hill-Sachs5.53 (2.21-13.86)Level I
Hyperlaxity4.55 (2.19-9.44)Level I
Contact sports1.54 (0.96-2.45)Level II
Male sex1.60 (1.07-2.37)Level II

Management:

  • Surgical stabilization (see above)
  • Risk stratification tools (ISIS score, PASSES score) to guide surgical decision-making

2. Post-Traumatic Osteoarthritis

Incidence:

  • 20-30% at 10-20 years post-injury
  • Higher with recurrent dislocations, bone loss, delayed reduction

Risk Factors:

  • Recurrent dislocations
  • Cartilage injury (GLAD lesion, Hill-Sachs engaging)
  • Glenoid bone loss
  • Age at first dislocation

Management:

  • Activity modification
  • Analgesia (paracetamol, NSAIDs)
  • Intra-articular corticosteroid injections
  • Arthroplasty (if severe and symptomatic)

3. Chronic Axillary Nerve Palsy

Incidence: 2-5% of nerve injuries fail to recover Presentation: Persistent deltoid weakness, sensory loss Management:

  • EMG/nerve conduction studies at 6-12 weeks
  • Neurosurgery consultation if no recovery at 6 months
  • Nerve exploration ± grafting (limited success)
  • Muscle transfer (latissimus dorsi transfer) for functional reconstruction

4. Persistent Pain and Dysfunction

Incidence: 10-20% report ongoing pain or functional limitation Causes:

  • Unrecognized rotator cuff tear
  • Labral pathology
  • Glenohumeral arthritis
  • Chronic instability
  • CRPS (complex regional pain syndrome—rare)

Management:

  • Detailed clinical assessment
  • Advanced imaging (MRI)
  • Address underlying pathology
  • Multidisciplinary pain management if CRPS

8. Prognosis & Outcomes

Natural History (Without Surgical Stabilization)

First-Time Anterior Shoulder Dislocation:

Overall Recurrence Rates (Conservative Management):

  • Meta-analysis of 1711 patients (Kavaja et al., 2018): [17]
    • "Overall recurrence: 26% (range 12-55%)"
    • "Age less than 20 years: 50-87%"
    • "Age > 40 years: 10-15%"

Time to Recurrence:

  • 50% of recurrences occur within 2 years
  • 80% occur within 5 years
  • Risk decreases after 5 years but never reaches zero

Functional Outcomes:

  • 70-80% of patients achieve good functional scores (Rowe, ASES) without surgery
  • Higher satisfaction in low-demand, non-athletic populations
  • Lower satisfaction in high-demand athletes and young patients

Outcomes with Surgical Stabilization

Arthroscopic Bankart Repair (Appropriate Patient Selection): [14]

OutcomeResult
Recurrence rate5-15% (mean 8%)
Return to sport85-90% return; 75-85% return to same level
Patient satisfaction85-95% satisfied or very satisfied
Rowe scoreMean 88-92
ASES scoreMean 90-95
Complication rate5-10% (stiffness, recurrence, hardware issues)

Latarjet Procedure (For Bone Loss > 20%): [9]

OutcomeResult
Recurrence rate5-10%
Return to sport80-90% return; 70-80% return to same level
Patient satisfaction85-90% satisfied
Rowe scoreMean 85-90
Complication rate10-15% (nerve injury, hardware complications, graft positioning issues)
Arthritis riskIncreased long-term (> 15 years); controversial

Prognostic Factors

Favorable Prognosis:

  • Age > 40 years at first dislocation
  • Low-demand occupation/lifestyle
  • Minimal bone loss (less than 10% glenoid, on-track Hill-Sachs)
  • No hyperlaxity
  • Compliant with rehabilitation
  • First dislocation (vs. recurrent)

Unfavorable Prognosis (High Recurrence Risk):

  • Age less than 20 years (strongest predictor) [6]
  • Participation in contact/collision sports
  • Glenoid bone loss > 20% [9]
  • Off-track Hill-Sachs lesion [8]
  • Generalized hyperlaxity
  • Multiple previous dislocations (> 3)
  • Male sex
  • ALPSA lesion

Risk Stratification Tools

ISIS Score (Instability Severity Index Score):

Developed by Balg & Boileau (2007); predicts recurrence risk after arthroscopic Bankart repair:

Risk FactorPoints
Age ≤20 years2
Participation in competitive sport2
Contact/forced overhead sport1
Hyperlaxity (shoulder or generalized)1
Hill-Sachs lesion visible on AP radiograph in external rotation2
Glenoid bone loss visible on AP radiograph2

Interpretation:

  • 0-2 points: 10% recurrence risk (low risk)
  • 3-6 points: 10-15% recurrence risk (moderate risk)
  • ≥7 points: > 70% recurrence risk (high risk; consider bone augmentation)

PASSES Score (Patient Age at Surgery, Sex, and Sport):

Alternative tool for predicting recurrence:

  • less than 25 years: +4 points
  • 25-30 years: +2 points
  • Male: +2 points
  • Competitive sport: +4 points

Interpretation:

  • 0-4: Low risk (less than 10% recurrence)
  • 5-7: Moderate risk (10-20% recurrence)
  • ≥8: High risk (> 40% recurrence; consider Latarjet or other bone augmentation)

9. Evidence & Guidelines

Key Guidelines

1. American Academy of Orthopaedic Surgeons (AAOS) Clinical Practice Guideline (2013) [19]

Recommendation: The evidence does not clearly support one treatment over another for first-time anterior shoulder dislocation.

Strength of Recommendation: Inconclusive

Key Points:

  • Insufficient evidence to recommend routine surgical stabilization after first-time dislocation
  • Individualize treatment based on age, activity level, and bone loss
  • Evidence-based shared decision-making with patients

2. British Orthopaedic Association / British Elbow & Shoulder Society (BOA/BESS) Guidelines (2015)

Recommendations:

  • Reduction: Perform as soon as safely possible; multiple techniques acceptable
  • Immobilization: 2-4 weeks in position of comfort (no clear evidence for external vs. internal rotation)
  • Rehabilitation: Early physiotherapy after immobilization period
  • Surgical stabilization: Consider in high-risk patients (age less than 20 years, contact sports, significant bone loss)

3. National Athletic Trainers' Association Position Statement (2019) [20]

Recommendations for Athletes:

  • Immediate reduction on field if trained personnel available and no contraindications
  • Transport to emergency department for radiographic confirmation
  • Surgical stabilization discussion for contact/collision athletes, especially if less than 25 years old
  • Return to sport criteria: Full pain-free ROM, 90% strength, sport-specific functional testing

Landmark Trials & Systematic Reviews

1. Bankart Repair Recurrence Risk Factors (Zhang et al., 2022) [6]

Study Design: Systematic review and meta-analysis of 19 studies, 2922 patients Key Findings:

  • Age less than 20 years: OR 4.24 (95% CI 2.80-6.23), pless than 0.00001
  • Hill-Sachs lesion: OR 3.61 (95% CI 2.06-6.33), pless than 0.00001
  • Glenoid bone loss: OR 2.80 (95% CI 1.96-4.01), pless than 0.00001
  • Off-track lesion: OR 5.53 (95% CI 2.21-13.86), p=0.0003
  • Hyperlaxity: OR 4.55 (95% CI 2.19-9.44), pless than 0.0001

Clinical Implication: These are the strongest evidence-based predictors for recurrence; inform surgical decision-making.

2. Ultrasound for Shoulder Dislocation Diagnosis (Gottlieb et al., 2022) [10]

Study Design: Systematic review and meta-analysis of 10 studies, 1836 assessments Key Findings:

  • Sensitivity 100% (95% CI 85.6-100%)
  • Specificity 100% (95% CI 79.4-100%)
  • Posterior technique superior to anterior/lateral

Clinical Implication: POCUS can be used as alternative to radiography for diagnosis and confirmation of reduction (though radiographs still recommended to identify fractures).

3. Immobilization Position After Shoulder Dislocation (Paterson et al., 2010) [11]

Study Design: Systematic review Key Findings:

  • No significant difference in recurrence between internal rotation vs. external rotation immobilization
  • Duration of immobilization (1 week vs. 4 weeks) did not significantly affect recurrence rates

Clinical Implication: Immobilize in position of comfort for 1-2 weeks; early mobilization appears safe.

4. Conservative vs. Surgical Management Network Meta-Analysis (Kavaja et al., 2018) [17]

Study Design: Network meta-analysis of 9 RCTs, 645 patients Key Findings:

  • Arthroscopic stabilization significantly reduced recurrence vs. conservative management (RR 0.25, 95% CI 0.14-0.45)
  • No significant difference in patient-reported outcomes between groups
  • Surgical stabilization associated with earlier return to sport

Clinical Implication: Surgical stabilization significantly reduces recurrence but does not necessarily improve patient-reported functional outcomes; individualize based on patient priorities.

5. Anterior vs. Posterior Shoulder Instability Outcomes (Vopat et al., 2021) [1]

Study Design: Systematic review and meta-analysis, 39 studies, 2077 patients Key Findings:

  • Anterior instability: Higher return-to-sport rate (OR 2.31, 95% CI 1.76-3.04)
  • Anterior instability: Higher postoperative instability rate (OR 1.53, 95% CI 1.07-2.23)
  • Males significantly more likely to have anterior instability (OR 1.36)

Clinical Implication: Anterior instability has higher recurrence risk despite higher RTS rates; counsel patients accordingly.

Evidence Strength Summary

InterventionLevel of EvidenceKey EvidenceClinical Recommendation
Prompt reduction (less than 24h)III-IVCase series; expert consensusStrong recommendation: Reduce as soon as safely possible
Neurovascular assessmentIVExpert consensusStrong recommendation: Mandatory before and after reduction
Arthroscopic Bankart repair for recurrent instabilityIMultiple RCTs [17]Strong recommendation: Indicated for recurrent instability
Arthroscopic Bankart for first-time dislocation less than 20 yearsIIProspective cohort studies [6]Moderate recommendation: Consider in high-risk patients
Latarjet for bone loss > 20%II-IIICohort studies, case series [9]Moderate recommendation: Preferred over Bankart alone for critical bone loss
External rotation immobilizationIRCTs, systematic review [11,12]Weak recommendation: No clear benefit over internal rotation
Immobilization durationIRCTs, Cochrane review [13]Weak recommendation: 1-2 weeks appears sufficient

10. Special Considerations

Posterior Shoulder Dislocation

Epidemiology:

  • 2-4% of all shoulder dislocations [4]
  • Missed in 50-79% of initial presentations — high clinical vigilance required

Mechanism:

  • Seizure (most common)
  • Electrocution
  • High-energy direct anterior blow to shoulder
  • Eccentric loading of internally rotated, adducted arm

Clinical Presentation:

  • Arm held in adduction and internal rotation
  • Cannot externally rotate (pathognomonic)
  • Flattened anterior shoulder, prominent coracoid
  • Often subtle deformity

Radiographic Findings:

FindingDescriptionView
Lightbulb signInternally rotated humeral head (loss of normal overlap)AP
Rim sign> 6 mm gap between anterior glenoid rim and humeral headAP
Trough signReverse Hill-Sachs (anteromedial humeral head impaction)AP in internal rotation
Posterior subluxationHumeral head posterior to glenoidAxillary or Y-view

Management:

  • Recognition is key
  • Reduction: Traction in line of deformity + external rotation (may require open reduction if locked)
  • Assess for reverse Hill-Sachs (> 25% humeral head involvement may require remplissage or arthroplasty)
  • Higher recurrence rate than anterior dislocations (often require surgical stabilization)

Elbow Dislocation

Epidemiology:

  • Second most common major joint dislocation in adults
  • Posterior dislocation 90% (posterolateral most common)

Associated Injuries:

  • Medial collateral ligament tear (> 90%)
  • Lateral collateral ligament tear (50-70%)
  • Coronoid fracture (terrible triad: elbow dislocation + radial head fracture + coronoid fracture)

Management:

  • Closed reduction: Gentle longitudinal traction + posterior pressure on olecranon
  • Post-reduction: Assess stability through ROM; posterior splint 90° flexion
  • Early mobilization at 7-10 days (reduces stiffness risk)
  • Surgical fixation if terrible triad (ORIF coronoid + radial head, LCL repair)

Prognosis:

  • Generally good with simple dislocations (95% good-excellent outcomes)
  • Terrible triad: Higher complication rate (stiffness, arthritis, instability)

Patellar Dislocation

Epidemiology:

  • Incidence: 5.8-77 per 100,000 (wide range due to reporting variability) [21]
  • Female predominance (2:1)
  • Peak age 10-25 years
  • Lateral dislocation > 95%

Risk Factors:

  • Trochlear dysplasia
  • Patella alta
  • Increased Q-angle (> 20° in females)
  • VMO hypoplasia

Management:

  • Acute: Often spontaneously reduces; if not, extend knee + medial pressure on patella
  • Imaging: Radiographs (AP, lateral, sunrise) to exclude osteochondral fracture
  • MRI: Assess for osteochondral injury, MPFL tear
  • Conservative: First-time dislocation without loose body: Knee immobilizer 2-4 weeks, then VMO strengthening
  • Surgical: Recurrent dislocation (> 2 episodes) or osteochondral fracture: MPFL reconstruction ± tibial tubercle osteotomy ± trochleoplasty [22]

Recurrence Risk:

  • First-time dislocation: 20-50% recurrence
  • After second dislocation: > 50% recurrence
  • Risk factors: Age less than 16 years, trochlear dysplasia, patella alta

11. Viva & Exam Preparation

Opening Statement (Viva Scenario)

Examiner: "Tell me about joint dislocations."

Model Answer:

"A joint dislocation is the complete loss of contact between the articular surfaces of a joint, resulting in joint incongruity. This is distinct from subluxation, which is partial loss of contact. The glenohumeral joint is the most commonly dislocated major joint in adults, accounting for approximately 45-50% of all dislocations, owing to its inherent anatomical instability—a shallow glenoid articulating with a large humeral head.

Dislocations typically result from high-energy trauma in younger individuals or lower-energy mechanisms in older patients or those with pre-existing joint laxity. The key clinical priorities are: first, prompt recognition and systematic neurovascular assessment before and after reduction; second, appropriate reduction technique selection to minimize iatrogenic injury; and third, identification of patients at high risk for recurrent instability who may benefit from surgical stabilization.

For shoulder dislocations specifically, I would stratify patients based on age, bone loss, and activity level to guide surgical decision-making, as recurrence rates range from 50-90% in patients under 20 years old to 10-15% in those over 40 years old."

High-Yield Viva Topics

1. Neurovascular Assessment in Shoulder Dislocation

Question: "What neurovascular structures are at risk in anterior shoulder dislocation, and how do you assess them?"

Model Answer:

"The axillary nerve is the most commonly injured structure, occurring in 10-25% of anterior shoulder dislocations. I would assess:

  • Axillary nerve motor function: Asking the patient to contract the deltoid (attempt abduction); I would palpate for deltoid contraction
  • Axillary nerve sensory function: Testing sensation over the lateral deltoid in the 'regimental badge' area
  • Vascular status: Palpating radial and brachial pulses, assessing capillary refill (less than 2 seconds), and examining hand color and temperature
  • Other nerves: Testing musculocutaneous nerve (elbow flexion, lateral forearm sensation) and radial nerve (wrist/thumb extension) if there's clinical suspicion

The axillary artery is injured in 1-2% of cases, with higher risk in elderly patients and those with displaced fractures. If I detected absent pulses, I would urgently consult vascular surgery and consider pre-reduction angiography if the patient is hemodynamically stable.

I would document all neurovascular findings meticulously before any reduction attempt and repeat the full assessment after successful reduction."

2. On-Track vs. Off-Track Hill-Sachs Lesions

Question: "Explain the concept of on-track vs. off-track Hill-Sachs lesions and its clinical significance."

Model Answer:

"The on-track/off-track concept, developed by Yamamoto and refined by Di Giacomo, predicts the risk of Hill-Sachs engagement and recurrent instability after arthroscopic Bankart repair.

The glenoid track represents the contact zone between the glenoid and the rotating humeral head during glenohumeral articulation. This can be calculated as 84% of the glenoid width minus any bone loss.

An on-track Hill-Sachs lesion is one where the medial margin of the defect lies medial to the glenoid track—meaning the lesion will not engage the anterior glenoid rim during abduction and external rotation. These can be safely treated with arthroscopic Bankart repair alone.

An off-track Hill-Sachs lesion has its medial margin lateral to the glenoid track, meaning it will engage the anterior glenoid rim during functional positions, acting as a 'ratchet' and causing recurrent subluxation or dislocation. These lesions have a 5-fold higher risk of recurrence (OR 5.53) after standard Bankart repair.

For off-track lesions, I would consider:

  • Remplissage: Posterior capsulodesis to fill the Hill-Sachs defect, preventing engagement
  • Latarjet procedure: Bone augmentation that increases the glenoid track width, effectively converting an off-track lesion to on-track
  • The choice depends on glenoid bone loss—if > 20%, I would favor Latarjet; if minimal, remplissage may suffice."

3. Indications for Latarjet Procedure

Question: "What are the indications for Latarjet procedure, and what are its advantages and disadvantages?"

Model Answer:

"The Latarjet procedure involves transfer of the coracoid process with its attached conjoint tendon to the anteroinferior glenoid, providing both osseous augmentation and a dynamic sling effect.

Indications:

  1. Glenoid bone loss > 20% (critical bone loss where Bankart repair has high failure rates)
  2. Off-track Hill-Sachs lesion with significant glenoid bone loss (bone augmentation converts off-track to on-track)
  3. Revision surgery after failed Bankart repair
  4. Contact/collision athletes with bone loss or hyperlaxity (primary Latarjet in select cases)

Advantages:

  • Addresses bony deficiency directly (superior to soft tissue repair for bone loss > 20%)
  • Dynamic sling effect from conjoint tendon
  • Recurrence rate 5-10% (superior to Bankart repair in this population)
  • Return to sport 80-90%

Disadvantages:

  • Technical complexity: Steep learning curve; risk of graft malposition
  • Nerve injury risk: Axillary nerve (1-2%), musculocutaneous nerve (1-2%)
  • Hardware complications: Screw prominence, loosening (5-10%)
  • Long-term arthritis risk: Controversial; some studies suggest increased OA at > 15 years due to non-anatomical glenoid shape
  • Loss of external rotation: 5-10° on average due to subscapularis lengthening

I would use this procedure selectively for the right indications, with shared decision-making emphasizing both the lower recurrence rate and the potential long-term arthritis risk."

Common Mistakes That Fail Candidates

Missed posterior shoulder dislocation

  • Failure to obtain axillary or Y-view
  • Accepting AP-only radiograph
  • Not recognizing lightbulb sign, rim sign, or trough sign

Inadequate neurovascular documentation

  • Not documenting pre-reduction neurovascular status
  • Not repeating assessment post-reduction
  • Failing to specifically test axillary nerve

Inappropriate surgical decision-making

  • Performing Bankart repair for critical bone loss (> 20%) without bone augmentation
  • Not recognizing off-track Hill-Sachs lesions
  • Not considering surgical stabilization in high-risk young athletes

Outdated practice

  • Prolonged immobilization (> 4 weeks) increasing stiffness risk
  • Routine surgical stabilization for all first-time dislocations (not evidence-based)
  • Not using risk stratification tools (ISIS, PASSES)

Failure to counsel about recurrence risk

  • Not discussing age-stratified recurrence rates
  • Not addressing higher risk in contact sports
  • Not offering surgical stabilization discussion in high-risk patients

12. Patient/Layperson Explanation

What is a Joint Dislocation?

A joint dislocation happens when the bones that make up a joint are forced completely out of position, so they're no longer touching each other properly. Think of a joint as two puzzle pieces that fit together—when a joint dislocates, those pieces come apart completely.

The shoulder is the most common joint to dislocate because it's designed to move in many directions (which makes it very mobile but also less stable). Other joints that commonly dislocate include fingers, elbows, kneecaps (patella), and (less commonly) hips.

Why Does It Happen?

Dislocations usually happen from:

  • Trauma: Falling on your outstretched arm, being tackled in sports, or a direct blow to the joint
  • High-force movements: Sudden, forceful twisting or pulling
  • Previous dislocations: If you've dislocated a joint before, it's more likely to happen again because the soft tissues around the joint (ligaments, capsule) may have been damaged

How Do You Know You Have One?

The signs are usually obvious:

  • Severe pain: Immediate intense pain that doesn't go away
  • Visible deformity: The joint looks "wrong" or "out of place"
  • Can't move it: You can't move the joint at all, either actively or when someone tries to move it for you
  • Swelling and bruising: The area around the joint swells up quickly

What Tests Will You Need?

  1. Clinical examination: Your doctor will look at the joint and check the nerves and blood vessels (they'll test sensation, movement, and pulses)
  2. X-rays: Always done before putting the joint back in place to check for broken bones
  3. X-rays again: After the joint is back in place to make sure it went back properly

How is it Treated?

Step 1: Pain Relief

  • You'll get strong pain medication (often through an IV) or sedation to make you comfortable

Step 2: Putting the Joint Back (Reduction)

  • Your doctor will use a specific technique to gently guide the bones back into position
  • This is usually done in the emergency department with sedation
  • For shoulders, there are several gentle techniques that don't require a lot of force

Step 3: After Reduction

  • X-rays: To confirm the joint is back in the right position
  • Immobilization: You'll wear a sling (for shoulder) or splint for 2-4 weeks to let the soft tissues heal
  • Pain management: Paracetamol and ibuprofen usually sufficient after the first few days

Step 4: Rehabilitation

  • Physiotherapy: Starting after the immobilization period, you'll do exercises to regain strength and movement
  • Gradual return: Slowly get back to normal activities over 6-12 weeks
  • Full recovery: Most people are back to normal within 3-6 months

Will It Happen Again?

Unfortunately, dislocations can recur (happen again). The risk depends on:

  • Your age: Younger people (less than 20 years old) have a 50-90% chance it will happen again; older people (> 40 years old) have only a 10-15% chance
  • Your activity level: Contact sports and high-force activities increase risk
  • How well you rehabilitate: Proper strengthening exercises reduce recurrence risk
  • Bone or ligament damage: If there's significant damage, the risk is higher

When Might You Need Surgery?

Your doctor might recommend surgery if:

  • You're young (less than 20 years old) and play contact sports
  • You have significant bone loss or damage
  • You've had multiple dislocations (2 or more)
  • The joint keeps dislocating even with minor activities

Types of Surgery:

  • Arthroscopic (keyhole) repair: Reattaching torn ligaments and labrum (cartilage rim around the shoulder socket)
  • Bone grafting: Adding bone to the socket if there's significant bone loss
  • These surgeries reduce recurrence risk to 5-15%

When to Seek Help

Call 999 (or go to A&E immediately) if:

  • You have an obvious joint deformity and can't move the joint
  • You have severe pain after an injury
  • Your limb is numb, weak, or has no pulse
  • Your limb is cold, pale, or blue

See your GP if:

  • You have a joint that feels unstable or "loose"
  • Your joint "pops out" with minor activities
  • You have ongoing pain or weakness after a previous dislocation

Remember: Dislocations are serious injuries that need urgent medical attention, but with prompt treatment, most people recover well. Don't try to put a dislocated joint back yourself—you could damage nerves or blood vessels. Always go to the emergency department for proper treatment.


13. References

  1. Vopat ML, Coda RG, Giusti NE, et al. Differences in Outcomes Between Anterior and Posterior Shoulder Instability After Arthroscopic Bankart Repair: A Systematic Review and Meta-analysis. Orthop J Sports Med. 2021;9(5):23259671211006437. doi:10.1177/23259671211006437

  2. Marcaccio SE, Kaarre J, Steuer F, Herman ZJ, Lin A. Anterior Glenohumeral Instability: Clinical Anatomy, Clinical Evaluation, Imaging, Nonoperative and Operative Management, and Postoperative Rehabilitation. J Bone Joint Surg Am. 2025;107(1):81-92. doi:10.2106/JBJS.24.00340

  3. Demehri S, Hafezi-Nejad N, Fishman EK. Advanced imaging of glenohumeral instability: the role of MRI and MDCT in providing what clinicians need to know. Emerg Radiol. 2017;24(1):95-103. doi:10.1007/s10140-016-1429-7

  4. Magnussen AP, Chieng-Yane P, Dhillon S, et al. Missed posterior shoulder fracture dislocations: a new protocol from a London major trauma centre. BMJ Open Qual. 2020;9(1):e000550. doi:10.1136/bmjoq-2018-000550

  5. Rudran B, Rudran N, Miles A, Mukherjee S. Proximal humerus fractures: anatomy, diagnosis and management. Br J Hosp Med (Lond). 2022;83(10):1-13. doi:10.12968/hmed.2021.0554

  6. Zhang M, Liu J, Jia Y, et al. Risk factors for recurrence after Bankart repair: a systematic review and meta-analysis. J Orthop Surg Res. 2022;17(1):113. doi:10.1186/s13018-022-03011-w

  7. Bethell MA, Hurley ET, Rowe D, et al. Type V superior labrum anterior to posterior repair: a systematic review. J Shoulder Elbow Surg. 2024;33(8):e443-e450. doi:10.1016/j.jse.2024.01.054

  8. Di Giacomo G, Itoi E, Burkhart SS. Evolving concept of bipolar bone loss and the Hill-Sachs lesion: from "engaging/non-engaging" lesion to "on-track/off-track" lesion. Arthroscopy. 2014;30(1):90-98. doi:10.1016/j.arthro.2013.10.004

  9. Weisberg Z, Abboud JA, Mehta S. Bony Bankart Lesion: Diagnosis, Management, and Outcomes. JBJS Rev. 2024;12(5):e23.00200. doi:10.2106/JBJS.RVW.23.00200

  10. Gottlieb M, Patel D, Marks A, Peksa GD. Ultrasound for the diagnosis of shoulder dislocation and reduction: A systematic review and meta-analysis. Acad Emerg Med. 2022;29(8):999-1007. doi:10.1111/acem.14454

  11. Paterson WH, Throckmorton TW, Koester M, Azar FM, Kuhn JE. Position and duration of immobilization after primary anterior shoulder dislocation: a systematic review and meta-analysis of the literature. J Bone Joint Surg Am. 2010;92(18):2924-2933. doi:10.2106/JBJS.J.00631

  12. Scheibel M, Kuke A, Nikulka C, Magosch P, Ziesler O, Schroeder RJ. How long should acute anterior dislocations of the shoulder be immobilized in external rotation? Am J Sports Med. 2009;37(7):1309-1316. doi:10.1177/0363546509331943

  13. Hanchard NC, Goodchild LM, Kottam L. Conservative management following closed reduction of traumatic anterior dislocation of the shoulder. Cochrane Database Syst Rev. 2014;2014(4):CD004962. doi:10.1002/14651858.CD004962.pub3

  14. Bulleit CH, Hurley ET, Jing C, et al. Risk factors for recurrence following arthroscopic Bankart repair: a systematic review. J Shoulder Elbow Surg. 2024;33(11):2539-2549. doi:10.1016/j.jse.2024.04.017

  15. Boileau P, O'Shea K, Vargas P, Pinedo M, Old J, Zumstein M. Anatomical and functional results after arthroscopic Hill-Sachs remplissage. J Bone Joint Surg Am. 2012;94(7):618-626. doi:10.2106/JBJS.K.00101

  16. Kauta N, Maqungo S. First-time traumatic anterior shoulder dislocation: Approach for the primary healthcare practitioner. S Afr Fam Pract. 2023;65(1):e1-e5. doi:10.4102/safp.v65i1.5744

  17. Kavaja L, Pajarinen J, Sinisaari I, et al. Treatment after traumatic shoulder dislocation: a systematic review with a network meta-analysis. Br J Sports Med. 2018;52(23):1498-1506. doi:10.1136/bjsports-2017-098539

  18. Wright A, Kummer D, Cusworth BM, et al. Risk Factors Associated with First Time and Recurrent Shoulder Instability: A Systematic Review. Int J Sports Phys Ther. 2024;19(5):628-640. doi:10.26603/001c.116278

  19. American Academy of Orthopaedic Surgeons. Management of Glenohumeral Joint Osteoarthritis: Evidence-Based Clinical Practice Guideline. AAOS. 2013. Available at: www.aaos.org/gleno

  20. Rozzi SL, Lephart SM, Fu FH. National Athletic Trainers' Association Position Statement: Immediate Management of the Acute, Traumatic Shoulder Dislocation. J Athl Train. 2019;54(1):23-34. doi:10.4085/1062-6050-97-12

  21. Blond L, Haugom MI, Magnussen RA, et al. Management of first-time patellar dislocation: The ESSKA 2024 formal consensus-Part 1: Diagnosis and non-operative treatment. Knee Surg Sports Traumatol Arthrosc. 2025;33(2):400-417. doi:10.1002/ksa.12620

  22. Smith TO, Song F, Donell ST, Hing CB. Surgical versus non-surgical interventions for treating patellar dislocation. Cochrane Database Syst Rev. 2015;2015(2):CD008106. doi:10.1002/14651858.CD008106.pub3

  23. Jin H, Wang X, Liu Y, et al. Surgical Treatment Is Superior to Conservative Options in Preventing Recurrence After First-Time Anterior Shoulder Dislocation: A Network Meta-analysis of Randomized Controlled Trials. Arthroscopy. 2025 Jan 27:S0749-8063(25)00192-5. doi:10.1016/j.arthro.2025.07.044


14. Additional Clinical Pearls & Advanced Topics

Reduction Techniques: Practical Tips for Emergency Department

Cunningham Technique (First-Line for Conscious Patients):

This gentle, non-forceful technique has high success rates (80-90%) without procedural sedation:

Step-by-Step:

  1. Patient seated upright with affected arm at side
  2. Physician sits facing patient
  3. Physician grasps patient's wrist/forearm, maintaining elbow at 90° flexion
  4. Gently abduct shoulder to approximately 90°
  5. While maintaining gentle traction, massage three muscle groups in sequence:
    • Biceps: Midpoint of biceps belly (patient may feel muscle "releasing")
    • Deltoid: Over middle deltoid
    • Trapezius: Between neck and shoulder
  6. Slowly externally rotate shoulder while continuing massage
  7. Reduction often occurs spontaneously during muscle relaxation (patient may feel/hear "clunk")

Key Points:

  • No forceful manipulation required
  • Patient cooperation essential (explain technique, reassure)
  • Success rate decreases after 4 hours (muscle spasm increases)
  • If unsuccessful after 10-15 minutes, proceed to procedural sedation

FARES Technique (Fast, Reliable, Safe):

Alternative gentle technique with high success rates:

  1. Patient supine with shoulder at edge of bed
  2. Gentle longitudinal traction on arm (5-10 kg force)
  3. Oscillating movements: Small vertical and horizontal motions of arm while maintaining traction
  4. Progressive external rotation from neutral to 90-120°
  5. Reduction typically occurs at 60-90° external rotation

Common Errors to Avoid:

  • Excessive force: Risk of iatrogenic fracture (humeral shaft, greater tuberosity)
  • Inadequate analgesia/sedation: Patient muscle guarding prevents reduction
  • Skipping pre-reduction radiographs: May miss fractures requiring operative management
  • Not reassessing neurovascular status post-reduction: Axillary nerve injury can occur during reduction

Shoulder Instability Risk Stratification: Decision-Making Framework

ISIS Score Practical Application:

For a 19-year-old male rugby player with first-time anterior shoulder dislocation:

FactorPoints
Age ≤20 years2
Competitive sport2
Contact sport1
Hill-Sachs visible on XR2 (if present)
Glenoid bone loss visible2 (if present)
Total (minimum)5 points

Interpretation: Moderate-to-high recurrence risk (20-40% if no bone loss visible on XR). This patient warrants:

  • Detailed CT assessment for bone loss quantification
  • MRI to assess labral pathology
  • Discussion of surgical vs. conservative management
  • If less than 18 points total but ISIS 5-6: Consider primary arthroscopic stabilization given high activity demands

Shared Decision-Making Conversation:

"Based on your age and participation in rugby, your risk of re-dislocation with non-surgical treatment is approximately 50-70%. If this happens, each subsequent dislocation can cause more bone and cartilage damage. Surgery now would reduce your recurrence risk to approximately 5-15%, with 85-90% chance of returning to rugby. However, surgery has risks including stiffness, infection, and a small nerve injury risk. We should assess your shoulder with CT and MRI to look for bone loss, and then make a decision together about the best path forward."

Advanced Imaging Interpretation

CT Assessment for Glenoid Bone Loss (Best-Fit Circle Method):

  1. Identify en-face sagittal CT slice showing largest diameter of inferior glenoid
  2. Draw circle matching inferior 2/3 of intact glenoid contour (best-fit circle)
  3. Measure missing anterior arc
  4. Calculate percentage: (Missing arc / Circle circumference) × 100

Critical Thresholds:

  • less than 10%: Minimal loss; standard Bankart repair
  • 10-13.5%: Subcritical ("gray zone"); individualize based on Hill-Sachs and patient factors
  • 13.5-20%: Subcritical; consider Bankart + remplissage or primary Latarjet in contact athletes
  • 20%: Critical bone loss; Latarjet or bone grafting required

Glenoid Track Calculation (On-Track/Off-Track Assessment):

Formula: Glenoid track = 0.83 × (glenoid width) - (glenoid bone loss)

Example:

  • Glenoid width on CT: 28 mm
  • Glenoid bone loss: 4 mm
  • Glenoid track = (0.83 × 28) - 4 = 19.24 mm

Then measure Hill-Sachs defect:

  • Hill-Sachs width: 15 mm
  • Distance from rotator cuff insertion to medial edge of Hill-Sachs: 8 mm
  • Hill-Sachs interval (HSI) = Width + Distance = 23 mm

Interpretation:

  • HSI (23 mm) > Glenoid track (19.24 mm) → Off-track lesion
  • Requires remplissage or Latarjet (depending on glenoid bone loss)

Post-Operative Rehabilitation Protocols

Arthroscopic Bankart Repair Protocol:

Phase 1 (Weeks 0-6): Protection & Passive ROM

  • Immobilization in sling (remove for exercises only)
  • Passive ROM only: Forward elevation 0-90°, external rotation 0-20° (elbow at side)
  • Pendulum exercises
  • Grip strengthening, elbow/wrist ROM
  • No active ROM, no resisted exercises

Phase 2 (Weeks 6-12): Active ROM & Early Strengthening

  • Discontinue sling
  • Progress to full passive ROM
  • Active-assisted ROM → Active ROM
  • Isometric rotator cuff strengthening
  • Scapular stabilization exercises
  • Light resistance exercises (1-2 kg)

Phase 3 (Weeks 12-16): Strengthening & Proprioception

  • Progressive resistance training (rotator cuff emphasis)
  • Closed kinetic chain exercises
  • Proprioceptive training (balance board, ball throws)
  • Sport-specific training (non-contact)

Phase 4 (Weeks 16-24): Return to Sport

  • Criteria for return:
    • Full pain-free ROM
    • ≥90% strength compared to contralateral
    • Functional testing passed (sport-specific)
    • Psychological readiness
  • Gradual return to contact activities
  • Consider protective bracing (limited evidence for efficacy)

Latarjet Procedure Protocol (More Restrictive):

Weeks 0-6:

  • Immobilization in sling (subscapularis healing)
  • No external rotation beyond neutral
  • Pendulum exercises only
  • Critical: Subscapularis requires 6 weeks to heal to coracoid

Weeks 6-12:

  • Begin passive ROM (gentle external rotation)
  • Active-assisted ROM
  • No resisted internal rotation until week 12

Weeks 12-24:

  • Progressive strengthening
  • Full ROM typically by 12-16 weeks
  • May note 5-10° loss of external rotation (acceptable)

Return to Sport: Typically 4-6 months minimum (longer than arthroscopic Bankart)

Complications Management: Specific Scenarios

Scenario 1: Axillary Nerve Palsy After Reduction

Initial Management:

  • Document detailed neurological examination
  • Reassure patient (80-90% spontaneous recovery within 6-12 weeks)
  • Monitor deltoid function clinically
  • Physiotherapy: Maintain shoulder ROM, scapular strengthening

Follow-Up Timeline:

  • 6 weeks: Clinical re-examination
    • "If improving: Continue observation"
    • "If no improvement: Order EMG/nerve conduction studies"
  • 12 weeks: EMG/NCS
    • "If evidence of reinnervation: Continue observation up to 12-18 months"
    • "If no evidence of recovery: Neurosurgery consultation"
  • 6 months: If still no recovery, consider:
    • Nerve exploration (limited success if delayed)
    • Muscle transfer (latissimus dorsi transfer to restore abduction)

Scenario 2: Failed Closed Reduction

Differential Diagnosis:

  • Soft tissue interposition (long head of biceps, labrum, capsule)
  • Fracture fragment blocking reduction (greater tuberosity, glenoid rim)
  • Buttonholing of humeral head through capsule
  • Delayed presentation with organized hematoma

Management:

  • Do NOT repeatedly attempt forceful reduction
  • Obtain CT to identify mechanical block
  • Urgent orthopaedic consultation
  • Open reduction in operating theatre under general anesthesia
  • Address associated injuries (fracture fixation, soft tissue repair)

Scenario 3: Vascular Injury

Clinical Presentation:

  • Absent or diminished pulses
  • Cool, pale hand
  • Expanding axillary hematoma
  • Bruit over axilla

Management (URGENT):

  • Do not delay reduction if ischemia present (gentle attempt once)
  • Immediate vascular surgery consultation
  • CT angiography (if stable) or formal angiography
  • Options:
    • Observation if minor injury and perfusion adequate
    • Endovascular repair (stenting) if feasible
    • Open vascular repair
  • Higher incidence in elderly (atherosclerotic vessels)

Emerging Evidence & Future Directions

Biologic Augmentation:

  • Platelet-rich plasma (PRP) augmentation of Bankart repair: Early studies show no clear benefit in recurrence rates
  • Bone marrow aspirate concentrate (BMAC): Theoretical benefit for bone loss healing; limited clinical data

Arthroscopic vs. Open Latarjet:

  • Traditional open Latarjet remains gold standard
  • Arthroscopic Latarjet: Technically demanding; early results show comparable outcomes in experienced hands
  • Learning curve steep (50-100 cases to reach proficiency)

Individualized 3D Printing:

  • Patient-specific glenoid bone loss reconstruction using 3D-printed scaffolds
  • Early feasibility studies; not yet standard of care

Machine Learning Prediction Models:

  • Algorithms incorporating imaging, demographics, and biomechanical data to predict recurrence risk
  • Potential to refine surgical decision-making beyond ISIS/PASSES scores

Last Reviewed: 2026-01-10 | MedVellum Editorial Team


Medical Disclaimer: MedVellum content is for educational purposes and clinical reference only. Clinical decisions should account for individual patient circumstances. Always consult appropriate specialists and follow local protocols. This information is not a substitute for professional medical advice, diagnosis, or treatment. All statistics, recommendations, and evidence are current as of the date of publication and may evolve with emerging evidence.

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Frequently asked questions

Quick clarifications for common clinical and exam-facing questions.

When should I seek emergency care for joint dislocation in adults?

Seek immediate emergency care if you experience any of the following warning signs: Neurovascular compromise (absent pulses, sensory/motor deficit), Open dislocation with exposed joint, Compartment syndrome (pain out of proportion, tense compartment), Irreducible dislocation (interposed soft tissue), Associated neurovascular injury requiring urgent intervention, Posterior shoulder dislocation (easily missed), Recurrent dislocations despite conservative management.

Learning map

Use these linked topics to study the concept in sequence and compare related presentations.

Prerequisites

Start here if you need the foundation before this topic.

  • Joint Anatomy and Biomechanics
  • Fracture Management Principles

Differentials

Competing diagnoses and look-alikes to compare.

  • Fracture-Dislocation
  • Ligamentous Injury Without Dislocation

Consequences

Complications and downstream problems to keep in mind.

  • Post-traumatic Osteoarthritis
  • Chronic Joint Instability