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Emergency Medicine
General Surgery
EMERGENCY

Abdominal Trauma

High EvidenceUpdated: 2025-12-22

On This Page

Red Flags

  • Hemodynamic instability with positive FAST
  • Peritonitis (guarding, rigidity)
  • Evisceration
  • Free air on imaging
  • Gunshot wound to abdomen
  • Seatbelt sign with mechanism
Overview

Abdominal Trauma

1. Clinical Overview

Summary

Abdominal trauma refers to injury to the abdomen from external forces, classified as blunt (MVA, falls, assault) or penetrating (stab wounds, gunshot wounds). It is a leading cause of preventable death in trauma patients. The abdomen is the third most commonly injured region. Blunt trauma typically injures solid organs (spleen, liver, kidney), while penetrating trauma has higher rates of hollow viscus injury. Management ranges from non-operative observation for stable solid organ injuries to emergent laparotomy for hemodynamic instability or peritonitis.

Key Facts

  • Incidence: 10% of trauma deaths; abdomen is 3rd most injured region
  • Blunt trauma: 80-90% of abdominal trauma cases
  • Most commonly injured (blunt): Spleen, followed by liver
  • Most commonly injured (penetrating): Small bowel
  • Laparotomy rate: GSW approximately 80-90%, stab wounds approximately 25-30%
  • Key decision: Hemodynamic stability determines operative vs non-operative management

Clinical Pearls

FAST Positive + Unstable = Theatre: Do not delay for CT. Positive FAST in a hemodynamically unstable patient mandates emergent laparotomy.

Seatbelt Sign: Ecchymosis across abdomen from seatbelt increases risk of hollow viscus and mesenteric injury. Have high suspicion even if initial imaging appears reassuring.

Delayed Splenic Rupture: Can occur hours to weeks after injury. Warn patients about warning signs at discharge.

Why This Matters Clinically

Missed abdominal injury leads to preventable death. Rapid identification using FAST, appropriate resuscitation with damage control principles, and timely operative intervention when indicated saves lives. Non-operative management of solid organ injuries has transformed outcomes in stable patients.


2. Epidemiology

Incidence & Prevalence

  • Incidence: Abdomen involved in 10% of trauma deaths
  • Blunt vs Penetrating: 80-90% blunt in developed countries
  • Trend: Decreasing mortality with improved trauma systems

Demographics

FactorDetails
AgePeak in young adults (MVA, assault); bimodal with elderly falls
SexMale predominance (2-3:1)
GeographyUrban: higher penetrating; Rural: higher blunt

Risk Factors

Non-Modifiable:

  • Age extremes (poor reserve)
  • Anticoagulation use

Modifiable:

Risk FactorRelative Risk
Alcohol/drug intoxication2-3x
Non-seatbelt use3-4x for serious injury
High-risk occupation/activitiesVariable

3. Pathophysiology

Mechanism

Blunt Trauma Mechanisms:

Step 1: The Physics of Collision (Kinematics)

  • Kinetic Energy: $KE = \frac12mv^2$. Velocity is the key determinant of damage. Doubling speed quadruples energy.
  • Cavitation: In solid organs, the energy wave creates a temporary cavity, tearing parenchyma far from the impact site.
  • Shearing: Ligamentous attachments (Ligamentum Tetes, Mesentery) hold organs in place while the body decelerates. This tears vessels (avulsion).

Step 2: The "Paper Bag" Effect (Burst Injury)

  • Mechanism: Sudden compression of a gas/fluid-filled hollow viscus (stomach, bladder, bowel).
  • Result: Sudden spike in intra-luminal pressure > wall tension -> Blow out.
  • Lap Belt Injury: Compression of bowel against the lumbar spine ("Chance Fracture" association).

Step 3: The Lethal Triad (Physiological Collapse)

  • Hypothermia: Blood loss + exposure + cold fluids. Enzymes (clotting cascade) fail at less than 35°C.
  • Acidosis: Hypoperfusion -> Anaerobic respiration -> Lactate. Acidosis inhibits thrombin generation.
  • Coagulopathy: Consumption of factors + Dilution + Fibrinolysis (driven by Protein C activation).
  • Result: "Bloody Vicious Cycle" -> Irreversible shock.

Step 4: Inflammatory Response

  • Tissue Trauma: Releases DAMPs (Damage Associated Molecular Patterns).
  • SIRS: Systemic inflammatory response -> Capillary leak -> Pulmonary Edema (ARDS) + Multi-Organ Failure.

Penetrating Trauma Mechanisms:

TypeVelocityInjury Pattern
Stab woundLowDirect tissue disruption along tract
Gunshot woundHighCavitation, blast effect, unpredictable trajectory

Classification

Anatomical Regions:

RegionBoundariesContents
Intrathoracic abdomenDiaphragm to costal marginsLiver, spleen, stomach
True abdomenCostal margins to iliac crestsBowel, mesentery
RetroperitoneumBehind peritoneumKidneys, pancreas, duodenum, great vessels
PelvisBelow pelvic brimBladder, rectum, iliac vessels

AAST Organ Injury Scale (Spleen Example):

GradeDescription
ISubcapsular hematoma less than 10%; laceration less than 1cm
IISubcapsular hematoma 10-50%; laceration 1-3cm
IIISubcapsular hematoma greater than 50% or expanding; laceration greater than 3cm
IVLaceration involving segmental or hilar vessels
VShattered spleen; hilar vascular injury

Anatomical Considerations

  • Intrathoracic abdomen (liver, spleen) protected by ribs but injured in thoracoabdominal trauma
  • Retroperitoneal injuries (pancreas, duodenum, kidneys) may have minimal initial findings
  • Hollow viscus injuries present late with peritonitis

4. Clinical Presentation

Symptoms

Typical Presentation:

Atypical Presentations:

Signs

Red Flags

[!CAUTION] Red Flags — Immediate surgical consultation if:

  • Hemodynamic instability with abdominal injury
  • Positive FAST with shock
  • Peritonitis (guarding, rigidity, rebound)
  • Evisceration
  • GSW to abdomen
  • Free air on imaging
  • Impaled object

Abdominal pain (60-70%)
Common presentation.
Nausea/vomiting (30%)
Common presentation.
Inability to urinate (bladder injury)
Common presentation.
5. Clinical Examination

Structured Approach

General:

  • Hemodynamic status (HR, BP, lactate)
  • Level of consciousness
  • Evidence of shock (pale, clammy, confused)

Abdominal Examination:

  • Inspect: wounds, seatbelt sign, distension, evisceration
  • Palpate: tenderness, guarding, rigidity
  • Percussion: shifting dullness (blood), tympany (free air)
  • Auscultate: absent bowel sounds, bowel sounds in chest (diaphragm rupture)

Adjuncts:

  • Log roll: inspect back for wounds
  • Perineal exam: blood at meatus (urethral injury)
  • Rectal exam: high-riding prostate, gross blood

Special Tests

TestTechniquePositive FindingSensitivity/Specificity
FAST ultrasound4 views: RUQ, LUQ, subxiphoid, pelvisFree fluid in Morrison's/splenorenal/pelvis85-95% / 95%+ for hemoperitoneum
Log rollTurn patient to inspect posteriorWounds, tenderness, deformityN/A
Rectal examDigital examinationBlood, high-riding prostate, loss of toneN/A

6. Investigations

First-Line (Bedside)

  • FAST ultrasound — Rapid bedside assessment for free fluid
  • Observations — HR, BP, RR, GCS, SpO2
  • Urine dipstick — Hematuria suggests GU injury

Laboratory Tests

TestExpected FindingPurpose
FBCHb may be normal initially; later dropsSerial trending for blood loss
CoagulationPT/APTT/INRBaseline; guide reversal if anticoagulated
Group & Save / CrossmatchType specific or O-negPrepare for transfusion
U&EsBaseline creatinineAssess renal function
LactateElevated = tissue hypoperfusionMarker of shock severity
Base deficitMore negative = worse shockPrognostic marker

Imaging

ModalityFindingsIndication
FASTFree fluid (blood)Unstable trauma; screening
CT abdomen/pelvis with IV contrastSolid organ injury, active extravasation, free air, bowel thickeningStable patients; gold standard
CXRDiaphragm rupture, pneumoperitoneumPart of trauma series
Pelvic XRPelvic fractureAssociated retroperitoneal hemorrhage

Diagnostic Criteria

  • Positive FAST: Free fluid in any view
  • Peritonitis: Clinical diagnosis (guarding, rigidity, rebound tenderness)
  • DPL Positive: greater than 10mL gross blood, RBC greater than 100,000/mm³, WBC greater than 500/mm³

7. Management

Management Algorithm

                  Trauma Call Activation
                           ↓
┌─────────────────────────────────────────────────────┐
│              PRIMARY SURVEY (ABCDE)                 │
│  - Airway secured?                                  │
│  - Breathing: Decompress Tension Pneumothorax       │
│  - Circulation: 2x Large Bore IV. TXA 1g.           │
│  - Massive Transfusion Protocol (MTP) if shock      │
└─────────────────────────────────────────────────────┘
                           ↓
┌─────────────────────────────────────────────────────┐
│             HEMODYNAMIC ASSESSMENT                  │
│  (BP, HR, Response to Volume, Lactate)              │
├────────────────────┬────────────────────────────────┤
│      UNSTABLE      │             STABLE             │
│ (Transient/Non-R)  │      (Responder, SBP >100)     │
├────────────────────┼────────────────────────────────┤
│        ↓           │                ↓               │
│    eFAST Scan      │         CT Pan-Scan            │
│   (Trauma Bay)     │      (Arterial/Portal)         │
├───────┬────────────┤                │               │
│ POS   │    NEG     │        ┌───────┴──────┐        │
│  ↓    │     ↓      │        ↓              ↓        │
│ LAP   │ Search for │   Solid Organ    Hollow Viscus │
│ NOW   │ other      │      Injury         Revascular │
│       │ bleeding   │        ↓              ↓        │
│       │ (Pelvis?)  │    Grade? Stable?   LAPAROTOMY │
└───────┴─────┬──────┘        ↓                       │
              ↓       ┌───────┴──────┐                │
      Stabilize + CT  │     NOM      │                │
                      │ (Observation)│                │
                      └───────┬──────┘                │
                              ↓                       │
                       Contrast Blush?                │
                              ↓                       │
                       ANGIOEMBOLIZATION              │

Acute/Emergency Management

Damage Control Resuscitation:

  1. Activate massive transfusion protocol if hemorrhagic shock
  2. 1:1:1 ratio (pRBC : FFP : Platelets)
  3. Tranexamic acid 1g IV (within 3 hours of injury)
  4. Permissive hypotension: target SBP 80-90mmHg (unless TBI)
  5. Limit crystalloid (worsens coagulopathy)
  6. Correct hypothermia (target temperature greater than 36°C)
  7. Maintain ionized calcium greater than 1.0 mmol/L

Conservative Management

Best Medical Therapy (for all patients):

  • Pain control
  • VTE prophylaxis once hemostasis achieved
  • Serial abdominal examinations
  • Serial hemoglobin monitoring

Medical Management

Drug ClassDrugDoseDuration
AntifibrinolyticTranexamic acid1g IV then 1g over 8hSingle course
AnalgesiaMorphineTitrate to effectAs needed
VTE prophylaxisEnoxaparin40mg SC dailyOnce bleeding controlled

Procedure Spotlight: Damage Control Surgery (DCS)

The Strategy of Survival.

  • Concept: Restore physiology, not anatomy. Limit operative time (less than 90 mins) to prevent the Lethal Triad.
  • Phases:
    1. Selection: Decision made early (pH less than 7.2, Temp less than 34, Transfusion greater than 10u).
    2. Phase 1 (The Operation):
      • Control Haemorrhage: Packing 4-quadrants. Ligating bleeders. Shunting vessels.
      • Control Contamination: Staple off bowel holes. No anastomoses!
      • Closure: Temporary Abdominal Closure (TAC) / Bogota Bag / Vac Pac.
    3. Phase 2 (ICU): Rewarm, correct acidosis, correct coagulopathy.
    4. Phase 3 (Re-look): Return to theatre (24-48h later) for definitive repair when stable.

Procedure Spotlight: REBOA

Resuscitative Endovascular Balloon Occlusion of the Aorta.

  • Concept: Internal aortic clamp via the femoral artery.
  • Indication: Sub-diaphragmatic life-threatening haemorrhage (Abdo/Pelvis) in arrest/peri-arrest.
  • Zones:
    • Zone 1: Descending thoracic aorta (For abdomen bleed).
    • Zone 3: Infrarenal aorta (For pelvic bleed).
  • Pros: Increases cerebral/coronary perfusion. Stops distal bleeding.
  • Cons: Ischaemia distal to balloon (max time 30-60 mins). Reperfusion injury.

Procedure Spotlight: Diagnostic Peritoneal Lavage (DPL)

The "Lost Art" (Used if FAST/CT unavailable).

  • Technique: Catheter enters infra-umbilical. Aspirate.
  • Positive: >10ml Gross blood = Positive.
  • Lavage: If no blood, infuse 1L saline. Drain. Lab count >100k RBCs = Positive.

Surgical Management

Indications for Emergent Laparotomy:

  • Hemodynamic instability with positive FAST
  • Peritonitis
  • Evisceration
  • Impaled object (remove in OR)
  • Free air on imaging
  • GSW to abdomen (most cases)
  • Failed non-operative management

Non-Operative Management (NOM):

CriteriaDetails
EligibleHemodynamically stable, no peritonitis, solid organ injury
SettingTrauma center with 24/7 OR capability
MonitoringICU or monitored bed, serial exams Q2-4h, serial Hb Q6h
InterventionAngioembolization if contrast blush on CT
FailureOngoing transfusion (greater than 4 units/24h), worsening exam

Disposition

  • Admit ICU if: Unstable, high-grade injury (Grade III+), post-operative
  • Admit ward if: Low-grade injury (Grade I-II), stable, reliable exam
  • Discharge if: Negative workup, stable, clear return precautions
  • Follow-up: Repeat imaging in 5-7 days for high-grade splenic/liver injuries; activity restriction 4-6 weeks

8. Complications

Immediate (Minutes-Hours)

ComplicationIncidencePresentationManagement
ExsanguinationVariableHemodynamic collapseMassive transfusion, emergent surgery
Cardiac arrestRarePEA/asystoleResuscitative thoracotomy consideration

Early (Days)

Abdominal Compartment Syndrome (ACS)

The Killer in the ICU.

  • Definition: Intra-abdominal pressure (IAP) >20 mmHg WITH new organ dysfunction.
  • Mechanism:
    • Edema (Capillary leak from resuscitation).
    • Bleeding (Ongoing).
    • Packing (Foreign bodies occupying space).
  • Pathology:
    • Kidney: Renal vein compression -> Acute Kidney Injury (Anuria).
    • Lungs: Diaphragm pushed up -> High ventilatory pressures, Hypoxia.
    • Heart: Decreased venous return (IVC compression) -> Hypotension.
    • Brain: Increased ICP (failed venous drainage).
  • Treatment: MEDICAL EMERGENCY.
    • Paralysis/Sedation (relax muscles).
    • NG tube decompression.
    • Surgical Decompression: Open the abdomen.

Missed Hollow Viscus Injury

  • The "Seatbelt" Trap: Bowel gets crushed. Initial CT may be normal.
  • Signs: Tachycardia, increasing pain, fever at 24-48h.
  • Management: High index of suspicion. Diagnostic Laparoscopy if unsure.

Late (Weeks-Months)

  • Delayed splenic rupture: 1-2 weeks post-injury
  • Pseudocyst (pancreatic injury): Weeks post-injury
  • Post-splenectomy infection (OPSI): Encapsulated organisms; requires vaccination
  • Incisional hernia: Post-laparotomy
  • Adhesive small bowel obstruction: Months to years post-operatively

9. Prognosis & Outcomes

Natural History

  • Untreated solid organ hemorrhage leads to exsanguination and death
  • Untreated hollow viscus injury leads to peritonitis, sepsis, and death
  • Delayed recognition increases mortality significantly

Outcomes with Treatment

VariableNOM SuccessOperative
Splenic injury (Grade I-II)greater than 95%N/A
Splenic injury (Grade III-V)75-90% (with angioembolization)Splenectomy mortality 1-5%
Hepatic injurygreater than 90%Variable by injury
Hollow viscus injuryN/ADepends on contamination, delay

Prognostic Factors

Good Prognosis:

  • Early recognition and intervention
  • Single organ injury
  • Low-grade injury
  • Young, fit patient
  • Rapid access to trauma center

Poor Prognosis:

  • Multi-organ injury
  • Delayed presentation
  • Elderly or comorbid patient
  • Associated head injury
  • Coagulopathy (lethal triad)

10. Evidence & Guidelines

Key Guidelines

  1. ATLS 10th Edition (2018) — American College of Surgeons. Standard trauma management principles. ACS ATLS
  2. Eastern Association for the Surgery of Trauma (EAST) — Practice management guidelines for solid organ injuries. EAST Guidelines
  3. Western Trauma Association (WTA) — Critical decisions in trauma. WTA

Landmark Trials

PROPPR Trial (2015) — Transfusion ratios in trauma

  • 680 patients with severe trauma
  • Key finding: 1:1:1 ratio (pRBC:FFP:Platelets) achieved hemostasis faster than 1:1:2
  • Clinical Impact: Established 1:1:1 as standard in massive transfusion

CRASH-2 Trial (2010) — Tranexamic acid in trauma

  • 20,211 patients
  • Key finding: TXA reduced mortality if given within 3 hours (RR 0.85)
  • Clinical Impact: TXA now standard in bleeding trauma

NOM for Solid Organ Injury Studies — Multiple observational series

  • Demonstrated high success rates (greater than 90%) for non-operative management of stable solid organ injuries
  • Clinical Impact: NOM is now standard of care for stable patients

Evidence Strength

InterventionLevelKey Evidence
1:1:1 transfusion ratio1bPROPPR: No mortality diff at 24h, but significantly less exsanguination death at 3h.
Tranexamic acid (within 3h)1aCRASH-2: Reduced all-cause mortality (14.5% vs 16%) with no increase in vascular occlusive events.
FAST for blunt trauma2aCochrane: High specificity (>95%) but variable sensitivity. Good rule-in, bad rule-out.
NOM for solid organ injury2aEAST Guidelines: >98% success for Grade I-II Spleen. 80-90% for Grade IV-V.
REBOA vs Thoracotomy2bUK-REBOA: Current evidence suggests no survival benefit over standard care (operator dependent).

11. Patient/Layperson Explanation

What is abdominal trauma?

Abdominal trauma means your belly has been injured, either from a direct blow (like in a car crash or fall) or from something sharp (like a knife or gunshot). Inside your belly are important organs like your liver, spleen, kidneys, and intestines. These can be bruised, torn, or bleeding.

Why does it matter?

Some injuries cause internal bleeding that you cannot see. This can be very dangerous if not found quickly. Other injuries can release infection into your belly (if the intestines are torn). Both situations can be life-threatening.

How is it treated?

  1. Observation: For smaller injuries to solid organs (liver, spleen, kidney), we often watch you closely in hospital without surgery. We check your blood levels and examine you regularly.
  2. Blood transfusion: If you are bleeding significantly, we give you blood transfusions.
  3. Surgery: If bleeding is severe or cannot be controlled, or if your intestines are injured, you will need an operation.
  4. Special procedures: Sometimes we can stop bleeding with a procedure done through your blood vessels (angioembolization) without a big surgery.

What to expect

  • If you had a solid organ injury treated without surgery, you will need to rest and avoid contact sports and strenuous activity for 4-6 weeks
  • You may need a follow-up scan to check healing
  • Most people recover fully

When to seek help

Return to hospital immediately if you experience:

  • Worsening abdominal pain
  • Dizziness, fainting, or feeling lightheaded
  • Blood in your urine or stool
  • Shoulder tip pain (can indicate internal bleeding)
  • Fever
  • Feeling unwell or "not right"

12. References

Primary Guidelines

  1. American College of Surgeons Committee on Trauma. Advanced Trauma Life Support (ATLS) Student Course Manual. 10th ed. 2018.
  2. Stassen NA, et al. Selective nonoperative management of blunt splenic injury: an Eastern Association for the Surgery of Trauma practice management guideline. J Trauma Acute Care Surg. 2012;73(5 Suppl 4):S294-300. PMID: 23114485

Key Trials

  1. Holcomb JB, et al. Transfusion of plasma, platelets, and red blood cells in a 1:1:1 vs a 1:1:2 ratio and mortality in patients with severe trauma (PROPPR). JAMA. 2015;313(5):471-482. PMID: 25647203
  2. CRASH-2 trial collaborators. Effects of tranexamic acid on death, vascular occlusive events, and blood transfusion in trauma patients with significant haemorrhage (CRASH-2). Lancet. 2010;376(9734):23-32. PMID: 20554319
  3. Moore EE, et al. Organ injury scaling: spleen, liver, and kidney. J Trauma. 1989;29(12):1664-1666. PMID: 2593197

Further Resources

  • Trauma.org: trauma.org
  • EAST Practice Guidelines: east.org


Medical Disclaimer: MedVellum content is for educational purposes and clinical reference. Clinical decisions should account for individual patient circumstances. Always follow local trauma protocols and guidelines.

13. Examination Focus

The "Trauma Call" Scenario

1. Primary Survey (Abdomen)

  • Look: "Seatbelt sign" (strip the patient). Distension. Evisceration. Stab wounds (log roll to check back).
  • Feel: Rigid? Guarding? (Signs of peritonitis = Laparotomy).
  • Listen: Useless in trauma bay (too noisy).

2. The eFAST Scan (Extended Focused Assessment with Sonography for Trauma)

  • Question: "Is there free fluid?" (Yes/No). NOT "Is the spleen injured?".
  • 4 Views:
    1. RUQ (Morrison's Pouch): Liver/Kidney interface. Most sensitive spot (fluid falls here).
    2. LUQ (Splenorenal): Spleen/Kidney.
    3. Pelvic (Suprapubic): Bladder/Rectum (Pouch of Douglas).
    4. Subxiphoid: Pericardial effusion (Tamponade).
    5. Extended: Lung sliding (Pneumothorax).

3. Interpreting CT Trauma

  • Contrast Phases:
    • Arterial: Active bleeding ("Blush"), Artery dissection.
    • Portal Venous: Parenchymal injury (Lacerations suitable for grading).
    • Delayed: Urine leaks (Ureter/Bladder).

Viva Questions:

  • Q: Define the Lethal Triad.
    • A: Coagulopathy, Acidosis, Hypothermia.
  • Q: What are the indications for immediate Laparotomy in trauma?
    • A: Unstable + Positive FAST/DPL, Peritonitis, Evisceration, GSW (trans-abdominal).
  • Q: What is the significance of a Seatbelt Sign?
    • A: High association with hollow viscus (bowel) injury and mesenteric tears.
  • Q: Explain "Permissive Hypotension".
    • A: Targeting SBP 80-90mmHg to prevent clot disruption ("Popping the clot") while maintaining cerebral perfusion, until hemostasis is achieved. Contraindicated in TBI.

Last updated: 2025-12-22

At a Glance

EvidenceHigh
Last Updated2025-12-22
Emergency Protocol

Red Flags

  • Hemodynamic instability with positive FAST
  • Peritonitis (guarding, rigidity)
  • Evisceration
  • Free air on imaging
  • Gunshot wound to abdomen
  • Seatbelt sign with mechanism

Clinical Pearls

  • **FAST Positive + Unstable = Theatre**: Do not delay for CT. Positive FAST in a hemodynamically unstable patient mandates emergent laparotomy.
  • **Seatbelt Sign**: Ecchymosis across abdomen from seatbelt increases risk of hollow viscus and mesenteric injury. Have high suspicion even if initial imaging appears reassuring.
  • **Delayed Splenic Rupture**: Can occur hours to weeks after injury. Warn patients about warning signs at discharge.
  • Anaerobic respiration -
  • Lactate. Acidosis inhibits thrombin generation.

Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines