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Infectious Diseases
General Practice

Roseola Infantum

High EvidenceUpdated: 2025-12-23

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Red Flags

  • Febrile status epilepticus (>5 mins)
  • Signs of meningitis (neck stiffness, photophobia)
  • Non-blanching rash (meningococcal sepsis)
  • Severe dehydration in infant
  • Bulging fontanelle
Overview

Roseola Infantum

1. Clinical Overview

Summary

Roseola infantum, also known as Exanthem Subitum or Sixth Disease, is a common benign viral exanthem of early childhood caused primarily by Human Herpesvirus 6 (HHV-6) and less commonly HHV-7. It is characterized by a specific clinical course: a sudden high fever (often >39°C) persisting for 3-5 days which abruptly resolves, followed immediately by the appearance of a maculopapular rash. It is a major cause of febrile seizures in infants under 2 years of age.

Key Facts

  • Definition: Acute viral infection of infants characterized by high fever followed by a rash upon defervescence.
  • Prevalence: Near universal infection (>90%) by age 2-3 years.
  • Mortality/Morbidity: Generally benign; primary morbidity arises from febrile seizures (occur in 10-15% of cases).
  • Key Management: Supportive care (hydration, antipyretics) and reassurance.
  • Critical Threshold: Seizure duration >5 minutes requires acute management.
  • Key investigation: Clinical diagnosis; investigations rarely indicated unless ruling out sepsis.

Clinical Pearls

The "Well" Febrile Infant: A classic feature is an infant with a very high fever (39-40°C) who appears surprisingly well, alert, and playful when the fever is actively controlled.

The "Defervescence Rash": The rash of Roseola is unique because it appears after the fever stops. If the rash appears while the fever is still high, consider Measles or Kawasaki disease.

Nagayama Spots: Look for erythematous papules on the soft palate and uvula (Nagayama spots); present in ~65% of cases and can help differentiate from other viral exanthems.

Why This Matters Clinically

Roseola is one of the most common causes of acute febrile illness and emergency department visits in infants 6-18 months old. Recognizing the classic pattern avoids unnecessary antibiotic use, lumbar punctures, and hospital admissions for suspected sepsis. It is also the most common identifiable trigger for a first febrile seizure.


2. Epidemiology

Incidence & Prevalence

  • Incidence: Extremely common; virtually all children are infected by age 3.
  • Prevalence: Seroprevalence of HHV-6 is >90% in adults.
  • Trend: Stable endemic infection; no clear seasonal variation (unlike Influenza or RSV).

Demographics

FactorDetails
AgePeak incidence 6 to 15 months. Rare <3 months (maternal antibodies) or > years.
SexMale:Female ratio is approximately 1:1.
EthnicityOccurs worldwide in all ethnic groups.
GeographyGlobal distribution.

Risk Factors

Non-Modifiable:

  • Age: Being between 6 months and 2 years.
  • Immune status: Immunocompromised children may have severe or atypical disease.

Modifiable:

  • Childcare attendance: Increased exposure risk (though transmission is often from asymptomatic adult saliva).
  • Siblings: Close contact with older siblings.
Risk FactorRelative Risk
Age 6-15 monthsHigh (Primary window of vulnerability)
Daycare attendanceModerate increase in exposure

3. Pathophysiology

Mechanism

Step 1: Viral Entry & Replication

  • Agent: Human Herpesvirus 6 (variants A and B, with B causing 99% of Roseola) or HHV-7.
  • Transmission: Respiratory droplets or saliva (often from asymptomatic caregivers causing horizontal transmission).
  • Entry: Virus enters via oropharynx, replicates in salivary glands.

Step 2: Viraemia & Dissemination

  • Incubation: 9-10 days (range 5-15 days).
  • Viraemia: Virus infects T-lymphocytes (CD4+ T cells) causing high-grade viraemia.
  • Cytokine Storm: Induction of cytokines (IFN-alpha, TNF-alpha, IL-1beta) correlates with the rapid onset of high fever.

Step 3: Clinical Manifestation (Rash)

  • Defervescence: Fever falls as host immunity (antibodies + cellular response) clears viraemia.
  • Rash Mechanism: The rash is believed to be an immune complex-mediated reaction (antigen-antibody deposition in skin) rather than direct viral replication in skin cells.
  • Latency: Like all herpesviruses, HHV-6 establishes lifelong latency in T-cells and monocytes.

Classification

StageDefinitionClinical Features
IncubationPost-exposure replicationAsymptomatic (approx. 10 days).
Febrile PhaseActive viraemiaSudden onset high fever (39-40.5°C), irritability, lymphadenopathy.
Exanthem PhaseImmune clearanceFever resolves abruptly; maculopapular rash appears centrally.

Anatomical/Physiological Considerations

The Developing Brain: The immature infant brain is more susceptible to fever-induced lowering of the seizure threshold. HHV-6 is also neurotropic and has been detected in CSF during febrile seizures, suggesting direct CNS invasion may play a role beyond simple febrile threshold.


4. Clinical Presentation

Symptoms

Typical Presentation:

Atypical Presentations:

Signs

Red Flags

[!CAUTION] Red Flags — Seek immediate help if:

  • Non-blanching rash: Indicates petechiae/purpura (Meningococcal sepsis).
  • Prolonged seizure: >5 minutes (Status Epilepticus).
  • Decreased consciousness: Lethargy, difficult to wake.
  • Bulging fontanelle: Sign of raised ICP (Meningitis).
  • Fever >5 days: Consider Kawasaki disease or other bacteria.

High Fever (100%)
Sudden onset, 39°C to 40.5°C.
Irritability/Fussiness (Common)
Especially when fever is peaking.
"Well" appearance (Common)
Child often alert and playful when fever breaks or is treated.
Rash (~20-30% of infected children)
Appears after fever goes away.
5. Clinical Examination

Structured Approach

General:

  • ABCDE Assessment: Prioritize Airway/Breathing/Circulation if child is seizing or drowsy.
  • Hydration Status: Check capillary refill, mucous membranes, skin turgor.
  • Alertness: "Tickle test" or toy interaction to assess neurological status.

Specific System Examination:

  • Skin: Perform "glass test" (diascopy) to ensure rash blanches. Check distribution (Trunk > Face).
  • ENT: Inspect throat for Nagayama spots. Check ears to rule out bacterial otitis media.
  • Neurology: Check for neck stiffness (meningism) and fontanelle tension.

Special Tests

TestTechniquePositive FindingSensitivity/Specificity
Glass TestPress clear glass against rashRash disappears (blanches)High specificity for ruling out purpura
Fontanelle CheckPalpate anterior fontanelle (sitting up)Soft and flat (Normal) vs Bulging (Meningitis)Crucial in infants
Kernig's SignFlex hip 90°, extend kneePain/resistance (Meningitis)Low sensitivity in infants

6. Investigations

First-Line (Bedside)

  • Urinalysis: Essential in all febrile infants without a clear focus to rule out UTI.
  • Blood Glucose: Check if drowsy or seizing (rule out hypoglycaemia).

Laboratory Tests

Routine bloods are generally NOT required for classic Roseola.

TestExpected FindingPurpose
FBCLeukopenia (low WBC) with lymphocytosisViral pattern; helps exclude bacterial sepsis.
CRPNormal or mild elevationExclude severe bacterial infection.
Blood CultureNegativeOnly indicated if sepsis suspected.
Urine CultureNegativeRule out UTI.

Imaging

Imaging is rarely indicated.

ModalityFindingsIndication
CXRNormalOnly if significant respiratory distress/signs.
CT/MRI BrainNormal (unless complications)Only for complex seizures or focal neuro signs.

Diagnostic Criteria

Diagnosis is primarily clinical based on the classic sequence:

  1. High fever (3-5 days) in a well-appearing infant.
  2. Defervescence (fever stops).
  3. Appearance of characteristic rose-pink rash on trunk.

7. Management

Management Algorithm

Acute/Emergency Management (Seizures)

Immediate Actions (Febrile Convulsion):

  1. Safety: Place child in recovery position; remove dangerous objects.
  2. Timing: Note start time. Most stop within 2-3 mins.
  3. Airway: Ensure airway is patent. Do not put anything in mouth.
  4. Medication: If >5 mins, Administer:
    • Buccal Midazolam: 0.5mg/kg
    • Or Rectal Diazepam: 0.5mg/kg
  5. Call for Help: Ambulance if first seizure or >5 mins.

Conservative Management

  • Reassurance: Explain the benign nature ("It will get better on its own").
  • Hydration: Encourage frequent breastfeeds or clear fluids.
  • Environment: Keep child cool and comfortable; avoid over-bundling.
  • Exclusion: Keep away from childcare until systemic symptoms resolve (rash itself is not contagious—viraemia has passed).

Medical Management

Antibiotics are NOT indicated.

Drug ClassDrugDoseDuration
AntipyreticParacetamol15mg/kg Q6H (Max 60mg/kg/day)PRN for distress
NSAIDIbuprofen10mg/kg Q8H (Max 30mg/kg/day)PRN for distress (> months old)

Surgical Management

Not applicable.

Disposition

  • Admit if: First febrile seizure (observation), bacterial sepsis cannot be excluded, severe dehydration.
  • Discharge if: Classic presentation, well hydrated, re-assuring parents.
  • Follow-up: GP review if fever persists >5 days or rash becomes non-blanching.

8. Complications

Immediate (Minutes-Hours)

ComplicationIncidencePresentationManagement
Febrile Seizure10-15%Tonic-clonic activity, loss of consciousnessairway protection, benzos if prolonged
DehydrationCommonDry mucous membranes, reduced wet nappiesOral rehydration solution

Early (Days)

  • Aseptic Meningitis: Rare. Headache, lethargy, photophobia. Usually self-limiting.
  • Encephalitis: Very rare. Altered mental status. Requires antivirals (Ganciclovir/Foscarnet) in immunocompromised.

Late (Weeks-Months)

  • Mesial Temporal Sclerosis: Controversial potential link between prolonged HHV-6 febrile seizures and later temporal lobe epilepsy (research ongoing).

9. Prognosis & Outcomes

Natural History

Roseola is a self-limiting illness.

  • Fever Phase: 3-5 days.
  • Rash Phase: 1-2 days (sometimes only hours).
  • Full Recovery: usually within a week.

Outcomes with Treatment

VariableOutcome
MortalityExtremely rare in healthy children.
MorbidityLow (seizures are terrifying for parents but benign).
RecurrenceSecond episodes possible (HHV-7 infection) but rare.
5-year survival~100%

Prognostic Factors

Good Prognosis:

  • Immunocompetent host.
  • Typical "well" appearance between fever spikes.
  • Rapid resolution of rash.

Poor Prognosis:

  • Immunocompromised state (risk of bone marrow suppression, pneumonitis, encephalitis).
  • Prolonged status epilepticus.

10. Evidence & Guidelines

Key Guidelines

  1. NICE CKS (2024) — Viral rash in pregnancy and childhood. Recommends supportive care and exclusion of serious causes. NICE
  2. American Academy of Pediatrics (2011) — Neurodiagnostic evaluation of the child with a simple febrile seizure. Evidence against routine lumbar puncture in vaccinated children. AAP

Landmark Trials

Hall et al. (1994) — HHV-6 is main cause of emergency visits.

  • 2000+ patients.
  • Key finding: HHV-6 accounted for 20% of ED visits for fever in infants 6-12 months.
  • Clinical Impact: Established Roseola as a major public health entity, not just a rash.

Yamanishi et al. (1988) — Identification of HHV-6 as cause of Exanthem Subitum.

  • Key finding: Isolated the virus from lymphocytes of patients.
  • Clinical Impact: Confirmed etiology.

Evidence Strength

InterventionLevelKey Evidence
Antipyretics for comfort1aCochrane Reviews (Paracetamol/Ibuprofen efficacy)
Antibiotics1a (Against)Multiple studies proving viral etiology
Routine LP for simple seizure2a (Against)AAP Guidelines

11. Patient/Layperson Explanation

What is Roseola?

Roseola (often called "Sixth Disease") is a very common viral infection in babies and toddlers. It is famous for causing a sudden high fever that lasts a few days, followed by a pink rash just as the fever breaks. It is almost a "rite of passage" for childhood.

Why does it matter?

While usually harmless, the high fever can be frightening for parents and can sometimes trigger a "febrile convulsion" (a seizure caused by high temperature). The good news is that once the rash appears, the child is usually getting better and is no longer contagious.

How is it treated?

  1. Comfort: Use Paracetamol or Ibuprofen if the child is miserable or in pain. Treating the fever number itself is not necessary if the child is happy.
  2. Hydration: Offer plenty of breastmilk, formula, or water.
  3. Patience: Antibiotics do not work because it is a virus. It will go away on its own.

What to expect

  • Days 1-3: High fever, maybe fussy, but plays in between.
  • Day 4: Fever disappears suddenly.
  • Day 5: Pink, spotty, non-itchy rash appears on tummy/back.
  • Day 6-7: Rash fades, child back to normal.

When to seek help

  • If the child has a seizure (convulsion).
  • If the rash does not fade when pressed under a glass (glass test).
  • If the baby is floppy, unresponsive, or not drinking fluids.
  • If the fever lasts more than 5 days.

12. References

Primary Guidelines

  1. Stone RC et al. Viral exanthems. Dermatol Online J. 2015;21(12):13030/qt98b6775z. PMID: 26990473

Key Trials

  1. Hall CB et al. Human herpesvirus 6 infection in children. A prospective study of complications and reactivation. N Engl J Med. 1994;331(7):432-8. PMID: 8035839
  2. Yamanishi K et al. Identification of human herpesvirus-6 as a causal agent for exanthem subitum. Lancet. 1988;1(8594):1065-7. PMID: 2896909
  3. Tesini BL et al. Human Herpesviruses 6 and 7. Principles and Practice of Pediatric Infectious Diseases. 2018.

Further Resources

  • DermNet NZ: Roseola Infantum
  • Radiopaedia: Human herpesvirus 6


Medical Disclaimer: MedVellum content is for educational purposes and clinical reference. Clinical decisions should account for individual patient circumstances. Always consult appropriate specialists.

Last updated: 2025-12-23

At a Glance

EvidenceHigh
Last Updated2025-12-23

Red Flags

  • Febrile status epilepticus (&gt;5 mins)
  • Signs of meningitis (neck stiffness, photophobia)
  • Non-blanching rash (meningococcal sepsis)
  • Severe dehydration in infant
  • Bulging fontanelle

Clinical Pearls

  • **The "Well" Febrile Infant**: A classic feature is an infant with a very high fever (39-40°C) who appears surprisingly well, alert, and playful when the fever is actively controlled.
  • **The "Defervescence Rash"**: The rash of Roseola is unique because it appears *after* the fever stops. If the rash appears while the fever is still high, consider Measles or Kawasaki disease.
  • **Nagayama Spots**: Look for erythematous papules on the soft palate and uvula (Nagayama spots); present in ~65% of cases and can help differentiate from other viral exanthems.
  • **Red Flags — Seek immediate help if:**
  • - **Non-blanching rash**: Indicates petechiae/purpura (Meningococcal sepsis).

Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines