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Otitis Externa

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Overview

Otitis Externa

Quick Reference

Critical Alerts

  • Necrotizing (malignant) otitis externa is life-threatening: Diabetics, immunocompromised; skull base osteomyelitis
  • Differentiate from otitis media: OE = canal involvement, pain with tragal pressure; OM = middle ear, TM abnormality
  • Topical antibiotics are first-line: Not systemic
  • Keep ear dry during treatment
  • Refer for persistent or severe cases: ENT for debridement

Key Diagnostics

FindingOtitis ExternaOtitis Media
Pain with tragal pressureYesNo
Canal erythema/edemaYesNo
Tympanic membraneNormal or obscuredBulging, erythematous
DischargeFrom canalFrom middle ear (if TM perforated)

Emergency Treatments

ConditionTreatment
Uncomplicated OETopical fluoroquinolone drops (ofloxacin, ciprofloxacin/dexamethasone) × 7-10 days
Severe canal edemaEar wick + topical drops
Necrotizing OEIV antipseudomonal antibiotics + CT + ENT
Pain controlNSAIDs, acetaminophen

Definition

Overview

Otitis externa (OE), also known as "swimmer's ear," is an infection of the external auditory canal. It is typically caused by bacteria (Pseudomonas aeruginosa, Staphylococcus aureus) and presents with ear pain, itching, and discharge. Topical antibiotic drops are the mainstay of treatment. Necrotizing (malignant) otitis externa is a severe form seen in diabetics and immunocompromised patients.

Classification

By Severity:

TypeFeatures
MildMinimal canal edema, mild discomfort
ModerateCanal partially occluded, moderate pain
SevereCanal fully occluded, severe pain, may extend beyond canal

By Chronicity:

TypeDuration
Acute<6 weeks
Chronic> months

Special Type:

TypeFeatures
Necrotizing (malignant) OEOsteomyelitis of skull base; diabetics, immunocompromised

Epidemiology

  • Common: 10% of people affected at some point
  • Peak age: 7-12 years; also common in adults
  • Risk increases with: Swimming, humid climates, hearing aid use

Etiology

Pathogens:

OrganismFrequency
Pseudomonas aeruginosa40-50%
Staphylococcus aureus20-30%
Other Gram-negatives10%
Fungi (Aspergillus, Candida)5-10% (otomycosis)

Risk Factors:

FactorMechanism
SwimmingMoisture, pH change
Ear instrumentation (Q-tips)Trauma, cerumen removal
Hearing aids/Ear plugsOcclusion, moisture
Eczema, psoriasisCompromised skin barrier
Diabetes, immunocompromiseRisk of necrotizing OE

Pathophysiology

Mechanism

  1. Disruption of protective cerumen: Caused by water, trauma, or instrumentation
  2. Moisture and pH change: Favor bacterial growth
  3. Bacterial colonization: Pseudomonas, Staph
  4. Inflammation: Edema, erythema, pain
  5. Extension (in severe cases): Cellulitis, perichondritis, necrotizing OE

Necrotizing Otitis Externa

  • Invasive infection extending to skull base
  • Osteomyelitis of temporal bone
  • Can involve cranial nerves (VII most common)
  • Life-threatening if untreated

Clinical Presentation

Symptoms

SymptomDescription
Ear pain (otalgia)Often severe; worse with jaw movement
ItchingEarly symptom
DischargeOften purulent
Hearing lossIf canal occluded
FullnessSensation of blockage

History

Key Questions:

Physical Examination

External Ear:

FindingSignificance
Pain with tragal pressureClassic for OE; not present in OM
Pain with pinna manipulationClassic for OE
Canal erythema and edemaHallmark
Purulent dischargeInfection
Debris in canalFungal or severe bacterial
Periauricular cellulitisSevere OE
Granulation tissue in canalNecrotizing OE

Tympanic Membrane:

Cranial Nerve Exam:


Ear pain, discharge, itching?
Common presentation.
Recent swimming or water exposure?
Common presentation.
Ear instrumentation (Q-tips)?
Common presentation.
Hearing aid use?
Common presentation.
Diabetes or immunocompromise?
Common presentation.
Prior ear infections or surgery?
Common presentation.
Duration of symptoms?
Common presentation.
Red Flags

Necrotizing (Malignant) Otitis Externa

FindingAction
Diabetes or immunocompromise + severe OEHigh suspicion
Granulation tissue at bone-cartilage junctionPathognomonic
Facial nerve palsy (CN VII)Indicates skull base involvement
Cranial nerve deficits (IX, X, XI, XII)Advanced disease
Fever, severe pain, refractory to topical treatmentCT, MRI

Other Concerning Features

FindingConcern
Periauricular cellulitisExtension of infection
PerichondritisAuricular cartilage involvement
Mastoid tendernessMastoiditis

Differential Diagnosis

Other Causes of Ear Pain

DiagnosisFeatures
Acute otitis mediaBulging TM, fever, middle ear effusion
Foreign bodyVisible on otoscopy
Cerumen impactionImpacted wax, hearing loss
Herpes zoster oticus (Ramsay Hunt)Vesicles in canal, facial palsy
Contact dermatitisItching, history of earring/product use
FurunculosisLocalized abscess in canal
Referred pain (TMJ, dental)Normal ear exam

Diagnostic Approach

Clinical Diagnosis

  • OE is a clinical diagnosis
  • Based on history and otoscopy

Otoscopy

FindingSignificance
Canal erythema, edemaHallmark
DischargeBacterial or fungal
TM normal (if visible)Differentiates from OM
Granulation tissueNecrotizing OE

Imaging

Not Routinely Needed

Indications for CT/MRI Temporal Bone:

IndicationImaging
Suspected necrotizing OECT with contrast
Cranial nerve involvementMRI
Refractory to treatmentCT

Laboratory

TestIndication
Ear cultureRefractory cases, immunocompromised
ESR, CRPNecrotizing OE
GlucoseUndiagnosed diabetes

Treatment

Principles

  1. Topical antibiotics are first-line: Not systemic
  2. Keep ear dry: Avoid water during treatment
  3. Pain control: NSAIDs, acetaminophen
  4. Ear wick if canal occluded: To deliver drops
  5. ENT referral for severe or necrotizing OE

Topical Antibiotic Drops

First-Line:

AgentDoseDuration
Ofloxacin otic 0.3%5-10 drops BID7-10 days
Ciprofloxacin/Dexamethasone (Ciprodex)4 drops BID7 days
Ciprofloxacin otic 0.3%3-4 drops BID7 days

Alternative (If Fungal Suspected):

AgentDose
Clotrimazole 1% solutionApply BID × 10-14 days
Acetic acid otic (VoSol)Acidifies canal

Notes:

  • Fluoroquinolones are safe if TM perforated
  • Aminoglycoside-containing drops (neomycin) are ototoxic if TM perforated—avoid

Ear Wick

Indication: Canal too edematous to allow drop penetration

Technique:

  • Insert wick gently into canal
  • Apply drops to wick; wick swells and delivers medication
  • Remove or replace in 24-48 hours

Pain Control

AgentDose
Ibuprofen400-600 mg q6-8h
Acetaminophen650-1000 mg q6h
Opioids (short-term)For severe pain

Necrotizing Otitis Externa

InterventionDetails
CT temporal boneAssess bone involvement
MRIIf cranial nerve involvement
IV antipseudomonal antibioticsCiprofloxacin IV, pip-tazo, cefepime
ENT consultationUrgent
Surgical debridementIf necrotic tissue
Hyperbaric oxygenAdjunct in refractory cases
Long duration of therapy6-8 weeks

Disposition

Discharge Criteria

  • Uncomplicated OE
  • Pain controlled
  • Able to administer drops
  • No signs of necrotizing OE

Admission Criteria

  • Necrotizing OE
  • Severe cellulitis
  • Immunocompromised with severe infection
  • Unable to tolerate oral intake

Referral

IndicationReferral
Suspected necrotizing OEENT (urgent)
Refractory to treatmentENT
Severe canal edema requiring debridementENT

Follow-Up

SituationFollow-Up
Uncomplicated OEPCP in 1-2 weeks if not improving
Wick placed24-48 hours for wick removal/replacement
Necrotizing OEENT and infectious disease

Patient Education

Condition Explanation

  • "You have an infection of the ear canal, often called swimmer's ear."
  • "Antibiotic ear drops will treat the infection."
  • "Keep your ear dry during treatment."

Home Care

  • Apply ear drops as directed
  • Keep ear dry (use cotton ball with petroleum jelly when showering)
  • Avoid swimming until healed
  • Do not use Q-tips or other objects in ear

Warning Signs to Return

  • Pain getting worse despite treatment
  • Fever
  • Facial weakness or drooping
  • Swelling or redness spreading around ear
  • No improvement after 48-72 hours

Special Populations

Diabetics

  • Higher risk of necrotizing OE
  • Maintain tight glucose control
  • Low threshold for imaging and ENT referral

Immunocompromised

  • Higher risk of severe and necrotizing OE
  • Broader antibiotic coverage
  • ENT involvement early

Perforated Tympanic Membrane

  • Avoid aminoglycoside-containing drops (ototoxic)
  • Fluoroquinolone drops are safe

Fungal Otitis Externa (Otomycosis)

  • More common in humid climates, after antibiotic use
  • Debris may appear fluffy or black
  • Treat with antifungal drops (clotrimazole)

Quality Metrics

Performance Indicators

MetricTargetRationale
Topical (not systemic) abx for uncomplicated OE>0%Guideline adherence
Tragal and pinna tenderness documented>0%Diagnostic
Necrotizing OE identified and referred100%Life-threatening
TM status documented100%Differentiate from OM

Documentation Requirements

  • Canal and TM appearance
  • Tragal/pinna tenderness
  • Risk factors (diabetes, immunocompromise)
  • Treatment prescribed
  • Return precautions

Key Clinical Pearls

Diagnostic Pearls

  • Tragal pressure pain = OE: Not OM
  • TM normal in OE: If visible
  • Granulation tissue = Necrotizing OE: CT, ENT
  • Diabetes + severe OE = High suspicion for necrotizing OE
  • Fungal OE (otomycosis): Fluffy debris, pruritus
  • Check CN VII if necrotizing OE suspected

Treatment Pearls

  • Topical drops are first-line: Not oral antibiotics
  • Fluoroquinolone drops are safe with TM perforation: Aminoglycosides are not
  • Wick for occluded canal: Delivers drops to canal
  • Keep ear dry: No swimming, use barriers when showering
  • Acetic acid drops help prevent recurrence: Acidifies canal
  • Necrotizing OE needs IV antibiotics and ENT

Disposition Pearls

  • Most uncomplicated OE can be discharged: With topical drops
  • Admit for necrotizing OE: High mortality if untreated
  • ENT for refractory or severe cases: Debridement may be needed
  • Follow-up if not improving in 48-72 hours

References
  1. Rosenfeld RM, et al. Clinical Practice Guideline: Acute Otitis Externa. Otolaryngol Head Neck Surg. 2014;150(1 Suppl):S1-S24.
  2. Kaushik V, et al. Interventions for acute otitis externa. Cochrane Database Syst Rev. 2010;(1):CD004740.
  3. Roland PS, et al. Acute otitis externa: Clinical practice guideline. 2006.
  4. Carfrae MJ, Kesser BW. Malignant otitis externa. Otolaryngol Clin North Am. 2008;41(3):537-549.
  5. Rubin Grandis J, et al. The changing face of malignant (necrotising) external otitis. Lancet Infect Dis. 2004;4(1):34-39.
  6. Hajjartabar M. Poor diabetic control as a risk factor for necrotizing otitis externa. J Laryngol Otol. 1999;113(11):1047-1049.
  7. Schaefer P, et al. Acute otitis externa: an update. Am Fam Physician. 2012;86(11):1055-1061.
  8. UpToDate. Acute otitis externa: clinical features and diagnosis. 2024.

At a Glance

EvidenceStandard
Last UpdatedRecently

Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines