Abdominal Trauma
Summary
Abdominal trauma refers to injury to the abdomen from external forces, classified as blunt (MVA, falls, assault) or penetrating (stab wounds, gunshot wounds). It is a leading cause of preventable death in trauma patients. The abdomen is the third most commonly injured region. Blunt trauma typically injures solid organs (spleen, liver, kidney), while penetrating trauma has higher rates of hollow viscus injury. Management ranges from non-operative observation for stable solid organ injuries to emergent laparotomy for hemodynamic instability or peritonitis.
Key Facts
- Incidence: 10% of trauma deaths; abdomen is 3rd most injured region
- Blunt trauma: 80-90% of abdominal trauma cases
- Most commonly injured (blunt): Spleen, followed by liver
- Most commonly injured (penetrating): Small bowel
- Laparotomy rate: GSW approximately 80-90%, stab wounds approximately 25-30%
- Key decision: Hemodynamic stability determines operative vs non-operative management
Clinical Pearls
FAST Positive + Unstable = Theatre: Do not delay for CT. Positive FAST in a hemodynamically unstable patient mandates emergent laparotomy.
Seatbelt Sign: Ecchymosis across abdomen from seatbelt increases risk of hollow viscus and mesenteric injury. Have high suspicion even if initial imaging appears reassuring.
Delayed Splenic Rupture: Can occur hours to weeks after injury. Warn patients about warning signs at discharge.
Why This Matters Clinically
Missed abdominal injury leads to preventable death. Rapid identification using FAST, appropriate resuscitation with damage control principles, and timely operative intervention when indicated saves lives. Non-operative management of solid organ injuries has transformed outcomes in stable patients.
Incidence & Prevalence
- Incidence: Abdomen involved in 10% of trauma deaths
- Blunt vs Penetrating: 80-90% blunt in developed countries
- Trend: Decreasing mortality with improved trauma systems
Demographics
| Factor | Details |
|---|---|
| Age | Peak in young adults (MVA, assault); bimodal with elderly falls |
| Sex | Male predominance (2-3:1) |
| Geography | Urban: higher penetrating; Rural: higher blunt |
Risk Factors
Non-Modifiable:
- Age extremes (poor reserve)
- Anticoagulation use
Modifiable:
| Risk Factor | Relative Risk |
|---|---|
| Alcohol/drug intoxication | 2-3x |
| Non-seatbelt use | 3-4x for serious injury |
| High-risk occupation/activities | Variable |
Mechanism
Blunt Trauma Mechanisms:
Step 1: The Physics of Collision (Kinematics)
- Kinetic Energy: $KE = \frac12mv^2$. Velocity is the key determinant of damage. Doubling speed quadruples energy.
- Cavitation: In solid organs, the energy wave creates a temporary cavity, tearing parenchyma far from the impact site.
- Shearing: Ligamentous attachments (Ligamentum Tetes, Mesentery) hold organs in place while the body decelerates. This tears vessels (avulsion).
Step 2: The "Paper Bag" Effect (Burst Injury)
- Mechanism: Sudden compression of a gas/fluid-filled hollow viscus (stomach, bladder, bowel).
- Result: Sudden spike in intra-luminal pressure > wall tension -> Blow out.
- Lap Belt Injury: Compression of bowel against the lumbar spine ("Chance Fracture" association).
Step 3: The Lethal Triad (Physiological Collapse)
- Hypothermia: Blood loss + exposure + cold fluids. Enzymes (clotting cascade) fail at less than 35°C.
- Acidosis: Hypoperfusion -> Anaerobic respiration -> Lactate. Acidosis inhibits thrombin generation.
- Coagulopathy: Consumption of factors + Dilution + Fibrinolysis (driven by Protein C activation).
- Result: "Bloody Vicious Cycle" -> Irreversible shock.
Step 4: Inflammatory Response
- Tissue Trauma: Releases DAMPs (Damage Associated Molecular Patterns).
- SIRS: Systemic inflammatory response -> Capillary leak -> Pulmonary Edema (ARDS) + Multi-Organ Failure.
Penetrating Trauma Mechanisms:
| Type | Velocity | Injury Pattern |
|---|---|---|
| Stab wound | Low | Direct tissue disruption along tract |
| Gunshot wound | High | Cavitation, blast effect, unpredictable trajectory |
Classification
Anatomical Regions:
| Region | Boundaries | Contents |
|---|---|---|
| Intrathoracic abdomen | Diaphragm to costal margins | Liver, spleen, stomach |
| True abdomen | Costal margins to iliac crests | Bowel, mesentery |
| Retroperitoneum | Behind peritoneum | Kidneys, pancreas, duodenum, great vessels |
| Pelvis | Below pelvic brim | Bladder, rectum, iliac vessels |
AAST Organ Injury Scale (Spleen Example):
| Grade | Description |
|---|---|
| I | Subcapsular hematoma less than 10%; laceration less than 1cm |
| II | Subcapsular hematoma 10-50%; laceration 1-3cm |
| III | Subcapsular hematoma greater than 50% or expanding; laceration greater than 3cm |
| IV | Laceration involving segmental or hilar vessels |
| V | Shattered spleen; hilar vascular injury |
Anatomical Considerations
- Intrathoracic abdomen (liver, spleen) protected by ribs but injured in thoracoabdominal trauma
- Retroperitoneal injuries (pancreas, duodenum, kidneys) may have minimal initial findings
- Hollow viscus injuries present late with peritonitis
Symptoms
Typical Presentation:
Atypical Presentations:
Signs
Red Flags
[!CAUTION] Red Flags — Immediate surgical consultation if:
- Hemodynamic instability with abdominal injury
- Positive FAST with shock
- Peritonitis (guarding, rigidity, rebound)
- Evisceration
- GSW to abdomen
- Free air on imaging
- Impaled object
Structured Approach
General:
- Hemodynamic status (HR, BP, lactate)
- Level of consciousness
- Evidence of shock (pale, clammy, confused)
Abdominal Examination:
- Inspect: wounds, seatbelt sign, distension, evisceration
- Palpate: tenderness, guarding, rigidity
- Percussion: shifting dullness (blood), tympany (free air)
- Auscultate: absent bowel sounds, bowel sounds in chest (diaphragm rupture)
Adjuncts:
- Log roll: inspect back for wounds
- Perineal exam: blood at meatus (urethral injury)
- Rectal exam: high-riding prostate, gross blood
Special Tests
| Test | Technique | Positive Finding | Sensitivity/Specificity |
|---|---|---|---|
| FAST ultrasound | 4 views: RUQ, LUQ, subxiphoid, pelvis | Free fluid in Morrison's/splenorenal/pelvis | 85-95% / 95%+ for hemoperitoneum |
| Log roll | Turn patient to inspect posterior | Wounds, tenderness, deformity | N/A |
| Rectal exam | Digital examination | Blood, high-riding prostate, loss of tone | N/A |
First-Line (Bedside)
- FAST ultrasound — Rapid bedside assessment for free fluid
- Observations — HR, BP, RR, GCS, SpO2
- Urine dipstick — Hematuria suggests GU injury
Laboratory Tests
| Test | Expected Finding | Purpose |
|---|---|---|
| FBC | Hb may be normal initially; later drops | Serial trending for blood loss |
| Coagulation | PT/APTT/INR | Baseline; guide reversal if anticoagulated |
| Group & Save / Crossmatch | Type specific or O-neg | Prepare for transfusion |
| U&Es | Baseline creatinine | Assess renal function |
| Lactate | Elevated = tissue hypoperfusion | Marker of shock severity |
| Base deficit | More negative = worse shock | Prognostic marker |
Imaging
| Modality | Findings | Indication |
|---|---|---|
| FAST | Free fluid (blood) | Unstable trauma; screening |
| CT abdomen/pelvis with IV contrast | Solid organ injury, active extravasation, free air, bowel thickening | Stable patients; gold standard |
| CXR | Diaphragm rupture, pneumoperitoneum | Part of trauma series |
| Pelvic XR | Pelvic fracture | Associated retroperitoneal hemorrhage |
Diagnostic Criteria
- Positive FAST: Free fluid in any view
- Peritonitis: Clinical diagnosis (guarding, rigidity, rebound tenderness)
- DPL Positive: greater than 10mL gross blood, RBC greater than 100,000/mm³, WBC greater than 500/mm³
Management Algorithm
Trauma Call Activation
↓
┌─────────────────────────────────────────────────────┐
│ PRIMARY SURVEY (ABCDE) │
│ - Airway secured? │
│ - Breathing: Decompress Tension Pneumothorax │
│ - Circulation: 2x Large Bore IV. TXA 1g. │
│ - Massive Transfusion Protocol (MTP) if shock │
└─────────────────────────────────────────────────────┘
↓
┌─────────────────────────────────────────────────────┐
│ HEMODYNAMIC ASSESSMENT │
│ (BP, HR, Response to Volume, Lactate) │
├────────────────────┬────────────────────────────────┤
│ UNSTABLE │ STABLE │
│ (Transient/Non-R) │ (Responder, SBP >100) │
├────────────────────┼────────────────────────────────┤
│ ↓ │ ↓ │
│ eFAST Scan │ CT Pan-Scan │
│ (Trauma Bay) │ (Arterial/Portal) │
├───────┬────────────┤ │ │
│ POS │ NEG │ ┌───────┴──────┐ │
│ ↓ │ ↓ │ ↓ ↓ │
│ LAP │ Search for │ Solid Organ Hollow Viscus │
│ NOW │ other │ Injury Revascular │
│ │ bleeding │ ↓ ↓ │
│ │ (Pelvis?) │ Grade? Stable? LAPAROTOMY │
└───────┴─────┬──────┘ ↓ │
↓ ┌───────┴──────┐ │
Stabilize + CT │ NOM │ │
│ (Observation)│ │
└───────┬──────┘ │
↓ │
Contrast Blush? │
↓ │
ANGIOEMBOLIZATION │
Acute/Emergency Management
Damage Control Resuscitation:
- Activate massive transfusion protocol if hemorrhagic shock
- 1:1:1 ratio (pRBC : FFP : Platelets)
- Tranexamic acid 1g IV (within 3 hours of injury)
- Permissive hypotension: target SBP 80-90mmHg (unless TBI)
- Limit crystalloid (worsens coagulopathy)
- Correct hypothermia (target temperature greater than 36°C)
- Maintain ionized calcium greater than 1.0 mmol/L
Conservative Management
Best Medical Therapy (for all patients):
- Pain control
- VTE prophylaxis once hemostasis achieved
- Serial abdominal examinations
- Serial hemoglobin monitoring
Medical Management
| Drug Class | Drug | Dose | Duration |
|---|---|---|---|
| Antifibrinolytic | Tranexamic acid | 1g IV then 1g over 8h | Single course |
| Analgesia | Morphine | Titrate to effect | As needed |
| VTE prophylaxis | Enoxaparin | 40mg SC daily | Once bleeding controlled |
Procedure Spotlight: Damage Control Surgery (DCS)
The Strategy of Survival.
- Concept: Restore physiology, not anatomy. Limit operative time (less than 90 mins) to prevent the Lethal Triad.
- Phases:
- Selection: Decision made early (pH less than 7.2, Temp less than 34, Transfusion greater than 10u).
- Phase 1 (The Operation):
- Control Haemorrhage: Packing 4-quadrants. Ligating bleeders. Shunting vessels.
- Control Contamination: Staple off bowel holes. No anastomoses!
- Closure: Temporary Abdominal Closure (TAC) / Bogota Bag / Vac Pac.
- Phase 2 (ICU): Rewarm, correct acidosis, correct coagulopathy.
- Phase 3 (Re-look): Return to theatre (24-48h later) for definitive repair when stable.
Procedure Spotlight: REBOA
Resuscitative Endovascular Balloon Occlusion of the Aorta.
- Concept: Internal aortic clamp via the femoral artery.
- Indication: Sub-diaphragmatic life-threatening haemorrhage (Abdo/Pelvis) in arrest/peri-arrest.
- Zones:
- Zone 1: Descending thoracic aorta (For abdomen bleed).
- Zone 3: Infrarenal aorta (For pelvic bleed).
- Pros: Increases cerebral/coronary perfusion. Stops distal bleeding.
- Cons: Ischaemia distal to balloon (max time 30-60 mins). Reperfusion injury.
Procedure Spotlight: Diagnostic Peritoneal Lavage (DPL)
The "Lost Art" (Used if FAST/CT unavailable).
- Technique: Catheter enters infra-umbilical. Aspirate.
- Positive: >10ml Gross blood = Positive.
- Lavage: If no blood, infuse 1L saline. Drain. Lab count >100k RBCs = Positive.
Surgical Management
Indications for Emergent Laparotomy:
- Hemodynamic instability with positive FAST
- Peritonitis
- Evisceration
- Impaled object (remove in OR)
- Free air on imaging
- GSW to abdomen (most cases)
- Failed non-operative management
Non-Operative Management (NOM):
| Criteria | Details |
|---|---|
| Eligible | Hemodynamically stable, no peritonitis, solid organ injury |
| Setting | Trauma center with 24/7 OR capability |
| Monitoring | ICU or monitored bed, serial exams Q2-4h, serial Hb Q6h |
| Intervention | Angioembolization if contrast blush on CT |
| Failure | Ongoing transfusion (greater than 4 units/24h), worsening exam |
Disposition
- Admit ICU if: Unstable, high-grade injury (Grade III+), post-operative
- Admit ward if: Low-grade injury (Grade I-II), stable, reliable exam
- Discharge if: Negative workup, stable, clear return precautions
- Follow-up: Repeat imaging in 5-7 days for high-grade splenic/liver injuries; activity restriction 4-6 weeks
Immediate (Minutes-Hours)
| Complication | Incidence | Presentation | Management |
|---|---|---|---|
| Exsanguination | Variable | Hemodynamic collapse | Massive transfusion, emergent surgery |
| Cardiac arrest | Rare | PEA/asystole | Resuscitative thoracotomy consideration |
Early (Days)
Abdominal Compartment Syndrome (ACS)
The Killer in the ICU.
- Definition: Intra-abdominal pressure (IAP) >20 mmHg WITH new organ dysfunction.
- Mechanism:
- Edema (Capillary leak from resuscitation).
- Bleeding (Ongoing).
- Packing (Foreign bodies occupying space).
- Pathology:
- Kidney: Renal vein compression -> Acute Kidney Injury (Anuria).
- Lungs: Diaphragm pushed up -> High ventilatory pressures, Hypoxia.
- Heart: Decreased venous return (IVC compression) -> Hypotension.
- Brain: Increased ICP (failed venous drainage).
- Treatment: MEDICAL EMERGENCY.
- Paralysis/Sedation (relax muscles).
- NG tube decompression.
- Surgical Decompression: Open the abdomen.
Missed Hollow Viscus Injury
- The "Seatbelt" Trap: Bowel gets crushed. Initial CT may be normal.
- Signs: Tachycardia, increasing pain, fever at 24-48h.
- Management: High index of suspicion. Diagnostic Laparoscopy if unsure.
Late (Weeks-Months)
- Delayed splenic rupture: 1-2 weeks post-injury
- Pseudocyst (pancreatic injury): Weeks post-injury
- Post-splenectomy infection (OPSI): Encapsulated organisms; requires vaccination
- Incisional hernia: Post-laparotomy
- Adhesive small bowel obstruction: Months to years post-operatively
Natural History
- Untreated solid organ hemorrhage leads to exsanguination and death
- Untreated hollow viscus injury leads to peritonitis, sepsis, and death
- Delayed recognition increases mortality significantly
Outcomes with Treatment
| Variable | NOM Success | Operative |
|---|---|---|
| Splenic injury (Grade I-II) | greater than 95% | N/A |
| Splenic injury (Grade III-V) | 75-90% (with angioembolization) | Splenectomy mortality 1-5% |
| Hepatic injury | greater than 90% | Variable by injury |
| Hollow viscus injury | N/A | Depends on contamination, delay |
Prognostic Factors
Good Prognosis:
- Early recognition and intervention
- Single organ injury
- Low-grade injury
- Young, fit patient
- Rapid access to trauma center
Poor Prognosis:
- Multi-organ injury
- Delayed presentation
- Elderly or comorbid patient
- Associated head injury
- Coagulopathy (lethal triad)
Key Guidelines
- ATLS 10th Edition (2018) — American College of Surgeons. Standard trauma management principles. ACS ATLS
- Eastern Association for the Surgery of Trauma (EAST) — Practice management guidelines for solid organ injuries. EAST Guidelines
- Western Trauma Association (WTA) — Critical decisions in trauma. WTA
Landmark Trials
PROPPR Trial (2015) — Transfusion ratios in trauma
- 680 patients with severe trauma
- Key finding: 1:1:1 ratio (pRBC:FFP:Platelets) achieved hemostasis faster than 1:1:2
- Clinical Impact: Established 1:1:1 as standard in massive transfusion
CRASH-2 Trial (2010) — Tranexamic acid in trauma
- 20,211 patients
- Key finding: TXA reduced mortality if given within 3 hours (RR 0.85)
- Clinical Impact: TXA now standard in bleeding trauma
NOM for Solid Organ Injury Studies — Multiple observational series
- Demonstrated high success rates (greater than 90%) for non-operative management of stable solid organ injuries
- Clinical Impact: NOM is now standard of care for stable patients
Evidence Strength
| Intervention | Level | Key Evidence |
|---|---|---|
| 1:1:1 transfusion ratio | 1b | PROPPR: No mortality diff at 24h, but significantly less exsanguination death at 3h. |
| Tranexamic acid (within 3h) | 1a | CRASH-2: Reduced all-cause mortality (14.5% vs 16%) with no increase in vascular occlusive events. |
| FAST for blunt trauma | 2a | Cochrane: High specificity (>95%) but variable sensitivity. Good rule-in, bad rule-out. |
| NOM for solid organ injury | 2a | EAST Guidelines: >98% success for Grade I-II Spleen. 80-90% for Grade IV-V. |
| REBOA vs Thoracotomy | 2b | UK-REBOA: Current evidence suggests no survival benefit over standard care (operator dependent). |
What is abdominal trauma?
Abdominal trauma means your belly has been injured, either from a direct blow (like in a car crash or fall) or from something sharp (like a knife or gunshot). Inside your belly are important organs like your liver, spleen, kidneys, and intestines. These can be bruised, torn, or bleeding.
Why does it matter?
Some injuries cause internal bleeding that you cannot see. This can be very dangerous if not found quickly. Other injuries can release infection into your belly (if the intestines are torn). Both situations can be life-threatening.
How is it treated?
- Observation: For smaller injuries to solid organs (liver, spleen, kidney), we often watch you closely in hospital without surgery. We check your blood levels and examine you regularly.
- Blood transfusion: If you are bleeding significantly, we give you blood transfusions.
- Surgery: If bleeding is severe or cannot be controlled, or if your intestines are injured, you will need an operation.
- Special procedures: Sometimes we can stop bleeding with a procedure done through your blood vessels (angioembolization) without a big surgery.
What to expect
- If you had a solid organ injury treated without surgery, you will need to rest and avoid contact sports and strenuous activity for 4-6 weeks
- You may need a follow-up scan to check healing
- Most people recover fully
When to seek help
Return to hospital immediately if you experience:
- Worsening abdominal pain
- Dizziness, fainting, or feeling lightheaded
- Blood in your urine or stool
- Shoulder tip pain (can indicate internal bleeding)
- Fever
- Feeling unwell or "not right"
Primary Guidelines
- American College of Surgeons Committee on Trauma. Advanced Trauma Life Support (ATLS) Student Course Manual. 10th ed. 2018.
- Stassen NA, et al. Selective nonoperative management of blunt splenic injury: an Eastern Association for the Surgery of Trauma practice management guideline. J Trauma Acute Care Surg. 2012;73(5 Suppl 4):S294-300. PMID: 23114485
Key Trials
- Holcomb JB, et al. Transfusion of plasma, platelets, and red blood cells in a 1:1:1 vs a 1:1:2 ratio and mortality in patients with severe trauma (PROPPR). JAMA. 2015;313(5):471-482. PMID: 25647203
- CRASH-2 trial collaborators. Effects of tranexamic acid on death, vascular occlusive events, and blood transfusion in trauma patients with significant haemorrhage (CRASH-2). Lancet. 2010;376(9734):23-32. PMID: 20554319
- Moore EE, et al. Organ injury scaling: spleen, liver, and kidney. J Trauma. 1989;29(12):1664-1666. PMID: 2593197
Further Resources
- Trauma.org: trauma.org
- EAST Practice Guidelines: east.org
Medical Disclaimer: MedVellum content is for educational purposes and clinical reference. Clinical decisions should account for individual patient circumstances. Always follow local trauma protocols and guidelines.
The "Trauma Call" Scenario
1. Primary Survey (Abdomen)
- Look: "Seatbelt sign" (strip the patient). Distension. Evisceration. Stab wounds (log roll to check back).
- Feel: Rigid? Guarding? (Signs of peritonitis = Laparotomy).
- Listen: Useless in trauma bay (too noisy).
2. The eFAST Scan (Extended Focused Assessment with Sonography for Trauma)
- Question: "Is there free fluid?" (Yes/No). NOT "Is the spleen injured?".
- 4 Views:
- RUQ (Morrison's Pouch): Liver/Kidney interface. Most sensitive spot (fluid falls here).
- LUQ (Splenorenal): Spleen/Kidney.
- Pelvic (Suprapubic): Bladder/Rectum (Pouch of Douglas).
- Subxiphoid: Pericardial effusion (Tamponade).
- Extended: Lung sliding (Pneumothorax).
3. Interpreting CT Trauma
- Contrast Phases:
- Arterial: Active bleeding ("Blush"), Artery dissection.
- Portal Venous: Parenchymal injury (Lacerations suitable for grading).
- Delayed: Urine leaks (Ureter/Bladder).
Viva Questions:
- Q: Define the Lethal Triad.
- A: Coagulopathy, Acidosis, Hypothermia.
- Q: What are the indications for immediate Laparotomy in trauma?
- A: Unstable + Positive FAST/DPL, Peritonitis, Evisceration, GSW (trans-abdominal).
- Q: What is the significance of a Seatbelt Sign?
- A: High association with hollow viscus (bowel) injury and mesenteric tears.
- Q: Explain "Permissive Hypotension".
- A: Targeting SBP 80-90mmHg to prevent clot disruption ("Popping the clot") while maintaining cerebral perfusion, until hemostasis is achieved. Contraindicated in TBI.