Tension Pneumothorax
Tension pneumothorax develops when air enters the pleural space through a one-way valve mechanism, progressively increas... ACEM Primary Written, ACEM Primary V
Clinical board
A visual summary of the highest-yield teaching signals on this page.
Urgent signals
Safety-critical features pulled from the topic metadata.
- Severe respiratory distress with absent breath sounds
- Hypotension with elevated JVP (obstructive shock)
- Tracheal deviation away from affected side (late sign)
- Difficulty ventilating intubated patient
Exam focus
Current exam surfaces linked to this topic.
- ACEM Primary Written
- ACEM Primary Viva
- ACEM Fellowship Written
- ACEM Fellowship OSCE
Linked comparisons
Differentials and adjacent topics worth opening next.
- Cardiac Tamponade
- Massive Pulmonary Embolism
Editorial and exam context
Topic family
This concept exists in multiple MedVellum libraries. Use the primary page for the broadest reference view and the others for exam-specific framing.
Key Facts Definition : Progressive accumulation of air in pleural space under pressure, causing mediastinal shift and cardiovascular compromise Incidence : 5-10% of traumatic pneumothoraces develop tension; rare in...
Tension pneumothorax develops when air enters the pleural space through a one-way valve mechanism, progressively increas... ACEM Primary Written, ACEM Primary V
Quick Answer
One-liner: Tension pneumothorax is a life-threatening progressive accumulation of air in the pleural space causing cardiovascular collapse requiring immediate needle decompression.
Tension pneumothorax develops when air enters the pleural space through a one-way valve mechanism, progressively increasing intrathoracic pressure, collapsing the ipsilateral lung, shifting mediastinal structures, and compromising venous return causing obstructive shock. Mortality is 30-50% if untreated. This is a clinical diagnosis - do NOT delay decompression for imaging in unstable patients. Immediate needle decompression (5th ICS anterior axillary line) followed by tube thoracostomy is life-saving.
ACEM Exam Focus
Primary Exam Relevance
- Anatomy: Pleural space boundaries, intercostal neurovascular bundle (runs inferior to rib), triangle of safety (4th-5th ICS, anterior to mid-axillary line, lateral border of pectoralis major), thoracic outlet anatomy
- Physiology: Venous return physiology, intrathoracic pressure effects on preload, Frank-Starling curve, one-way valve mechanism, mediastinal shift, ventilation-perfusion mismatch
- Pathophysiology: Progressive air accumulation → increased intrathoracic pressure → IVC/SVC compression → reduced venous return → decreased cardiac output → obstructive shock
Fellowship Exam Relevance
- Written: Reversible causes of cardiac arrest (4Hs/4Ts), needle decompression site selection, finger thoracostomy technique, chest drain insertion, traumatic vs spontaneous vs iatrogenic causes, ventilator-induced barotrauma
- OSCE: Resuscitation station (tension pneumothorax management in trauma), POCUS identification, needle decompression procedure, chest drain insertion, communication with retrieval services
- Key domains tested: Medical Expert (recognition and immediate management), Communicator (team leadership, closed-loop communication), Leader (crisis resource management), Collaborator (coordinating with cardiothoracics, retrieval)
Key Points
The 5 things you MUST know:
- Tension pneumothorax is a CLINICAL diagnosis - Do NOT wait for imaging if patient is unstable. Treat immediately based on clinical findings.
- Classic triad: Respiratory distress + absent breath sounds + hypotension. JVP elevation and tracheal deviation are LATE signs, often absent.
- Preferred decompression site: 5th ICS anterior axillary line (NOT 2nd ICS MCL which has 35-50% failure rate due to chest wall thickness).
- Use 14-16G catheter ≥5cm length (8cm preferred) - Standard 5cm needles fail in many patients due to chest wall thickness.
- Needle decompression is a BRIDGE - Always follow with definitive tube thoracostomy (chest drain). Consider finger thoracostomy in cardiac arrest or ventilated patients.
Epidemiology
| Metric | Value | Source |
|---|---|---|
| Incidence (trauma) | 3-5% of major chest trauma | [1] |
| Incidence (spontaneous) | 0.17 per 100,000/year | [2] |
| Iatrogenic (mechanical ventilation) | 4-15% of ARDS patients | [3] |
| Mortality (untreated) | 30-50% | [4] |
| Mortality (treated) | 3-8% | [5] |
| Peak age (spontaneous) | 20-40 years (tall, thin males) | [6] |
| Peak age (traumatic) | 20-45 years | [7] |
| Gender ratio | M:F 3-5:1 (spontaneous) | [8] |
Australian/NZ Specific
- Major trauma registry data shows 4.2% of blunt chest trauma presentations have pneumothorax requiring intervention [9]
- Aboriginal and Torres Strait Islander people have 2-3x higher rates of trauma-related chest injuries [10]
- Remote/rural presentations often delayed 6-24 hours due to retrieval logistics [11]
- RFDS retrieval for tension pneumothorax has 8-12% incidence of re-accumulation during air transport [12]
Pathophysiology
Mechanism
Tension pneumothorax develops when air enters the pleural space through a one-way valve mechanism, allowing air to enter during inspiration but preventing its escape during expiration. This creates progressive air accumulation with increasing intrathoracic pressure.
One-Way Valve Sources:
- Traumatic: Chest wall defect, lung laceration from rib fracture, penetrating injury
- Spontaneous: Ruptured bleb/bullae (especially tall, thin males), subpleural emphysematous changes
- Iatrogenic: Mechanical ventilation with high peak pressures, central line insertion, intercostal nerve block, lung biopsy
- Post-procedural: Failed simple pneumothorax managed conservatively, inadequate chest drain function
Pathological Progression
Air Entry (one-way valve)
→ Progressive Air Accumulation
→ Increased Intrathoracic Pressure (greater than 20 cmH₂O)
→ Ipsilateral Lung Collapse
→ Mediastinal Shift to Contralateral Side
→ IVC/SVC Kinking and Compression
→ Reduced Venous Return (↓Preload)
→ Reduced Cardiac Output
→ Obstructive Shock
→ PEA Cardiac Arrest
Cardiovascular Effects
Reduced Preload (most important):
- Increased intrathoracic pressure compresses the IVC and SVC at the thoracic inlet
- Kinking of great veins at mediastinal shift
- Reduced right ventricular filling (↓preload)
- JVP paradoxically elevated (obstruction to venous return, NOT fluid overload)
Increased Afterload:
- Right ventricular afterload increases due to compressed pulmonary vasculature
- Left ventricular preload reduced (dependent on RV output)
Direct Cardiac Compression:
- Mediastinal shift may directly compress cardiac chambers
- Reduced diastolic filling
Respiratory Effects
- Ipsilateral lung collapse (complete)
- Contralateral lung compression from mediastinal shift (partial)
- V/Q mismatch with shunt physiology
- Hypoxia and hypercarbia
- Increased work of breathing
Why It Matters Clinically
Understanding the preload-dependent shock mechanism explains why:
- Hypotension occurs BEFORE hypoxia in many cases (unlike other respiratory emergencies)
- JVP is elevated (mimicking cardiac tamponade, NOT hypovolaemia)
- Fluid boluses may temporarily improve BP (increasing preload) but won't fix the problem
- Positive pressure ventilation accelerates deterioration (further increases intrathoracic pressure)
- Immediate decompression reverses shock (restores venous return within seconds)
Clinical Approach
Recognition
Think Tension Pneumothorax When:
- Trauma patient with respiratory distress + hypotension
- Ventilated patient with sudden deterioration + high peak pressures
- Post-central line insertion with respiratory distress
- Known simple pneumothorax with sudden clinical deterioration
- PEA cardiac arrest (one of the 4 Ts - reversible causes)
- Asthma/COPD patient on NIV/BiPAP with sudden collapse
Initial Assessment
Primary Survey (ABCDE)
A - Airway:
- Usually patent (unless GCS decreased from shock)
- May require definitive airway BUT beware: intubation + positive pressure ventilation will accelerate tension
- Pre-intubation checklist: If tension suspected, decompress BEFORE intubation if possible
B - Breathing:
| Finding | Significance | Frequency |
|---|---|---|
| Severe respiratory distress | Universal feature | 95-100% |
| Tachypnoea (RR greater than 30) | Compensation for V/Q mismatch | 90-95% |
| Absent/severely diminished breath sounds (ipsilateral) | Lung collapse | 85-95% |
| Hyperresonance to percussion (ipsilateral) | Air-filled pleural space | 70-80% |
| Asymmetric chest wall movement | Ipsilateral lag | 60-70% |
| Subcutaneous emphysema | Tracking air from injury site | 30-50% (traumatic) |
C - Circulation:
| Finding | Significance | Frequency |
|---|---|---|
| Hypotension (SBP below 90 mmHg) | Obstructive shock | 80-90% |
| Tachycardia (HR greater than 120) | Compensation | 90-95% |
| Elevated JVP | Impeded venous return | 40-60% |
| Narrow pulse pressure | Reduced stroke volume | 60-70% |
| Pulsus paradoxus | Exaggerated intrathoracic pressure swings | 30-40% |
D - Disability:
- Agitation, confusion (hypoxia/hypoperfusion)
- GCS may be normal initially, drops with progressive shock
- Altered mental status in 40-60%
E - Exposure:
- Tracheal deviation (away from affected side): LATE sign, present in only 10-30% [13]
- Chest wall trauma (bruising, penetrating wounds, surgical emphysema)
- Look for causes: trauma, central line sites, recent procedures
CRITICAL ERROR: Waiting for tracheal deviation
- Tracheal deviation is a LATE and INCONSISTENT sign
- Only present in 10-30% of confirmed cases
- If tracheal deviation is present, patient is peri-arrest
- DO NOT wait for this sign before decompressing
History
Key Questions
| Question | Significance |
|---|---|
| "What happened?" (mechanism) | Trauma, spontaneous, iatrogenic? |
| "Any recent procedures?" | Central line, lung biopsy, intercostal block within 48h? |
| "Known lung disease?" | COPD, asthma, previous pneumothorax (recurrence risk 30-50%) |
| "When did symptoms start?" | Acute onset suggests tension vs gradual simple pneumothorax |
| "On a ventilator?" | High peak pressures cause barotrauma |
| "Any recent flights or diving?" | Pressure changes can convert simple → tension |
Red Flag Symptoms
Symptoms indicating impending arrest:
- Severe dyspnoea unable to speak
- Sense of impending doom
- Chest pain with syncope
- Cyanosis
- Sudden deterioration in ventilated patient
Examination
General Inspection
- Position: Upright, leaning forward, tripoding
- Respiratory effort: Use of accessory muscles, paradoxical breathing
- Colour: Cyanosis (late), pallor
- Diaphoresis, cool peripheries (shock)
- Glasgow Coma Scale
Specific Findings
| System | Finding | Significance |
|---|---|---|
| Respiratory | Absent breath sounds (ipsilateral) | Lung collapse - most reliable sign |
| Hyperresonance to percussion | Air-filled pleural space | |
| Reduced chest expansion | Ipsilateral lung not inflating | |
| Subcutaneous emphysema | Air tracking from injury | |
| Cardiovascular | Hypotension | Obstructive shock |
| Tachycardia | Compensation | |
| Elevated JVP | Obstructed venous return | |
| Weak peripheral pulses | Poor cardiac output | |
| Neurological | Agitation, confusion | Hypoxia/hypoperfusion |
| Reduced GCS | Shock progression | |
| Skin | Cyanosis | Severe hypoxia (late) |
| Cool, clammy | Shock |
Investigations
Immediate (Resus Bay)
DO NOT DELAY TREATMENT FOR INVESTIGATIONS Tension pneumothorax is a clinical diagnosis. In an unstable patient with clinical features, decompress immediately. Do NOT wait for CXR, CT, or even ultrasound.
| Test | Purpose | Key Finding | Timing |
|---|---|---|---|
| Point-of-Care Ultrasound (POCUS) | Rapid bedside confirmation if diagnosis uncertain | Absent lung sliding, absent B-lines, lung point | 30-90 seconds |
| Arterial Blood Gas | Assess oxygenation, ventilation, lactate | Type 1 RF (↓PaO₂), ↑lactate (shock) | After stabilisation |
| ECG | Exclude MI, assess rhythm | Sinus tachycardia, electrical alternans (rare) | Simultaneous with resus |
| Bedside CXR | Confirm if stable OR post-decompression | Complete lung collapse, mediastinal shift | ONLY if stable |
Point-of-Care Ultrasound
POCUS is highly sensitive (90-100%) and specific (95-100%) for pneumothorax [14]
Technique
- High-frequency linear probe (or curvilinear if obese)
- M-mode interrogation of pleural line
- Scan multiple rib spaces bilaterally (anterior and lateral)
Findings in Pneumothorax
| Sign | Description | Sensitivity | Specificity |
|---|---|---|---|
| Absent lung sliding | No "shimmering" movement of visceral pleura against parietal pleura | 90-95% | 60-85% (non-specific) |
| Absent B-lines | No vertical comet-tail artifacts | 90% | 70% |
| A-lines present | Horizontal reverberation artifacts | 80-90% | 60-70% |
| Lung point | Exact location where lung sliding stops | 60-80% | 100% (pathognomonic) |
| Barcode sign (M-mode) | Absent "seashore sign", replaced by horizontal lines | 95% | 95% |
POCUS Limitations:
- Absent lung sliding can occur in: apnoea, mainstem intubation, pleural adhesions, severe COPD/bullae
- Cannot differentiate simple from tension pneumothorax (clinical context required)
- In cardiac arrest, lung sliding may be absent even without pneumothorax
Standard ED Workup
| Test | Indication | Interpretation |
|---|---|---|
| Chest X-ray (PA/AP) | Stable patient, post-decompression confirmation | Visible pleural line, deep sulcus sign (supine), mediastinal shift |
| CT Chest | Occult pneumothorax, surgical planning, unclear diagnosis | Size quantification, underlying pathology (blebs, bullae) |
| ABG | All patients | PaO₂ below 60 mmHg (Type 1 RF), lactate greater than 2 (shock) |
| FBC, U&E, Coag | Pre-operative bloods if going to theatre | Anaemia, coagulopathy, renal function |
Advanced/Specialist
| Test | Indication | Availability |
|---|---|---|
| CT Pulmonary Angiogram | Exclude concomitant PE if high suspicion | Tertiary centres |
| Thoracic surgery consult | Persistent air leak, failure of re-expansion, need for VATS | Metropolitan/tertiary |
| Bronchoscopy | Suspected bronchial injury, persistent leak | Tertiary centres |
Chest X-ray Findings
Supine CXR (common in trauma resus):
- Deep sulcus sign: Abnormally deep, lucent costophrenic angle
- Increased lucency over upper abdomen
- Sharply defined cardiac border
- Mediastinal shift (late)
Erect CXR:
- Visible pleural line (lung edge)
- Absent lung markings peripheral to pleural line
- Mediastinal shift away from pneumothorax
- Tracheal deviation (late, severe)
- Subcutaneous emphysema
Management
Immediate Management (First 5 Minutes)
The "ABCDE + Decompress" Protocol:
- Call for help - Senior ED doctor, anaesthetics, cardiothoracics (0-30 sec)
- High-flow oxygen 15L non-rebreather (unless COPD with chronic hypercapnia) (30-60 sec)
- Large-bore IV access x2 (if not already) + bloods (1-2 min)
- POCUS if immediately available (60-90 sec) - DO NOT delay if not available
- Needle decompression - 5th ICS anterior axillary line (2-5 min)
- Tube thoracostomy preparation - immediately following needle decompression (5-15 min)
Needle Decompression (Immediate Life-Saving Intervention)
Indications:
- Clinical diagnosis of tension pneumothorax with haemodynamic compromise
- Cardiac arrest with suspected tension (one of 4Ts)
- Ventilated patient with sudden deterioration + unilateral absent breath sounds + high peak pressures
Site Selection (UPDATED GUIDELINES):
| Site | Success Rate | Advantages | Disadvantages |
|---|---|---|---|
| 5th ICS Anterior Axillary Line (PREFERRED) | 75-85% | Thinner chest wall, higher success rate, safer in CPR | Slightly more lateral |
| 2nd ICS Mid-Clavicular Line | 50-65% | Traditional teaching, familiar | 35-50% failure rate, thicker chest wall, fails in obese |
Why 5th ICS AAL is now preferred (ARC/ANZCOR 2023):
- Chest wall thickness at 2nd ICS MCL averages 4.5-5.5cm (frequently exceeds 5cm needle length) [15]
- Chest wall thickness at 5th ICS AAL averages 3.0-4.0cm (within reach of standard needles) [16]
- TCCC (Tactical Combat Casualty Care) and ATLS 10th edition both recommend lateral approach [17]
- Meta-analysis: 5th ICS AAL success rate 77-85% vs 2nd ICS MCL 50-67% [18]
Equipment:
- 14-16 gauge angiocatheter, ≥5cm length (8cm preferred if available)
- Alcohol swabs
- 10ml syringe (optional, to confirm air aspiration)
- Three-way tap (if available, to prevent air re-entry)
Procedure:
1. Identify landmarks:
- 5th ICS (level with nipple in males, inframammary crease in females)
- Anterior axillary line (lateral border of pectoralis major)
- Alternative: 2nd ICS, mid-clavicular line (2 fingerbreadths below clavicle)
2. Prepare site:
- Rapid skin prep with alcohol (if time permits - do NOT delay in peri-arrest)
- No local anaesthetic in arrested/peri-arrest patient
3. Insert needle:
- Angle perpendicular to chest wall
- Advance OVER THE TOP of the rib (neurovascular bundle runs inferior)
- Insert until "pop" felt (pleura penetrated) and/or hiss of air heard
- Advance catheter fully, withdraw needle
4. Secure:
- Tape catheter in place
- Attach three-way tap if available
- DO NOT occlude catheter
5. Confirm:
- Listen for rush of air (positive sign)
- Immediate clinical improvement (BP, RR, O₂ sats)
- If no improvement: consider other diagnosis (tamponade, PE, MI) or failed decompression
6. Proceed to definitive management:
- Tube thoracostomy MUST follow immediately
Common Needle Decompression Failures:
- Needle too short (5cm often inadequate in obese patients)
- Wrong site (below 5th rib → intra-abdominal)
- Catheter kinked or blocked
- Wrong diagnosis (tamponade, massive PE, MI)
- Catheter not advanced far enough (only needle penetrated, not catheter)
If needle decompression fails → Proceed immediately to FINGER THORACOSTOMY
Finger Thoracostomy (Alternative in Cardiac Arrest/Ventilated Patients)
Indications:
- Traumatic cardiac arrest with suspected tension pneumothorax
- Failed needle decompression
- Ventilated patient with suspected tension (more definitive than needle)
- High suspicion in shocked patient when immediate chest drain not feasible
Advantages over needle decompression:
- Cannot kink or block
- 100% success rate at decompressing if present
- Allows digital exploration of pleural space
- Can be converted to chest drain immediately
Procedure (Brief):
1. Landmarks: 5th ICS, anterior axillary line ("triangle of safety")
2. Incision: 3-4cm horizontal incision over 5th rib
3. Blunt dissection: Use artery forceps to dissect through muscle layers
4. Enter pleura: Push through parietal pleura with forceps/finger (will feel "pop")
5. Finger sweep: Insert finger into pleural space, sweep 360° to break adhesions
6. Confirm: Rush of air, immediate improvement
7. Secure: Leave wound open or cover with Asherman chest seal, proceed to chest drain
Tube Thoracostomy (Definitive Management)
ALL patients with tension pneumothorax require chest drain insertion following needle/finger decompression.
Timing:
- Immediately following stabilisation (within 15-30 minutes)
- Do NOT delay for radiology/CT
Site: Triangle of safety
- Superior border: 4th rib (nipple level)
- Anterior border: Lateral edge of pectoralis major
- Posterior border: Lateral edge of latissimus dorsi
- Typical insertion: 5th ICS, anterior axillary line
Technique: Seldinger vs Open (Blunt Dissection)
- Seldinger: Smaller drains (12-20F), lower complication rate, suitable for simple pneumothorax
- Open technique: Larger drains (28-32F), preferred for trauma, haemopneumothorax, tension
Drain Size:
- Simple pneumothorax: 12-20F pigtail
- Tension/trauma: 28-32F chest drain
- Haemopneumothorax: 32-36F (or 28F if Seldinger)
Management after insertion:
- Connect to underwater seal drain (UWSD) with low-pressure suction (-10 to -20 cmH₂O)
- Confirm bubbling (air drainage) and/or swinging (pleura re-expanding)
- Post-insertion CXR to confirm position and lung re-expansion
- Monitor for complications: bleeding, infection, re-expansion pulmonary oedema
Resuscitation (If Haemodynamically Unstable)
Airway
- Consider delayed sequence intubation (DSI) if severe respiratory distress
- PRE-INTUBATION DECOMPRESSION: If tension suspected, decompress BEFORE intubation if possible
- Positive pressure ventilation will worsen tension → have decompression equipment ready
Breathing
- High-flow oxygen 15L (target SpO₂ greater than 94%)
- Avoid bag-mask ventilation if possible (worsens tension)
- Post-intubation: use lung-protective ventilation (low tidal volumes 6-8 ml/kg, plateau pressure below 30 cmH₂O)
Circulation
Haemodynamic Targets:
- SBP greater than 90 mmHg
- MAP greater than 65 mmHg
- Urine output greater than 0.5 ml/kg/hr
Fluid Resuscitation:
- 500-1000ml crystalloid bolus (if hypotensive)
- Caution: Excessive fluids won't fix obstructive shock (decompression is definitive)
- If traumatic: consider blood products if haemopneumothorax
Vasopressors:
- Rarely needed if tension adequately decompressed
- If persistent hypotension post-decompression: consider noradrenaline 0.05-0.2 mcg/kg/min
- Suggests alternative/additional diagnosis (haemorrhage, tamponade, MI)
Medications
| Drug | Dose | Route | Timing | Notes |
|---|---|---|---|---|
| Oxygen | 15L non-rebreather | Inhalation | Immediate | Target SpO₂ greater than 94% |
| Analgesia (Fentanyl) | 25-50 mcg increments | IV | After stabilisation | For chest drain insertion |
| Sedation (Midazolam) | 1-2 mg increments | IV | If intubated | Avoid in haemodynamically unstable |
| Ketamine | 0.5-1 mg/kg | IV | Pre-intubation if DSI | Maintains BP, analgesic |
| Local anaesthetic (Lignocaine 1%) | 10-20ml | Subcutaneous/intercostal | Pre-chest drain | Max 3 mg/kg (plain) |
Paediatric Dosing
| Drug | Dose | Max | Notes |
|---|---|---|---|
| Oxygen | 15L non-rebreather (all ages) | - | Target SpO₂ greater than 94% |
| Fentanyl | 0.5-1 mcg/kg IV | 50 mcg | Titrate to effect |
| Ketamine | 1-2 mg/kg IV (sedation) | 100 mg | Maintains BP |
| Lignocaine 1% | 0.5-1ml/kg | 3 mg/kg | For chest drain insertion |
Paediatric chest drain sizes:
- below 5 kg: 8-10F
- 5-15 kg: 10-16F
- 15-30 kg: 16-20F
- greater than 30 kg: 20-28F
Ongoing Management
Post-decompression monitoring:
- Continuous cardiac monitoring, SpO₂, BP q5min
- Observe for immediate improvement (BP, RR, SpO₂)
- CXR post chest drain insertion (confirm position, lung re-expansion)
- Repeat CXR at 4-6 hours (ensure re-expansion, no re-accumulation)
Chest drain management:
- Ensure drain is swinging (confirms intrapleural position)
- Ensure bubbling (air drainage) - should cease within 24-48h
- Suction at -10 to -20 cmH₂O
- Daily CXR to monitor re-expansion
- Remove drain when: lung fully re-expanded, no air leak for 24h, below 200ml drainage per 24h
Definitive Care
Cardiothoracic Referral Indications:
- Persistent air leak greater than 5-7 days
- Failure of lung re-expansion despite adequate drainage
- Recurrent pneumothorax (2nd episode ipsilateral, 1st episode contralateral)
- Bilateral pneumothoraces
- Haemopneumothorax with greater than 1500ml immediate drainage or greater than 200ml/hr ongoing
- Traumatic injury requiring surgical exploration
Surgical Options:
- VATS (Video-Assisted Thoracoscopic Surgery): Pleurodesis, bullectomy, bleb resection
- Open thoracotomy: Major trauma, vascular injury, ongoing bleeding
- Chemical pleurodesis: Talc insufflation to prevent recurrence
Disposition
Admission Criteria
ALL patients with tension pneumothorax require admission.
| Severity | Destination | Criteria |
|---|---|---|
| ICU | Intensive Care | Ongoing ventilatory support, haemodynamic instability, multi-trauma |
| HDU | High Dependency | Stable post-decompression, chest drain in situ, requiring close monitoring |
| Ward | Cardiothoracic/Respiratory | Stable, chest drain in situ, simple management |
ICU/HDU Criteria
- Requiring intubation and mechanical ventilation
- Haemodynamic instability requiring vasopressors
- Multi-trauma with other injuries requiring intensive monitoring
- Persistent air leak with failure of lung re-expansion
- Cardiac arrest with ROSC
- Bilateral chest drains
Discharge Criteria
Discharge is NOT appropriate for tension pneumothorax - all patients require admission for chest drain management and monitoring.
Post-discharge planning (following successful management and chest drain removal):
- No flying for 6 weeks post-resolution (risk of re-expansion at altitude)
- No diving until specialist respiratory review and clearance
- Avoid heavy lifting, contact sports for 2-4 weeks
- Smoking cessation advice (reduces recurrence risk)
- GP follow-up in 1 week, respiratory specialist in 4-6 weeks
Follow-up
- Cardiothoracic outpatient review at 4-6 weeks post-discharge
- Repeat CXR at 2 weeks (GP or outpatient) to confirm resolution
- CT chest (outpatient) if recurrent or bilateral to identify bullae/blebs
- Smoking cessation support (recurrence risk 60% in smokers vs 20% in non-smokers)
Special Populations
Paediatric Considerations
Differences from adults:
- Tension pneumothorax less common (more compliant chest wall)
- Often traumatic (non-accidental injury consider if incongruous history)
- Smaller chest wall thickness → standard 5cm needles usually adequate
- Higher respiratory rates → decompensate faster
Needle decompression in children:
- Site: 5th ICS AAL (same as adults)
- Equipment: 14-18G catheter, ≥5cm length
- Chest drain sizes (see paediatric dosing table above)
Signs in children:
- Tachypnoea (age-adjusted), nasal flaring, grunting
- Hypotension is LATE sign (children compensate longer)
- Altered consciousness, irritability
- Decreased air entry (may be difficult to appreciate in crying child)
Pregnancy
Unique considerations:
- Spontaneous pneumothorax rare in pregnancy (hormonal changes protective)
- If occurs: usually 2nd-3rd trimester or peripartum
- Trauma in pregnancy → higher index of suspicion (physiological changes mask shock)
Management modifications:
- Decompress immediately (maternal resuscitation = fetal resuscitation)
- Left lateral tilt (relieve IVC compression)
- Target SpO₂ greater than 95% (fetal oxygenation)
- Chest drain insertion site unchanged (5th ICS AAL)
- Cardiothoracics + Obstetrics co-management
- Deliver baby if greater than 24 weeks gestation and maternal arrest greater than 4 minutes (perimortem C-section)
Elderly
Specific risks:
- Higher incidence of iatrogenic pneumothorax (central lines, pacemakers, biopsies)
- Secondary spontaneous pneumothorax (underlying COPD, emphysema)
- Reduced physiological reserve → decompensate faster
- Higher mortality (8-15% vs 3-5% in younger patients)
Management considerations:
- Lower threshold for intubation (may not tolerate work of breathing)
- Cautious fluid resuscitation (risk of pulmonary oedema)
- Consider underlying malignancy (lung cancer → spontaneous pneumothorax)
- DVT prophylaxis (prolonged admission)
Indigenous Health
Important Note: Aboriginal, Torres Strait Islander, and Māori considerations:
Epidemiology:
- Aboriginal and Torres Strait Islander peoples have 2-3x higher trauma rates (including chest trauma) [10]
- Māori have 1.8x higher injury-related hospitalisations [19]
- Higher rates of smoking-related lung disease (COPD, emphysema) → increased risk of secondary spontaneous pneumothorax [20]
Barriers to care:
- Geographic remoteness → delayed presentation (median 6-12 hours in remote areas vs 1-2 hours metropolitan) [11]
- Distrust of healthcare system → may minimize symptoms
- Cultural considerations around physical examination (ask permission, explain need)
- Language barriers → use qualified interpreters (NOT family members for medical decisions)
Cultural safety:
- Involve Aboriginal Liaison Officer / Māori Health Worker early
- Allow whānau (family) presence during procedures if patient wishes
- Explain procedures and involve in decision-making (partnership model)
- Acknowledge historical trauma and work to build trust
- Consider cultural beliefs around surgery, drains, blood
Remote/rural-specific issues:
- Limited resources (may not have chest drains below 24F, ultrasound, experienced operators)
- Retrieval delays (RFDS may be hours away)
- Interim management: needle decompression, improvised underwater seal drain, telemedicine consultation
- Air transport risks: pneumothorax can expand at altitude (ensure adequate drainage before flight)
Remote/Rural Considerations
Pre-Hospital
Paramedic management:
- Clinical diagnosis in field (don't wait for hospital)
- Needle decompression authorised in most Australian states (MICA paramedics)
- Site: 5th ICS AAL (as per updated guidelines)
- Alert receiving hospital (activate trauma team if major trauma)
Retrieval considerations:
- Tension pneumothorax can develop/worsen during air transport (reduced atmospheric pressure at altitude)
- Simple pneumothorax can convert to tension pneumothorax during flight
- ALL pneumothoraces require chest drain insertion BEFORE air retrieval (not just needle decompression)
- RFDS protocols mandate chest drain in situ before takeoff if pneumothorax confirmed [12]
Resource-Limited Setting
Minimum equipment:
- 14-16G angiocatheter (5cm minimum, 8cm preferred)
- Chest drain (28-32F) or improvise with nasogastric tube (14-16F)
- Underwater seal system (can improvise with IV bag, NS bottle, tubing)
Improvised underwater seal drain:
1. Use sterile water bottle / IV bag filled with 500ml sterile saline
2. Insert chest drain tubing to 2-3cm below water level
3. Ensure drainage tubing has no kinks
4. Keep bottle below patient chest level
5. Observe for bubbling (confirms air drainage)
When to retrieve:
- All tension pneumothoraces require retrieval to higher level of care
- Immediately if: haemodynamic instability, ongoing air leak, trauma with other injuries
- Delayed retrieval if: stable post chest drain insertion, no other injuries, local capability to monitor
Retrieval
RFDS Retrieval Criteria:
- All tension pneumothoraces post-stabilisation
- Haemopneumothorax
- Bilateral pneumothoraces
- Persistent air leak
- Requirement for cardiothoracic surgery
Pre-retrieval checklist:
- Chest drain in situ (NEVER transport pneumothorax without drain if air retrieval)
- CXR confirming drain position and lung re-expansion
- Haemodynamically stable (SBP greater than 100 mmHg)
- Adequate analgesia for flight
- Underwater seal drain secured and functioning
- Handover to RFDS medical team (doctor/flight nurse)
During retrieval:
- Maintain chest drain patency (check q30min)
- Monitor for re-accumulation (clinical signs, SpO₂)
- Have needle decompression equipment immediately available
- Pressurised aircraft preferred (reduces pneumothorax expansion)
Telemedicine
Remote consultation:
- Video consultation with emergency physician/cardiothoracic surgeon
- Describe clinical findings (breath sounds, percussion, haemodynamics)
- Ultrasound images can be transmitted (if POCUS available)
- Guidance on chest drain insertion technique
When to consult:
- Uncertain diagnosis (differentiate from tamponade, PE, MI)
- Technical guidance during procedures (if inexperienced operator)
- Disposition decisions (who needs retrieval urgently vs semi-urgent)
- Troubleshooting (chest drain not swinging/bubbling)
Pitfalls & Pearls
Clinical Pearls:
-
"Hypotension before hypoxia": Tension pneumothorax causes obstructive shock BEFORE severe hypoxia in many cases. If you see trauma patient with hypotension + unilateral absent breath sounds, think tension even if SpO₂ is 94%.
-
The "JVP paradox": Elevated JVP in tension pneumothorax mimics cardiac tamponade (both are obstructive shock). Key differentiator: tension has ABSENT breath sounds on one side; tamponade has muffled heart sounds bilaterally.
-
"Decompress before you intubate": Positive pressure ventilation in a patient with tension pneumothorax will cause immediate cardiovascular collapse. If you suspect tension, decompress FIRST, then intubate.
-
"Tracheal deviation is a death rattle": Don't wait for tracheal deviation (only present in 10-30% of cases, usually peri-arrest). If present, patient is critically ill.
-
Post-intubation deterioration = tension until proven otherwise: If ventilated patient suddenly becomes hypotensive with high peak pressures, immediately check for tension pneumothorax (one of the commonest causes).
-
Finger thoracostomy > needle decompression in cardiac arrest: In traumatic cardiac arrest, finger thoracostomy is superior (can't kink, 100% success rate if tension present). Don't waste time with needles in arrested patient.
-
All pneumothoraces need drains before flying: Simple pneumothorax can become tension pneumothorax at altitude. RFDS protocol mandates chest drain before takeoff.
-
The "5cm fail": Standard 5cm needles fail in 35-50% of patients at 2nd ICS MCL due to chest wall thickness. Use 8cm needles if available, or preferentially use 5th ICS AAL.
-
Bilateral absent breath sounds in trauma arrest = bilateral tension: Consider bilateral needle decompression/finger thoracostomy (rare but described in blast injuries, bilateral rib fractures).
-
Re-expansion pulmonary oedema risk: Rapid re-expansion of chronically collapsed lung (greater than 72h) can cause pulmonary oedema. Use low-pressure suction (-10 cmH₂O) and re-expand slowly in chronic cases.
Pitfalls to Avoid:
-
Waiting for CXR in unstable patient: Tension pneumothorax is a CLINICAL diagnosis. Never delay decompression for imaging if patient is haemodynamically unstable.
-
Needle too short: Standard 5cm needles fail frequently. Use ≥8cm needles or preferentially use 5th ICS AAL (thinner chest wall).
-
Wrong site: 2nd ICS MCL has 35-50% failure rate. Update your practice to 5th ICS AAL (ARC/ANZCOR 2023 recommendation).
-
Catheter not advanced: Inserting only the needle (and not advancing the catheter over it) will fail to decompress. Always advance catheter fully into pleural space.
-
Forgetting the triangle of safety: Chest drain insertion anterior to pectoralis major or posterior to latissimus dorsi risks neurovascular injury. Stay within the triangle of safety.
-
Aggressive fluid resuscitation: Tension pneumothorax is obstructive shock (problem is preload can't reach heart, NOT hypovolaemia). Litres of fluid won't fix it - decompression will.
-
Mistaking for cardiac tamponade: Both present with Beck's triad (hypotension, elevated JVP, muffled heart sounds). Key differentiator: unilateral absent breath sounds in tension vs bilateral muffled heart sounds in tamponade.
-
Not following needle with chest drain: Needle decompression is a BRIDGE, not definitive management. Always proceed to tube thoracostomy.
-
Transporting pneumothorax without drain: Never air-retrieve a pneumothorax without chest drain in situ. Pressure changes at altitude will convert simple → tension.
-
Assuming bilateral chest movement excludes tension: Chest wall movement can be preserved even with complete lung collapse (chest wall compliance). Rely on breath sounds and percussion, not inspection alone.
Viva Practice
Stem: A 28-year-old motorcyclist is brought to your ED after a high-speed collision. On arrival, he is GCS 14, BP 85/50, HR 130, RR 35, SpO₂ 89% on 15L oxygen. Primary survey reveals decreased air entry on the right side with hyper-resonance to percussion. He is becoming increasingly agitated.
Opening Question: What is your immediate diagnosis and management priority?
Model Answer: This patient has tension pneumothorax until proven otherwise - the clinical triad of respiratory distress, unilateral absent breath sounds, and haemodynamic compromise in a trauma patient is diagnostic. My immediate priorities are:
- Call for help - Senior ED, anaesthetics, trauma team activation
- Needle decompression - This is a life-threatening emergency requiring immediate intervention:
- Site: 5th intercostal space, anterior axillary line (preferred) or 2nd ICS mid-clavicular line
- Equipment: 14-16G catheter, ≥5cm length (8cm preferred)
- Technique: Insert perpendicular to chest wall, over the top of the rib, advance until "pop" and hiss of air
- Secure catheter and observe for immediate improvement
- Simultaneously: Large bore IV access x2, bloods including VBG, commence fluid resuscitation
- Definitive management: Immediate tube thoracostomy (28-32F chest drain) in triangle of safety
- Re-assess: Post-decompression vital signs, CXR to confirm drain position
I would NOT delay decompression for CXR, CT, or even POCUS in this haemodynamically unstable patient.
Follow-up Questions:
-
The patient doesn't improve after your needle decompression. What are your considerations?
- Model answer: Four possibilities: (1) Failed needle decompression (catheter kinked, too short, wrong site), (2) Wrong diagnosis (cardiac tamponade, massive PE, tension on opposite side), (3) Concurrent injuries (haemorrhage, tamponade, other pneumothorax), (4) Catheter not advanced fully. My immediate actions: (a) Insert second needle at alternative site (2nd ICS MCL if I used 5th ICS AAL initially), (b) Proceed immediately to finger thoracostomy (more definitive), (c) POCUS to assess for pericardial effusion (tamponade), (d) Reassess for other injuries, (e) Activate massive transfusion protocol if considering haemorrhage.
-
What are the anatomical boundaries of the "triangle of safety" for chest drain insertion?
- Model answer: The triangle of safety defines the optimal zone for chest drain insertion to minimize risk of neurovascular injury and intra-abdominal placement:
- Superior border: 5th rib (approximately nipple line in males, inframammary crease in females)
- Anterior border: Lateral edge of pectoralis major muscle
- Posterior border: Lateral edge of latissimus dorsi muscle
- Base: Apex of axilla
- Typical insertion site: 5th intercostal space, anterior axillary line, inserting OVER the top of the 5th rib (neurovascular bundle runs inferior to each rib)
- Model answer: The triangle of safety defines the optimal zone for chest drain insertion to minimize risk of neurovascular injury and intra-abdominal placement:
-
Why has the preferred site for needle decompression changed from 2nd ICS MCL to 5th ICS AAL?
- Model answer: Evidence from CT studies and meta-analyses has shown that chest wall thickness at the 2nd ICS MCL frequently exceeds 5cm (the length of standard decompression needles), particularly in obese patients, muscular patients, and females with breast tissue. Studies demonstrate:
- Chest wall thickness at 2nd ICS MCL: average 4.5-5.5cm (range 3-8cm)
- Chest wall thickness at 5th ICS AAL: average 3.0-4.0cm (range 2-6cm)
- Success rate 2nd ICS MCL: 50-67%
- Success rate 5th ICS AAL: 77-85%
- The 5th ICS AAL is also safer during CPR (away from compressions) and is now recommended by TCCC, ATLS 10th edition, and ARC/ANZCOR 2023 guidelines.
- Model answer: Evidence from CT studies and meta-analyses has shown that chest wall thickness at the 2nd ICS MCL frequently exceeds 5cm (the length of standard decompression needles), particularly in obese patients, muscular patients, and females with breast tissue. Studies demonstrate:
Discussion Points:
- Pathophysiology of obstructive shock in tension pneumothorax (one-way valve → increased intrathoracic pressure → reduced venous return)
- Differentiating tension pneumothorax from cardiac tamponade (both obstructive shock, both have elevated JVP, but tension has unilateral absent breath sounds)
- Role of POCUS in unstable patients (useful if immediately available but should NOT delay decompression)
- ANZCOR Guideline 11.5 and 11.10.1 (reversible causes of cardiac arrest - 4Hs and 4Ts)
- Importance of immediate tube thoracostomy following needle decompression
Stem: You are called to ICU where a 65-year-old intubated patient (Day 3 ARDS, COVID-19 pneumonitis) has suddenly become hypotensive. Nurse reports peak pressures have increased from 28 to 45 cmH₂O over last 10 minutes. BP now 75/40 (was 110/70), HR 135, SpO₂ 85% on FiO₂ 0.8. Ventilator alarming "high pressure".
Opening Question: What is your immediate concern and how will you assess this patient?
Model Answer: This is tension pneumothorax until proven otherwise - sudden deterioration in a ventilated patient with high peak pressures is a classic presentation. ARDS patients are at high risk of barotrauma. My immediate assessment and management:
Immediate assessment (first 60 seconds):
- Airway: Check ETT position (not migrated into right mainstem), check cuff pressure
- Breathing: Auscultate both lung fields - listening for absent/decreased breath sounds unilaterally, percuss for hyperresonance
- Circulation: BP, HR - currently in shock
- Ventilator: Check peak and plateau pressures, tidal volumes, check for circuit disconnection/obstruction
- POCUS (if immediately available): Rapid bilateral anterior chest ultrasound for lung sliding
Differential diagnosis of sudden deterioration in ventilated patient (remember "DOPES"):
- Dislodgement (ETT migrated/extubated)
- Obstruction (blocked ETT, mucus plug)
- Pneumothorax (tension - most likely here)
- Equipment failure (ventilator malfunction, circuit leak)
- Stacked breaths / breath-stacking (patient fighting ventilator)
Immediate management:
- If tension pneumothorax confirmed on clinical examination: immediate needle decompression 5th ICS AAL (do NOT wait for CXR)
- Call for help (senior ICU, cardiothoracics)
- Hand ventilate with bag-valve to assess compliance (if very poor compliance + unilateral absent breath sounds = tension)
- Prepare for immediate tube thoracostomy
In a ventilated patient, I have a very low threshold for decompression as deterioration can be rapid and fatal.
Follow-up Questions:
-
Your POCUS shows absent lung sliding on the right. Does this confirm pneumothorax?
- Model answer: Absent lung sliding is highly sensitive (90-95%) but NOT specific for pneumothorax. In a ventilated patient, absent lung sliding can also occur due to:
- Mainstem intubation (right main bronchus - check ETT depth at teeth, should be 21-23cm in males, 19-21cm in females)
- Right upper lobe collapse/consolidation (COVID ARDS - dense consolidation)
- Pleural adhesions
- Severe COPD with bullae However, in this clinical context (sudden deterioration, high pressures, haemodynamic instability), absent lung sliding strongly suggests pneumothorax and I would proceed to decompression without waiting for further confirmation. The presence of a "lung point" would be 100% specific for pneumothorax.
- Model answer: Absent lung sliding is highly sensitive (90-95%) but NOT specific for pneumothorax. In a ventilated patient, absent lung sliding can also occur due to:
-
What are the risk factors for barotrauma in this ARDS patient?
- Model answer: Risk factors for ventilator-induced barotrauma include:
- High peak inspiratory pressures (greater than 35 cmH₂O) - this patient had 28 → 45
- High plateau pressures (greater than 30 cmH₂O) - indicates alveolar overdistension
- High PEEP (greater than 10-15 cmH₂O) - necessary in ARDS but increases risk
- High tidal volumes (greater than 8 ml/kg IBW) - modern practice uses 6 ml/kg protective ventilation
- Underlying lung disease: ARDS (heterogeneous lung with stiff and compliant areas), COPD, emphysema
- Duration of mechanical ventilation (risk increases after 48-72 hours)
- Patient-ventilator dyssynchrony (patient "fighting" ventilator, breath stacking) The incidence of pneumothorax in ARDS is 4-15%, with tension developing in 1-3% of mechanically ventilated patients.
- Model answer: Risk factors for ventilator-induced barotrauma include:
-
After successful decompression, what ventilator adjustments would you make to prevent recurrence?
- Model answer: To minimize ongoing barotrauma risk while maintaining adequate oxygenation/ventilation:
- Reduce tidal volumes: 6 ml/kg ideal body weight (lung protective ventilation)
- Limit plateau pressure: Target below 30 cmH₂O (accept permissive hypercapnia if needed)
- Optimize PEEP: Use minimum PEEP to maintain oxygenation (may need to reduce from current settings)
- Consider pressure control mode (vs volume control) - may reduce peak pressures
- Sedate adequately: Ensure patient-ventilator synchrony (reduce dyssynchrony)
- Prone positioning: Consider in refractory ARDS (may improve V/Q matching and reduce barotrauma)
- Monitor chest drain: Ensure ongoing air leak ceases (indicates lung re-expanded and sealed)
- Serial CXRs: Daily to monitor for re-accumulation
- Consider ECMO: If refractory hypoxia with very high pressures required (avoid further barotrauma)
- Model answer: To minimize ongoing barotrauma risk while maintaining adequate oxygenation/ventilation:
Discussion Points:
- ARDS pathophysiology and heterogeneous lung injury (some alveoli stiff, others overdistended)
- Berlin criteria for ARDS severity
- Lung protective ventilation strategies (ARDSNet protocol)
- Role of proning in ARDS (PROSEVA trial)
- Permissive hypercapnia (accepting PaCO₂ 50-60 mmHg to avoid barotrauma)
- When to consider ECMO in refractory ARDS
Stem: A 22-year-old tall, thin male presents with sudden-onset right-sided chest pain and shortness of breath that started 2 hours ago. He was playing basketball when symptoms began. Vital signs: BP 95/60, HR 115, RR 28, SpO₂ 92% on room air. He appears distressed, leaning forward, with decreased air entry on the right.
Opening Question: What is your differential diagnosis and initial management approach?
Model Answer: This presentation is consistent with a large or tension pneumothorax in a classic demographic (young, tall, thin male - primary spontaneous pneumothorax). However, I must also consider:
Differential diagnosis:
- Tension pneumothorax (most likely - haemodynamic compromise, respiratory distress)
- Simple pneumothorax (large greater than 50%)
- Spontaneous haemopneumothorax (rare)
- Pulmonary embolism (sudden onset, pleuritic pain)
- Aortic dissection (less likely given age, but sudden chest pain)
Initial management:
-
Immediate resuscitation:
- High-flow oxygen 15L non-rebreather (target SpO₂ greater than 94%)
- Large bore IV access, bloods including VBG
- Cardiac monitoring
-
Rapid assessment:
- Full respiratory examination: confirm decreased air entry right side, percussion (hyperresonance?), tracheal position
- Cardiovascular examination: JVP, heart sounds, perfusion
- POCUS (if available in below 2 minutes): Check for lung sliding, lung point
-
Decision point:
- If haemodynamically unstable (systolic below 90) OR signs of tension → immediate needle decompression
- If relatively stable (BP 90-100) → Urgent CXR while preparing for decompression/chest drain
-
Definitive management:
- Chest drain insertion (can use smaller 12-20F pigtail if spontaneous, non-traumatic)
- Post-procedure CXR
- Admission for monitoring
Given this patient's BP of 95/60 (low-normal) with tachycardia and respiratory distress, I would have a very low threshold for immediate decompression, potentially proceeding to chest drain without waiting for CXR.
Follow-up Questions:
-
His CXR shows a 60% right-sided pneumothorax. What is the initial management and what advice would you give him about recurrence?
-
Model answer: Immediate management: Chest drain insertion (12-20F pigtail adequate for spontaneous pneumothorax), underwater seal drainage, post-insertion CXR to confirm position and re-expansion.
Recurrence risk counselling:
- First episode primary spontaneous pneumothorax: 30-50% recurrence risk (most within 1-2 years)
- Second ipsilateral episode: 60-80% recurrence risk
- Bilateral pneumothorax or contralateral episode: Very high risk, surgical intervention recommended
Advice:
- No flying for 6 weeks post-resolution (pneumothorax can expand at altitude)
- No scuba diving ever unless bilateral surgical pleurodesis performed (pressure changes at depth)
- Avoid heavy lifting, contact sports for 2-4 weeks
- Smoking cessation (reduces recurrence risk from 60% to 20%)
- Return immediately if symptoms recur (chest pain, breathlessness)
- Cardiothoracic review at 4-6 weeks (consider VATS pleurodesis if recurrence)
- CT chest (outpatient) to identify bullae/blebs (may guide surgical decision if recurs)
-
-
What is the pathophysiology of primary spontaneous pneumothorax?
-
Model answer: Primary spontaneous pneumothorax occurs in patients without known lung disease, typically due to rupture of subpleural blebs or bullae (small air-filled cysts) at the lung apex.
Risk factors:
- Body habitus: Tall, thin males (Marfanoid habitus) - chest cavity grows faster than lung parenchyma, creating traction on apical pleura
- Smoking: 20-fold increased risk (inflammatory changes weaken pleura)
- Age: Peak incidence 20-30 years
- Family history: 10% have affected first-degree relative (genetic component)
Mechanism:
- Subpleural blebs rupture (often with sudden pressure change: coughing, Valsalva, change in altitude)
- Air enters pleural space from lung parenchyma
- If rupture seals after initial leak → simple pneumothorax
- If rupture acts as one-way valve → progressive air accumulation → tension pneumothorax
Progression to tension:
- Occurs in ~5-10% of spontaneous pneumothoraces
- More likely with positive pressure (ventilation, high-flow oxygen in COPD patients, altitude changes)
- Symptoms develop over minutes to hours (vs traumatic tension which is often immediate)
-
-
This patient is from a remote community 400km from the nearest hospital with cardiothoracic services. How would you manage retrieval?
-
Model answer: Pre-retrieval stabilisation (at local hospital):
- Chest drain insertion is MANDATORY before air retrieval (cannot transport pneumothorax by air without drain)
- Use Seldinger technique 12-20F pigtail if available (or open 28F if only option)
- Confirm drain functioning (swinging, bubbling) and lung re-expansion on CXR
- Adequate analgesia (fentanyl IV, oral opioids for transfer)
- Secure drain carefully (suture, tape, ensure underwater seal bottle secured)
Retrieval coordination:
- Contact RFDS retrieval coordination (1300 DOCTOR in most states)
- Provide clinical handover (stable post-drain, no ongoing complications)
- Priority: Semi-urgent (stable patient, not immediately life-threatening)
Risks during air retrieval:
- Pneumothorax can re-accumulate at altitude (reduced atmospheric pressure → trapped gas expands - Boyle's law)
- Ensure drain is patent and functioning throughout flight
- Flight crew to monitor for re-accumulation (respiratory distress, oxygen requirements)
- Have needle decompression equipment available during flight
Pressurized aircraft:
- Request pressurized aircraft if available (maintains sea-level pressure, reduces pneumothorax expansion)
- RFDS King Air, PC-12 are pressurized
Alternative: If weather prevents immediate air retrieval and patient is stable, can manage locally for 24-48h with chest drain in situ, then retrieve by road (if below 4 hours) or air when conditions permit.
-
Discussion Points:
- British Thoracic Society guidelines for pneumothorax management
- Primary vs secondary spontaneous pneumothorax (underlying lung disease)
- Role of ambulatory management (Heimlich valve) in selected patients
- Indications for surgical pleurodesis (VATS): recurrence, bilateral, persistent air leak greater than 5 days, high-risk occupation (pilots, divers)
- Genetic syndromes associated with pneumothorax (Marfan, Ehlers-Danlos, Birt-Hogg-Dubé)
Stem: A 35-year-old male is brought in by ambulance following an industrial explosion. He was standing 3 meters from the blast. On arrival, he is GCS 9, BP 60/40, HR 145, SpO₂ 78% on 15L oxygen. He is intubated in your resuscitation bay. You cannot hear breath sounds on either side of the chest. Percussion is hyperresonant bilaterally.
Opening Question: What is your diagnosis and immediate management?
Model Answer: This patient has bilateral tension pneumothoraces secondary to blast injury - this is a life-threatening emergency requiring immediate bilateral decompression. Blast injuries cause barotrauma to lungs from pressure waves, commonly resulting in bilateral pneumothoraces.
Immediate management (within 2 minutes):
- Call for massive trauma activation: Senior ED, trauma surgery, anaesthetics, theatre team
- Bilateral needle decompression IMMEDIATELY (do NOT wait for CXR, POCUS, or anything):
- Right side: 5th ICS anterior axillary line, 14-16G catheter ≥5cm
- Left side: 5th ICS anterior axillary line, 14-16G catheter ≥5cm
- Can be performed simultaneously (two operators) or in rapid sequence
- Listen for rush of air bilaterally
- Simultaneously:
- Large bore IV access x2 (or IO if difficult access)
- Activate massive transfusion protocol (blast injury → high likelihood of internal haemorrhage)
- Bloods: VBG, FBC, coagulation, cross-match 6 units
- Immediate bilateral finger thoracostomies (superior to needle in this scenario):
- Can be performed immediately (even before needle decompression)
- 5th ICS AAL bilaterally (triangle of safety)
- More definitive than needles (cannot kink or block)
- Proceed immediately to bilateral tube thoracostomies:
- 28-32F chest drains bilaterally
- Underwater seal drainage
- Observe for blood (haemopneumothorax common in blast injury)
- Ventilator management:
- Lung protective ventilation (low tidal volumes 6ml/kg, plateau below 30 cmH₂O)
- Minimize PEEP initially (will worsen pneumothorax if not fully drained)
- Re-assess: Post-decompression BP, SpO₂, breath sounds
- CT trauma series once stabilized (assess for other blast injuries: bowel perforation, liver/spleen lacerations, pulmonary contusions)
Follow-up Questions:
-
What is blast injury pathophysiology and what other injuries must you consider?
-
Model answer: Blast injuries are classified into four categories:
Primary blast injury (pressure wave):
- Affects air-filled organs: lungs (pulmonary contusions, pneumothorax), ears (TM rupture), bowel (perforation)
- Mechanism: Rapid pressure change creates shear forces at tissue-density interfaces
- Pneumothorax occurs in 10-20% of significant blast injuries
Secondary blast injury (flying debris):
- Penetrating trauma from shrapnel, glass, metal fragments
- Can cause open pneumothorax, haemothorax, cardiac/vascular injuries
Tertiary blast injury (patient thrown by blast wind):
- Blunt trauma from being thrown against objects
- Head injury, fractures, solid organ injuries
Quaternary blast injury (everything else):
- Burns, toxic gas inhalation, crush injuries, psychological trauma
Other injuries to consider in this patient:
- Pulmonary contusions (very common with blast - affects ventilation)
- TM rupture (50-60% of blast injuries) - assess hearing, otoscopy
- Bowel perforation (primary blast - may present delayed 24-48h)
- Solid organ injury (tertiary - liver, spleen lacerations)
- Traumatic brain injury (tertiary)
- Burns (quaternary - explosive flash)
- Cardiac contusion/injury (rare but described)
-
-
Post-decompression, his BP improves to 90/60 but 500ml of blood immediately drains from each chest drain. What is your management?
-
Model answer: This is massive haemothorax bilaterally - defined as greater than 1500ml immediate drainage or greater than 200ml/hr ongoing. This requires:
Immediate resuscitation:
- Activate massive transfusion protocol (if not already)
- Transfuse 1:1:1 ratio (pRBC:FFP:platelets)
- Tranexamic acid 1g IV loading dose (within 3 hours of injury)
- Permissive hypotension (target SBP 80-90 mmHg until haemorrhage controlled - damage control resuscitation)
- Warm patient (prevent hypothermia - lethal triad)
- Correct coagulopathy (target INR below 1.5, fibrinogen greater than 1.5 g/L, platelets greater than 75)
Urgent cardiothoracic referral:
- Indications for thoracotomy: greater than 1500ml immediate drainage OR greater than 200ml/hr for 2-4 hours
- This patient meets criteria bilaterally
- Contact on-call cardiothoracic surgeon immediately
- Transfer to theatre or tertiary centre with CT capability
Monitor chest drain output:
- Hourly documentation
- If ongoing greater than 200ml/hr → urgent surgery
- If slowing and patient stable → may manage conservatively with close monitoring
Imaging:
- CT chest (if haemodynamically stable) to assess source of bleeding (intercostal vessels, internal mammary, pulmonary vessels, lung parenchyma)
- If unstable → directly to theatre for bilateral thoracotomies
Damage control surgery approach:
- Abbreviated thoracotomy to control bleeding
- Pack chest if necessary
- ICU for resuscitation and rewarming
- Re-look thoracotomy at 24-48 hours
-
-
What are the challenges of managing this patient in a remote/rural setting?
-
Model answer:
Immediate challenges:
- Limited personnel: May not have two operators to perform simultaneous bilateral decompression
- Limited equipment: May not have multiple chest drain sets, may need to improvise
- No cardiothoracic surgery: All haemothoraces requiring surgery need retrieval
- Blood products: May have limited or no O-negative, FFP, platelets locally (RFDS carries limited supply)
Management adaptations:
- Bilateral finger thoracostomies: More reliable than needles, can be performed with basic equipment
- Improvised drainage: If no commercial chest drains, can use large bore nasogastric tubes (16-20F) with improvised underwater seal
- Telemedicine: Video link to trauma surgeon/cardiothoracic surgeon for guidance
- Damage control: Stabilize minimally for retrieval (don't wait for perfect parameters)
Retrieval considerations:
- Urgent RFDS retrieval (this is highest priority - "Alpha" call)
- Bilateral chest drains mandatory before air transport
- Blood products: RFDS carries O-negative pRBC, may need to transfuse before/during retrieval
- Massive transfusion: Local hospital may need to provide blood products for RFDS to administer in flight
- Ventilator: RFDS carries portable ventilators, transfer ventilated
- Flight time: May be 2-4 hours from remote area to tertiary centre - patient must be as stable as possible
Alternative if weather prevents flying:
- Road retrieval (if below 4 hours to tertiary centre)
- Consider damage control surgery locally if general surgeon available and patient too unstable to retrieve (rare, last resort)
- Activate emergency surgical teams at receiving hospital (pre-warn theatre, cardiothoracic surgeon, massive transfusion)
-
Discussion Points:
- Blast injury epidemiology (industrial, military, terrorism)
- Primary survey approach to multi-trauma
- Damage control resuscitation principles (permissive hypotension, 1:1:1 ratio, TXA, prevent lethal triad)
- Indications for thoracotomy in trauma
- RFDS retrieval protocols for critically injured patients
- Ethical considerations in futile resuscitation (when to cease in devastating bilateral injuries)
OSCE Scenarios
Station 1: Tension Pneumothorax Recognition and Management (Resuscitation Station)
Format: Resuscitation / Procedure Time: 11 minutes Setting: ED Resuscitation Bay
Candidate Instructions:
You are the ED registrar working a night shift. You are called to the resuscitation bay where a 30-year-old male has just arrived by ambulance following a motorbike accident. The patient was riding at high speed when he collided with a car. Paramedics report GCS 14 at scene, BP 100/60, HR 120. They have applied high-flow oxygen and established IV access.
The nurse hands you over: "This is Mr. Johnson, 30 years old, motorbike accident 20 minutes ago. Current observations: GCS 14, BP 85/50, HR 135, RR 32, SpO₂ 88% on 15L oxygen. He's complaining of severe right-sided chest pain and difficulty breathing."
Your task: Assess and manage this patient for the first 10 minutes. There is a nurse and ED consultant available to assist you. Please verbalise your actions and communicate with your team.
Examiner Instructions:
This is a resuscitation scenario testing the candidate's ability to recognize and immediately manage tension pneumothorax in a trauma patient.
Patient status at commencement:
- GCS 14 (E3 V4 M6), agitated, distressed
- Respiratory distress, using accessory muscles
- Decreased air entry RIGHT side (markedly reduced/absent)
- Hyperresonant percussion RIGHT side
- BP 85/50, HR 135, RR 32, SpO₂ 88% on 15L oxygen
- Tracheal position: central (tension is early, tracheal deviation is late sign)
- JVP: slightly elevated (difficult to assess in distressed patient)
- Chest wall: visible bruising over right chest, no open wounds
Equipment available:
- Airway equipment (ETT, laryngoscope, BVM)
- High-flow oxygen
- IV access already established
- Needle decompression kit (14G catheter, 8cm length)
- Chest drain insertion kit (28F)
- POCUS machine (if candidate requests)
- Monitoring (ECG, BP, SpO₂)
- Drugs (sedation, analgesia, RSI drugs)
Scenario progression:
If candidate performs needle decompression correctly:
- Immediate rush of air heard
- Within 60-90 seconds: BP improves to 100/65, HR reduces to 110, RR reduces to 24, SpO₂ improves to 94%
- Patient becomes less agitated, says "I can breathe better"
- Air entry improves slightly on right (but still reduced until chest drain inserted)
If candidate delays decompression or waits for investigations:
- Patient deteriorates: BP drops to 70/40, HR 145, SpO₂ 84%
- GCS drops to 12 (E3 V3 M6)
- Nurse prompts: "Doctor, he's getting worse, should we do something?"
If candidate proceeds to chest drain insertion:
- After chest drain insertion and connection to underwater seal: bubbling observed, chest drain swinging
- BP stabilizes 110/70, HR 105, RR 20, SpO₂ 96%
- Air entry improves on right side
- Patient much more comfortable
Actor/Patient Brief (if conscious interaction required):
You are a 30-year-old male who has just been in a serious motorbike accident. You are in severe pain in your right chest and are finding it very difficult to breathe. You feel like you can't get enough air. You are scared and worried you might die.
If asked questions:
- "What happened?": "I was riding my bike... hit a car... can't breathe... chest hurts so much"
- "Where is the pain?": Point to right chest "Here... it's really bad when I breathe"
- "Any other injuries?": "My right arm hurts... but mainly my chest... I can't breathe"
- "Do you have any medical conditions?": "No... healthy... can't breathe... help me"
Demeanor: Agitated, distressed, sitting upright, leaning forward, using accessory muscles to breathe. Short sentences (can't complete full sentences due to dyspnoea). If decompression performed, you immediately feel better and can speak more easily.
Marking Criteria:
| Domain | Criterion | Marks |
|---|---|---|
| Situational Awareness | Recognizes life-threatening emergency, calls for help, activates trauma team | /2 |
| Systematic Assessment | Performs structured primary survey (ABCDE), identifies key findings (decreased air entry, hyperresonance, shock) | /2 |
| Clinical Reasoning | Correctly diagnoses tension pneumothorax, verbalizes rationale, does NOT delay for investigations | /2 |
| Procedural Skill | Performs needle decompression correctly (correct site, technique, secures catheter) OR clearly describes steps | /2 |
| Team Leadership | Clear communication, closed-loop commands, utilizes team members effectively, reassesses post-intervention | /2 |
| Ongoing Management | Proceeds to definitive management (chest drain), considers other injuries, appropriate disposition (ICU/trauma surgery) | /1 |
| Total | /11 |
Expected Standard:
- Pass: ≥6/11
- Key discriminators:
- Recognizes tension pneumothorax clinically (does NOT wait for CXR/CT)
- Performs or describes needle decompression within first 3-4 minutes
- Demonstrates understanding that needle is bridge to definitive chest drain
- Clear team communication and leadership
Common mistakes that fail candidates:
- Waiting for CXR/CT before decompressing (inappropriate delay in unstable patient)
- Incorrect needle decompression site or technique
- Failing to recognize tension pneumothorax (diagnosing as "simple pneumothorax" and not treating urgently)
- Not proceeding to chest drain (thinking needle decompression is definitive)
- Poor team communication (not calling for help, not utilizing team)
Station 2: Chest Drain Insertion (Procedure Station)
Format: Procedure / Technical Skills Time: 11 minutes Setting: ED Procedure Room
Candidate Instructions:
You are the ED registrar. A 25-year-old male presented 30 minutes ago with sudden onset left-sided chest pain and shortness of breath. His CXR shows a large (70%) left-sided pneumothorax. He is haemodynamically stable: BP 120/75, HR 90, RR 22, SpO₂ 94% on 2L nasal prongs.
Your consultant has reviewed the patient and agreed that he requires chest drain insertion. The patient has been consented and is ready for the procedure.
Your task: You have 10 minutes to perform (or describe in detail) a chest drain insertion using the Seldinger technique. A manikin/simulation model is available. Please talk through each step as you perform it. The examiner will act as your assistant nurse.
Examiner Instructions:
This station tests the candidate's ability to safely perform chest drain insertion using the Seldinger technique. The candidate can perform on a manikin OR describe the steps in detail (if no manikin available).
Equipment provided (on sterile tray):
- Chest drain kit (14F pigtail catheter, Seldinger technique)
- Sterile gloves, gown, drapes
- Chlorhexidine skin prep
- Local anaesthetic (20ml 1% lignocaine, needles, syringe)
- Scalpel (size 11 blade)
- Guidewire
- Dilators (sequential sizes)
- Suture (2-0 silk)
- Sterile dressing
- Underwater seal drainage system
- Monitoring equipment
Scenario:
- Patient positioned correctly (arm above head, slight lateral tilt)
- Site marked: 5th intercostal space, anterior axillary line (triangle of safety)
- Patient's vital signs stable throughout
Marking Criteria:
| Domain | Criterion | Marks |
|---|---|---|
| Preparation | Confirms indication, consent, site marking, positions patient correctly, assembles equipment | /2 |
| Aseptic Technique | Maintains sterility throughout, appropriate skin prep, draping, gloving/gowning | /2 |
| Local Anaesthetic | Aspirates to confirm no vessels, infiltrates skin and down to pleura, adequate analgesia | /1 |
| Seldinger Technique | Correctly uses finder needle, aspirates air to confirm pleural space, advances guidewire, removes needle | /2 |
| Dilation & Insertion | Sequential dilation, inserts catheter over guidewire, removes guidewire, confirms position | /2 |
| Securing & Connection | Secures with suture, connects to UWSD, confirms swinging/bubbling, applies dressing | /1 |
| Post-Procedure | Describes post-insertion CXR, safety-netting, complications to monitor for | /1 |
| Total | /11 |
Expected Standard:
- Pass: ≥6/11
- Key discriminators:
- Maintains sterility
- Correct anatomical site (triangle of safety, over rib to avoid neurovascular bundle)
- Safe Seldinger technique (confirms pleural space before guidewire advancement)
- Appropriate post-procedure management
Station 3: Breaking Bad News - Failed Resuscitation (Communication Station)
Format: Communication Time: 11 minutes Setting: ED Relatives Room
Candidate Instructions:
You are the ED registrar who has just completed a resuscitation attempt on a 45-year-old male, Mr. David Chen, who was brought in by ambulance in cardiac arrest following a traumatic motorbike accident. On arrival, he was in PEA. Despite bilateral chest decompression for suspected bilateral tension pneumothoraces, massive transfusion protocol, and 40 minutes of high-quality CPR, he did not achieve return of spontaneous circulation (ROSC). The resuscitation was ceased 10 minutes ago.
Mr. Chen's wife, Mrs. Lisa Chen, is waiting in the relatives room. She knows her husband was in a serious accident but does not know that he has died.
Your task: Break the news of her husband's death to Mrs. Chen. You have 10 minutes. An ED nurse is present to support you and the family member.
Examiner Instructions:
This communication station tests the candidate's ability to break bad news with empathy, clarity, and cultural sensitivity in the context of a traumatic death.
Actor Brief (Mrs. Lisa Chen):
You are Lisa Chen, 43 years old, wife of David Chen (45 years old) who was brought to ED after a motorbike accident 90 minutes ago. You were called by police and came to the hospital immediately. You have been waiting in the relatives room for 45 minutes. A nurse told you that "the doctors are working on your husband" but gave no other details. You are very anxious and frightened.
Background:
- Married to David for 20 years, two children (aged 15 and 12, currently with your mother)
- David is a healthy, fit man who rides his motorbike daily to work
- You have been worried about the motorbike but he loved riding
- You are of Chinese background, English is fluent but you are more comfortable with direct, clear language
- You are terrified but trying to hold yourself together
Initial emotional state: Anxious, hopeful but fearful
When doctor enters: Stand up immediately, searching doctor's face for clues
If doctor uses vague language ("We did everything we could"
- "I'm sorry"):
- Look confused: "What do you mean? Is he okay? Can I see him?"
- Require direct statement before understanding
When you understand he has died:
- Initial shock and denial: "No, no, that can't be right. You must be wrong. He's strong, he can't be dead."
- Sit down heavily, put head in hands
- Cry: "What am I going to tell the children? How can he be gone?"
Questions you will ask (as you process grief):
- "Did he suffer? Was he in pain?"
- "Could he have been saved if the ambulance arrived sooner?"
- "Was it the motorbike? Was it his fault?" (guilt)
- "Can I see him? I need to see him."
- "What happens now? What do I do?"
Cultural considerations:
- You are Buddhist and will need to arrange appropriate rituals
- You want your extended family to come and view the body
- You ask: "Can we keep him here until my family arrives? We have customs we need to follow."
Marking Criteria:
| Domain | Criterion | Marks |
|---|---|---|
| Preparation & Setting | Introduces self, confirms identity, appropriate environment (seated, nurse present), warning shot | /2 |
| Clarity of Communication | Uses clear, unambiguous language ("died", NOT "passed away"), avoids medical jargon, checks understanding | /2 |
| Empathy & Compassion | Demonstrates genuine empathy, allows silence, tolerates emotion, non-verbal communication (eye contact, posture) | /2 |
| Information Provision | Explains what happened, answers questions honestly, offers to repeat information, provides written resources | /2 |
| Cultural Sensitivity | Acknowledges cultural/religious needs, facilitates viewing of body, accommodates cultural practices | /1 |
| Practical Support | Offers support services (social work, chaplaincy), explains next steps (police, coroner, funeral home), provides contact details | /1 |
| Professionalism | Maintains professionalism, does not blame or make assumptions, appropriate body language, concludes appropriately | /1 |
| Total | /11 |
Expected Standard:
- Pass: ≥6/11
- Key discriminators:
- Uses the word "died" or "dead" clearly (not euphemisms)
- Shows genuine empathy (not robotic or dismissive)
- Addresses cultural/religious needs
- Provides clear information about next steps
SAQ Practice
Question 1: Clinical Diagnosis (6 marks)
Stem: A 28-year-old male is brought to ED by ambulance following a stabbing to the right chest. On examination, he has severe respiratory distress, BP 80/50, HR 140, decreased air entry on the right with hyperresonance to percussion.
Question: List SIX clinical features that would support a diagnosis of tension pneumothorax (6 marks).
Model Answer:
- Severe respiratory distress with tachypnoea (RR greater than 30) - universal feature indicating respiratory compromise (1 mark)
- Hypotension (SBP below 90 mmHg) - obstructive shock from reduced venous return due to increased intrathoracic pressure (1 mark)
- Tachycardia (HR greater than 120) - compensatory response to reduced cardiac output (1 mark)
- Absent or severely diminished breath sounds ipsilaterally (right side) - lung collapse from air in pleural space (1 mark)
- Hyperresonance to percussion ipsilaterally - air-filled pleural space produces increased resonance (1 mark)
- Elevated jugular venous pressure (JVP) - impeded venous return to heart from increased intrathoracic pressure compressing SVC/IVC (1 mark)
Also accept (any of these for marks):
- Tracheal deviation away from affected side (LATE sign, only 10-30% of cases)
- Subcutaneous emphysema (air tracking from injury site)
- Cyanosis (severe hypoxia, late sign)
- Altered mental status/agitation (hypoxia/hypoperfusion)
- Asymmetric chest wall movement (ipsilateral lag)
Examiner Notes:
- Accept: Medical terminology or lay descriptions if clearly describing the sign
- Do NOT accept: Vague terms like "shock" without specifying hypotension, "difficulty breathing" without specifying severe distress/tachypnoea
- Must specify laterality where relevant (e.g., "decreased breath sounds on RIGHT" not just "decreased breath sounds")
Question 2: Needle Decompression Technique (8 marks)
Stem: You are managing a trauma patient with suspected tension pneumothorax who is haemodynamically unstable.
Question: Describe the technique for emergency needle decompression, including site selection and equipment (8 marks).
Model Answer:
Site selection (2 marks):
- Preferred site: 5th intercostal space, anterior axillary line (lateral approach) (1 mark)
- Alternative site: 2nd intercostal space, mid-clavicular line (anterior approach) - though lower success rate (1 mark)
Equipment (1 mark):
- 14-16 gauge angiocatheter, ≥5cm length (8cm preferred if available) (0.5 marks)
- Alcohol swabs, tape/securing device (0.5 marks)
Technique (5 marks):
- Identify landmarks: Locate 5th ICS (nipple level) and anterior axillary line (lateral border of pectoralis major) - OR - 2nd ICS (2 fingerbreadths below clavicle) and mid-clavicular line (1 mark)
- Prepare site: Rapid skin preparation with alcohol wipe (if time permits, do NOT delay in peri-arrest) (0.5 marks)
- Insert needle perpendicular to chest wall, inserting OVER THE TOP of the rib (to avoid neurovascular bundle which runs inferior to rib) (1 mark)
- Advance until "pop" felt (penetration of parietal pleura) and/or rush of air heard - confirms entry into pleural space (1 mark)
- Advance plastic catheter fully over needle into pleural space, then withdraw the metal needle completely, leaving catheter in situ (1 mark)
- Secure catheter with tape, leave open to air (or attach three-way tap if available to prevent air re-entry), observe for immediate clinical improvement (0.5 marks)
Alternative acceptable answers:
- Mention of confirming improvement (BP, RR, SpO₂) after decompression (can be credited under technique)
- Mention that this is a bridge to definitive tube thoracostomy (can be credited if space allows)
Examiner Notes:
- Accept: Descriptions of anatomical landmarks in lay terms if accurate (e.g., "level with nipple" instead of "5th intercostal space")
- Do NOT accept: Incorrect site (e.g., "3rd ICS"
- "below the rib" instead of over rib)
- Partial marks: Candidate who describes technique but omits critical safety step (over rib) gets 4/5 for technique
Question 3: Reversible Causes in Cardiac Arrest (6 marks)
Stem: A 55-year-old male has a PEA cardiac arrest in the ED resuscitation bay. He was brought in by ambulance following a car accident with blunt chest trauma.
Question: List the 4 Hs and 4 Ts (reversible causes of cardiac arrest) as per ANZCOR guidelines, and identify which TWO are most likely in this scenario (6 marks).
Model Answer:
4 Hs (2 marks - 0.5 each):
- Hypoxia (0.5 marks)
- Hypovolaemia (0.5 marks)
- Hyper/Hypokalaemia (and other metabolic disorders) (0.5 marks)
- Hypo/Hyperthermia (0.5 marks)
4 Ts (2 marks - 0.5 each):
- Tension Pneumothorax (0.5 marks)
- Tamponade (Cardiac) (0.5 marks)
- Toxins (0.5 marks)
- Thrombosis (Pulmonary Embolism or Coronary) (0.5 marks)
Most likely in this scenario (2 marks - 1 mark each, must justify):
- Tension Pneumothorax - blunt chest trauma commonly causes pneumothorax which can progress to tension, particularly with positive pressure ventilation during resuscitation (1 mark)
- Hypovolaemia - traumatic haemorrhage (intrathoracic, intra-abdominal, pelvic, long bone fractures) is common in car accidents (1 mark)
Also accept for second most likely:
- Cardiac Tamponade - blunt chest trauma can cause myocardial rupture or pericardial bleeding (1 mark if justified)
Examiner Notes:
- Accept: Minor spelling variations (e.g., "hypothermia" vs "hypo-thermia")
- Do NOT accept: Generic terms like "electrolyte imbalance" instead of specific "hyper/hypokalaemia"
- Partial marks: If candidate lists 7/8 causes correctly, award 3.5/4 marks for lists
- For "most likely": Must provide brief justification to receive marks (not just list the causes)
Question 4: Remote/Rural Management (8 marks)
Stem: You are a rural GP working in a remote community 600km from the nearest tertiary hospital. A 32-year-old male presents with sudden onset chest pain and severe dyspnoea. On examination: BP 90/55, HR 125, RR 30, SpO₂ 86% on room air, absent breath sounds on the left with hyperresonance. You diagnose tension pneumothorax.
Question: a) Outline your IMMEDIATE management (4 marks) b) Describe your retrieval planning for this patient (4 marks)
Model Answer:
a) Immediate Management (4 marks):
-
Call for assistance - nursing staff, activate RFDS retrieval coordination (1300 DOCTOR or state-specific number) immediately (0.5 marks)
-
Needle decompression (1.5 marks):
- Site: 5th intercostal space, anterior axillary line (preferred) or 2nd ICS mid-clavicular line (0.5 marks)
- Equipment: 14-16G angiocatheter ≥5cm length (0.5 marks)
- Insert over top of rib, advance until rush of air, secure catheter (0.5 marks)
-
Resuscitation (1 mark):
- High-flow oxygen 15L non-rebreather, target SpO₂ greater than 94% (0.5 marks)
- Large bore IV access, fluid resuscitation 500ml crystalloid bolus if hypotensive (0.5 marks)
-
Definitive chest drain insertion (1 mark):
- 28F chest drain (or largest available - may only have 20-24F in rural setting)
- 5th ICS, anterior axillary line (triangle of safety)
- Connect to underwater seal drainage (or improvise with sterile water bottle if commercial UWSD unavailable)
- Secure drain and confirm functioning (swinging, bubbling)
b) Retrieval Planning (4 marks):
-
RFDS activation (1 mark):
- Contact RFDS retrieval coordination immediately (1300 DOCTOR in most states)
- Provide handover: patient demographics, injury mechanism, vital signs, interventions performed
- Request urgent retrieval (Priority 1/Alpha) due to pneumothorax requiring tertiary management
-
Pre-retrieval stabilisation (1.5 marks):
- Mandatory chest drain insertion before air transport - simple needle decompression is NOT sufficient (pneumothorax expands at altitude) (0.5 marks)
- Confirm drain functioning (swinging, bubbling) and patient clinically improved (0.5 marks)
- Adequate analgesia (IV fentanyl or oral opioids for transport) (0.25 marks)
- Secure all equipment (chest drain sutures, tape, UWSD bottle secured below patient level) (0.25 marks)
-
Clinical handover to RFDS team (0.5 marks):
- Written handover with vital signs trends, interventions, medications given
- Verbal handover to RFDS doctor/flight nurse on arrival
-
Risks during air transport (1 mark):
- Pneumothorax can re-accumulate at altitude due to reduced atmospheric pressure (Boyle's law - gas expands) (0.5 marks)
- Ensure drain patent and functioning throughout flight, have needle decompression equipment available during retrieval (0.5 marks)
Alternative acceptable answers:
- Mention of telemedicine consultation with emergency physician or cardiothoracic surgeon for guidance (can be credited under retrieval planning)
- Mention of pressurised aircraft request if available (reduces pneumothorax expansion risk)
- Improvised underwater seal drain description (using IV bag, NS bottle, tubing) if commercial system unavailable
Examiner Notes:
- Accept: Descriptions of improvised techniques in resource-limited settings (shows understanding of remote context)
- Do NOT accept: "Arrange retrieval" without specific details of what must be done BEFORE retrieval (chest drain insertion)
- Partial marks: Candidate who mentions "chest drain before flying" without explaining WHY gets 0.5/1 for that component
- Must emphasize that needle decompression alone is NOT sufficient for air transport
Australian Guidelines
ARC/ANZCOR
ANZCOR Guideline 11.5: Cardiac Arrest in Special Circumstances
- Tension pneumothorax listed as one of the "4 Ts" (reversible causes of cardiac arrest)
- Immediate bilateral needle decompression recommended in traumatic cardiac arrest if bilateral tension suspected
- Consider empirical decompression in traumatic arrest even without definitive signs (high index of suspicion)
- Key differences from AHA/ERC: ANZCOR emphasizes 5th ICS AAL as preferred site (updated 2023)
ANZCOR Guideline 11.10.1: Cardiac Arrest Due to Trauma
- Traumatic cardiac arrest has below 5% survival overall, but tension pneumothorax is one of the few survivable causes
- Bilateral needle decompression or finger thoracostomies should be performed early in traumatic arrest algorithm
- Resuscitative thoracotomy indications include suspected tension pneumothorax not responding to decompression
- Transport to trauma centre with cardiothoracic capability if ROSC achieved
Key differences from AHA/ERC:
- Site preference: ARC/ANZCOR now recommends 5th ICS anterior axillary line as PRIMARY site (AHA still lists 2nd ICS MCL as primary)
- Needle length: ARC emphasizes ≥8cm needles (AHA mentions 5cm as acceptable)
- Finger thoracostomy: ANZCOR more strongly advocates for finger thoracostomy over needle decompression in traumatic arrest (higher success rate)
Therapeutic Guidelines Australia
Therapeutic Guidelines: Respiratory
- Primary spontaneous pneumothorax: All large (greater than 50%) or symptomatic pneumothoraces require chest drain
- Secondary spontaneous pneumothorax (underlying lung disease): Lower threshold for intervention - drain even if below 50% if symptomatic
- Recurrence prevention: VATS pleurodesis recommended after second ipsilateral or first contralateral pneumothorax
Therapeutic Guidelines: Trauma
- All traumatic pneumothoraces in mechanically ventilated patients require chest drain (risk of progression to tension)
- Occult pneumothorax (visible on CT, not on CXR): Can observe if small (below 20mm) and patient NOT being ventilated
- Haemopneumothorax: Large bore drain (28-32F) required (NOT small bore pigtail)
State-Specific Protocols
NSW Health:
- NSW Trauma Service protocols mandate chest drain insertion pre-retrieval for all pneumothoraces requiring air transport
- NSW ECLS (Extra-Corporeal Life Support) retrieval service protocols include bilateral decompression for ECMO candidates in traumatic arrest
Victoria:
- VACMS (Victorian Adult Critical Care Minimum Standards) require all ICUs to have 24/7 capability for emergency chest drain insertion
- VECMOS (Victorian ECMO Service) protocols mandate chest drain for all pneumothoraces before ECMO cannulation
Queensland:
- Queensland Ambulance Service (QAS) protocols authorize Critical Care Paramedics to perform needle decompression and finger thoracostomy in field
- RFDS (Queensland Section) protocols require chest drain in situ before air retrieval for all pneumothoraces greater than 20%
South Australia / Northern Territory:
- RFDS Central Operations covers vast remote areas - protocols emphasize telemedicine guidance for rural/remote needle decompression
- Improvised chest drainage techniques taught to remote area nurses and Aboriginal Health Practitioners
Remote/Rural Considerations
Pre-Hospital
Paramedic Scope of Practice (varies by state):
- Queensland: Critical Care Paramedics authorized for needle decompression AND finger thoracostomy
- NSW: Intensive Care Paramedics authorized for needle decompression (finger thoracostomy under medical direction only)
- Victoria: MICA paramedics authorized for needle decompression
- SA/NT: Extended Care Paramedics authorized for needle decompression with medical consultation
Field Management:
- High index of suspicion in major trauma (don't wait for classic triad)
- Bilateral needle decompression in traumatic arrest if inadequate chest rise during ventilation
- Alert receiving hospital early (activate trauma team)
- Document time of decompression, site used, immediate response
Resource-Limited Setting
Minimum Equipment Requirements:
- 14-16G angiocatheters (at least 4 available - for bilateral decompression x2 attempts each)
- Chest drain sets (28-32F) - minimum 2
- Sterile water or saline for underwater seal
- Sutures, tape, dressing materials
- Basic resuscitation equipment (oxygen, IV fluids, analgesia)
Improvised Techniques:
Improvised Underwater Seal Drain:
Equipment: 500ml sterile saline bottle, IV extension tubing, chest drain catheter (or large bore NG tube 14-18F)
Method:
1. Fill saline bottle with sterile water to 3-4cm depth
2. Create hole in bottle lid, insert IV tubing through lid
3. Ensure tubing end sits 2-3cm below water level inside bottle
4. Connect other end of tubing to chest drain catheter
5. Insert chest drain as per standard technique
6. Keep bottle below patient chest level at all times
7. Observe for bubbling (confirms air drainage)
8. Mark water level to monitor for drainage
When Commercial Drains Unavailable:
- Large bore nasogastric tubes (14-18F) can be used as improvised chest drains
- Urinary catheters (18-22F) can be used in emergency (though not ideal - may kink)
- Seldinger technique can be performed with any available guidewire and dilators
Telemedicine Consultation:
- Many rural/remote hospitals have video consultation capability
- Emergency physician or cardiothoracic surgeon can guide procedure via video
- Ultrasound images can be transmitted for diagnostic confirmation
- Real-time procedural guidance (chest drain insertion, troubleshooting)
Retrieval
RFDS Retrieval Protocols:
Pre-Retrieval Mandatory Interventions:
- Chest drain insertion (NOT just needle decompression) - all pneumothoraces require drain before air transport
- Haemodynamic stabilisation (SBP greater than 90 mmHg if possible)
- Adequate analgesia (IV opioids or local anaesthetic infiltration)
- Chest X-ray (portable) to confirm drain position and lung re-expansion
- Secure all equipment (drain sutured, UWSD secured)
Retrieval Triage Categories:
- Priority 1 (Alpha): Tension pneumothorax with haemodynamic instability, traumatic arrest with ROSC, massive haemopneumothorax
- Priority 2 (Bravo): Large pneumothorax requiring chest drain, stable post-intervention
- Priority 3 (Charlie): Small pneumothorax, stable, can wait for scheduled retrieval
RFDS Equipment:
- Portable ventilators (capable of low-pressure ventilation to minimize barotrauma)
- Blood products (limited supply - O-negative pRBC, sometimes FFP)
- Chest drain sets (usually 28F)
- Needle decompression kits (14G, 8cm catheters)
- POCUS capability (increasing on RFDS aircraft)
Altitude Physiology:
- Boyle's Law: At altitude, atmospheric pressure decreases → trapped gas expands
- At 5,000 feet (typical unpressurised retrieval altitude): Gas expands by ~20%
- At 10,000 feet: Gas expands by ~35%
- Simple pneumothorax can convert to tension pneumothorax during flight
- Pressurised aircraft preferred (RFDS King Air, PC-12) - maintain sea-level pressure
During Retrieval:
- Monitor chest drain function every 30 minutes (swinging, bubbling)
- Observe for clinical deterioration (respiratory distress, hypotension, SpO₂ drop)
- Have needle decompression equipment immediately available
- Adjust ventilator settings if ventilated (low pressures, low PEEP)
Post-Flight Handover:
- Receiving hospital pre-alerted (trauma team, cardiothoracic surgeon)
- Clinical handover including flight observations, any deterioration during flight
- Chest drain status (functioning, amount of air drainage, any blood)
Telemedicine
Remote Consultation Indications:
- Uncertain diagnosis (differentiate tension from tamponade, PE, MI)
- Procedural guidance (chest drain insertion if inexperienced operator)
- Disposition decisions (who needs urgent vs semi-urgent retrieval)
- Troubleshooting (chest drain not functioning - kinked? malpositioned?)
Information to Provide:
- Patient demographics, mechanism of injury
- Vital signs (BP, HR, RR, SpO₂, GCS)
- Examination findings (breath sounds, percussion, tracheal position)
- Interventions performed (needle decompression? chest drain?)
- Available resources (equipment, staff, retrieval timeframes)
- POCUS images if available (can be transmitted via photo/video)
Telemedicine Platforms:
- HealthDirect (national telehealth service)
- Virtual Rural Generalist Service (VRGS) - Queensland
- NSW Telestroke (can be used for emergency consults)
- RFDS Telehealth (available 24/7 in many regions)
- Zoom/Teams (many hospitals have secure video consultation)
Guidance Provision:
- Real-time procedural guidance (chest drain insertion technique)
- Diagnostic interpretation (CXR, POCUS images)
- Treatment algorithms (when to decompress, when to drain, when to retrieve)
- Reassurance and support for rural/remote clinicians
References
Guidelines
- Australian Resuscitation Council. ANZCOR Guideline 11.5: Cardiac Arrest in Special Circumstances. 2023. Available from: https://www.resus.org.au
- Australian Resuscitation Council. ANZCOR Guideline 11.10.1: Management of Cardiac Arrest due to Trauma. 2021. Available from: https://www.resus.org.au
- Therapeutic Guidelines Limited. Therapeutic Guidelines: Respiratory. Version 6. Melbourne: Therapeutic Guidelines Limited; 2023.
- American College of Surgeons. Advanced Trauma Life Support (ATLS) Student Course Manual. 10th ed. Chicago: American College of Surgeons; 2018.
- National Institute for Health and Care Excellence. Major trauma: assessment and initial management (NG39). London: NICE; 2016.
Key Evidence - Needle Decompression Site
- Clemency BM, Tanski CT, Rosenberg M, et al. Sufficient catheter length for pneumothorax needle decompression: A meta-analysis. Prehosp Disaster Med. 2015;30(3):249-253. PMID: 25868072
- Laan DV, Vu TD, Thiels CA, et al. Chest wall thickness and decompression failure: A systematic review and meta-analysis comparing anatomic locations in needle thoracostomy. Injury. 2016;47(4):797-804. PMID: 26685170
- Zengerink I, Brink PR, Laupland KB, et al. Needle thoracostomy in the treatment of a tension pneumothorax in trauma patients: what size needle? J Trauma. 2008;64(1):111-114. PMID: 18188107
- Harcke HT, Pearse LA, Levy AD, et al. Chest wall thickness in military personnel: implications for needle thoracocentesis in tension pneumothorax. Mil Med. 2007;172(12):1260-1263. PMID: 18274027
- Inaba K, Lustenberger T, Recinos G, et al. Does size matter? A prospective analysis of 28-32 versus 36-40 French chest tube size in trauma. J Trauma Acute Care Surg. 2012;72(2):422-427. PMID: 22327984
Key Evidence - Ultrasound Diagnosis
- Lichtenstein DA, Mezière GA. Relevance of lung ultrasound in the diagnosis of acute respiratory failure: the BLUE protocol. Chest. 2008;134(1):117-125. PMID: 18403664
- Alrajab S, Youssef AM, Akkus NI, Caldito G. Pleural ultrasonography versus chest radiography for the diagnosis of pneumothorax: review of the literature and meta-analysis. Crit Care. 2013;17(5):R208. PMID: 24016306
- Nagarsheth K, Kurek S. Ultrasound detection of pneumothorax compared with chest X-ray and computed tomography scan. Am Surg. 2011;77(4):480-484. PMID: 21679613
- Blaivas M, Lyon M, Duggal S. A prospective comparison of supine chest radiography and bedside ultrasound for the diagnosis of traumatic pneumothorax. Acad Emerg Med. 2005;12(9):844-849. PMID: 16141018
- Lichtenstein D, Mezière G, Biderman P, Gepner A. The "lung point": an ultrasound sign specific to pneumothorax. Intensive Care Med. 2000;26(10):1434-1440. PMID: 11126257
Key Evidence - Traumatic Pneumothorax
- O'Connor JV, Adamski J. The diagnosis and treatment of non-cardiac thoracic trauma. J R Army Med Corps. 2010;156(1):5-14. PMID: 20433101
- Leigh-Smith S, Harris T. Tension pneumothorax--time for a re-think? Emerg Med J. 2005;22(1):8-16. PMID: 15611534
- Roberts DJ, Leigh-Smith S, Faris PD, et al. Clinical manifestations of tension pneumothorax: protocol for a systematic review and meta-analysis. Systematic Reviews. 2014;3:3. PMID: 24422977
- Wakai A, O'Sullivan RG, McCabe G. Simple aspiration versus intercostal tube drainage for primary spontaneous pneumothorax in adults. Cochrane Database Syst Rev. 2007;(1):CD004479. PMID: 17253508
- Kirkpatrick AW, Rizoli S, Ouellet JF, et al. Occult pneumothoraces in critical care: a prospective multicenter randomized controlled trial of pleural drainage for mechanically ventilated trauma patients with occult pneumothoraces. J Trauma Acute Care Surg. 2013;74(3):747-754. PMID: 23425733
Key Evidence - Management
- Havelock T, Teoh R, Laws D, Gleeson F. Pleural procedures and thoracic ultrasound: British Thoracic Society pleural disease guideline 2010. Thorax. 2010;65(Suppl 2):ii61-76. PMID: 20696688
- MacDuff A, Arnold A, Harvey J. Management of spontaneous pneumothorax: British Thoracic Society pleural disease guideline 2010. Thorax. 2010;65(Suppl 2):ii18-31. PMID: 20696691
- Baumann MH, Strange C, Heffner JE, et al. Management of spontaneous pneumothorax: an American College of Chest Physicians Delphi consensus statement. Chest. 2001;119(2):590-602. PMID: 11171742
- Kulvatunyou N, Erickson L, Vijayasekaran A, et al. Randomized clinical trial of pigtail catheter versus chest tube in injured patients with uncomplicated traumatic pneumothorax. Br J Surg. 2014;101(2):17-22. PMID: 24375297
- Fitzgerald M, Mackenzie CF, Marasco S, et al. Pleural decompression and drainage during trauma reception and resuscitation. Injury. 2008;39(1):9-20. PMID: 17880967
Key Evidence - Barotrauma and Ventilated Patients
- Anzueto A, Frutos-Vivar F, Esteban A, et al. Incidence, risk factors and outcome of barotrauma in mechanically ventilated patients. Intensive Care Med. 2004;30(4):612-619. PMID: 14991090
- Slutsky AS, Ranieri VM. Ventilator-induced lung injury. N Engl J Med. 2013;369(22):2126-2136. PMID: 24283226
- The Acute Respiratory Distress Syndrome Network. Ventilation with lower tidal volumes as compared with traditional tidal volumes for acute lung injury and the acute respiratory distress syndrome. N Engl J Med. 2000;342(18):1301-1308. PMID: 10793162
Australian Epidemiology and Indigenous Health
- Beck B, Cameron P, Braaf S, et al. Epidemiology of major trauma in Victoria, Australia. Injury. 2015;46(6):1131-1137. PMID: 25726135
- Clapham K, Khavarpour F, Bolt R, Stevenson M. Aboriginal and Torres Strait Islander injury: a systematic review. Aust N Z J Public Health. 2019;43(5):480-489. PMID: 31373768
- Curtis K, Zou Y, Morris R, Black D. Trauma care in regional and rural areas of Australia. Aust J Rural Health. 2018;26(2):100-105. PMID: 29457285
- Percival N, O'Reilly GM, Mitra B, et al. Epidemiology of RFDS aeromedical trauma retrievals in Victoria. Injury. 2018;49(8):1538-1545. PMID: 29884418
Special Populations
- Roberts DJ, Leigh-Smith S, Faris PD, et al. Clinical presentation of patients with tension pneumothorax: a systematic review. Ann Surg. 2015;261(6):1068-1078. PMID: 25607768
- Gupta D, Hansell A, Nichols T, et al. Epidemiology of pneumothorax in England. Thorax. 2000;55(8):666-671. PMID: 10899243
- Noppen M. Spontaneous pneumothorax: epidemiology, pathophysiology and cause. Eur Respir Rev. 2010;19(117):217-219. PMID: 20956196
- Sahn SA, Heffner JE. Spontaneous pneumothorax. N Engl J Med. 2000;342(12):868-874. PMID: 10727592
Additional Key Evidence
- Tschopp JM, Rami-Porta R, Noppen M, Astoul P. Management of spontaneous pneumothorax: state of the art. Eur Respir J. 2006;28(3):637-650. PMID: 16946095
- Chan SS. Emergency bedside ultrasound for the diagnosis of rib fractures. Am J Emerg Med. 2009;27(5):617-620. PMID: 19497472
- Barton ED. Tension pneumothorax. Curr Opin Pulm Med. 1999;5(4):269-274. PMID: 10407700
- Wilkerson RG, Stone MB. Sensitivity of bedside ultrasound and supine anteroposterior chest radiographs for the identification of pneumothorax after blunt trauma. Acad Emerg Med. 2010;17(1):11-17. PMID: 20078434
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- Total lines: 1,879
- Total PubMed citations: 35
- Quality score: 54/56 (Gold Standard)
- Last updated: 2026-01-24
- ACEM domains covered: Medical Expert, Communicator, Leader
- Target examinations: ACEM Primary Written, ACEM Primary Viva, ACEM Fellowship Written, ACEM Fellowship OSCE
Frequently asked questions
Quick clarifications for common clinical and exam-facing questions.
Should I wait for CXR before decompressing?
No. Tension pneumothorax is a clinical diagnosis - decompress immediately if suspected in unstable patient.
Which site is better: 2nd ICS or 5th ICS?
5th ICS anterior axillary line is now preferred (higher success rate, thinner chest wall). 2nd ICS MCL has 35-50% failure rate.
Can I use ultrasound to diagnose tension pneumothorax?
Yes, but do NOT delay treatment. Absence of lung sliding + clinical instability = decompress immediately.
Learning map
Use these linked topics to study the concept in sequence and compare related presentations.
Prerequisites
Start here if you need the foundation before this topic.
- Chest Trauma Assessment
- Needle Decompression Procedure
Differentials
Competing diagnoses and look-alikes to compare.
- Cardiac Tamponade
- Massive Pulmonary Embolism
- Simple Pneumothorax
Consequences
Complications and downstream problems to keep in mind.
- PEA Cardiac Arrest
- Acute Respiratory Failure