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Bell's Palsy

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Overview

Bell's Palsy

Quick Reference

Critical Alerts

  • Rule out stroke: Central facial weakness spares forehead (stroke); peripheral involves entire face (Bell's)
  • Eye protection is essential: Prevent corneal damage from incomplete closure
  • Steroids improve outcomes: Start within 72 hours of symptom onset
  • Antivirals controversial: Consider adding if severe
  • Consider Lyme disease in endemic areas: Treat appropriately
  • Ramsay Hunt syndrome (zoster oticus): Vesicles in ear canal—add antivirals

Central vs Peripheral Facial Weakness

FeatureCentral (Stroke)Peripheral (Bell's)
ForeheadSPARED (bilateral innervation)INVOLVED
Lower faceWeakWeak
Eye closureIntactWeak/incomplete
Additional deficitsArm/leg weakness, speechNone

Emergency Treatments

InterventionDetails
SteroidsPrednisone 60-80 mg/day × 7 days (taper optional)
Eye protectionArtificial tears, lubricating ointment, tape/patch at night
Antivirals (optional)Valacyclovir 1g TID × 7 days (consider if severe)
Ramsay HuntValacyclovir + prednisone

Definition

Overview

Bell's palsy is an acute, idiopathic, unilateral peripheral facial nerve (CN VII) paralysis. It is the most common cause of acute facial paralysis, affecting all age groups. While most patients recover fully, early treatment with corticosteroids improves outcomes. The key ED task is distinguishing peripheral (Bell's) from central (stroke) facial weakness.

Classification

By Etiology:

TypeCause
Idiopathic (Bell's palsy)Most common
Ramsay Hunt syndromeHerpes zoster reactivation (VZV)
Lyme diseaseBorrelia burgdorferi (endemic areas)
Otitis media/MastoiditisExtension of infection
TraumaTemporal bone fracture
TumorParotid, cerebellopontine angle
Guillain-Barré syndromeBilateral facial weakness

Epidemiology

  • Incidence: 20-30 per 100,000/year
  • Peak age: 15-45 years
  • Equal gender: Slightly higher in pregnancy (3rd trimester)
  • Recurrence: 5-15%
  • Most recover: 80-90% complete recovery

Etiology

Proposed Mechanism:

  • Herpes simplex virus (HSV-1) reactivation most commonly implicated
  • Inflammation and edema of CN VII in facial canal
  • Compression leads to demyelination and axonal damage

Risk Factors:

FactorNotes
DiabetesHigher incidence
Pregnancy3rd trimester, postpartum
ImmunocompromiseHSV, VZV reactivation
Recent viral URIPreceding illness common

Pathophysiology

Mechanism

  1. HSV-1 reactivation (or other trigger): Viral involvement of geniculate ganglion
  2. Inflammation: Edema of facial nerve within bony canal
  3. Compression: Facial canal is rigid
  4. Ischemia: Compromised vascular supply
  5. Demyelination/Axonal damage: Degree determines recovery

Anatomy of Facial Nerve (CN VII)

  • Motor: Facial expression muscles
  • Parasympathetic: Salivation (submandibular, sublingual), lacrimation
  • Sensory: Taste anterior 2/3 tongue, sensation external ear

Clinical Presentation

Symptoms

SymptomDescription
Acute onset facial weaknessOver hours to 1-3 days
UnilateralAlmost always
Forehead weaknessInability to raise eyebrow
Eye closure weaknessIncomplete (Bell's phenomenon: eye rolls up)
Mouth droopInability to smile, drooling
Ear pain (retroauricular)Prodrome or concurrent
HyperacusisStapedius weakness → Sensitivity to loud sounds
Taste disturbanceAnterior 2/3 tongue
Dry eyeDecreased lacrimation

History

Key Questions:

Physical Examination

Facial Nerve Assessment:

FunctionTest
ForeheadRaise eyebrows
EyeClose eyes tightly
MouthSmile, puff cheeks, purse lips
Bell's phenomenonEye rolls upward with attempted closure (normal protective reflex)
Taste(Not typically tested in ED)
Lacrimation(Not typically tested in ED)

Central vs Peripheral:

FeatureCentral (UMN)Peripheral (LMN/Bell's)
ForeheadSparedAffected
Eye closureIntactAffected
Lower faceWeakWeak
Other deficitsArm/leg weakness, dysarthriaNone

Other Exam:

FindingSignificance
Vesicles in ear canal or TMRamsay Hunt syndrome (VZV)
Parotid massTumor
Other cranial nerve deficitsStroke, tumor, GBS
Mastoid tendernessMastoiditis

Time of onset (sudden vs gradual)
Common presentation.
Associated symptoms (ear pain, vesicles, hearing loss)
Common presentation.
Weakness elsewhere (arms, legs, speech)
Common presentation.
Recent tick bite or travel (Lyme)
Common presentation.
Diabetes or pregnancy
Common presentation.
Prior episodes of facial weakness
Common presentation.
Immunocompromise
Common presentation.
Red Flags

Must Exclude Serious Causes

FindingConcernAction
Forehead sparingCentral lesion (stroke)Stroke workup
Arm/leg weaknessStrokeEmergent CT/MRI
Bilateral facial weaknessGBS, Lyme, tumorWorkup
Gradual onset (> weeks)TumorMRI
No improvement by 3-6 monthsTumor, incomplete recoveryMRI, ENT/Neurology
Vesicles in earRamsay HuntAdd antivirals
Multiple cranial nerve deficitsBrainstem lesion, carcinomatous meningitisMRI

Differential Diagnosis

Other Causes of Facial Weakness

DiagnosisFeatures
StrokeForehead spared, other deficits
Ramsay Hunt syndromeVZV vesicles in ear, hearing loss
Lyme diseaseEndemic area, tick bite, bilateral possible
Otitis media/MastoiditisEar pain, fever, TM abnormality
Parotid tumorGradual, mass palpable
Acoustic neuromaHearing loss, CN VIII involvement
Guillain-Barré syndromeAscending weakness, bilateral facial
SarcoidosisBilateral, systemic symptoms
TraumaTemporal bone fracture

Diagnostic Approach

Clinical Diagnosis

  • Bell's palsy is a clinical diagnosis of exclusion
  • Must differentiate from central causes (stroke)

Imaging

Not Routinely Required for Typical Bell's Palsy

Consider MRI if:

  • Atypical presentation
  • No improvement by 3-4 weeks
  • Recurrent episodes
  • Gradual onset
  • Other neurological deficits

CT Head (If stroke suspected):

  • Forehead sparing
  • Other focal deficits

Laboratory Studies

Not Routinely Needed

Consider:

TestIndication
Lyme serologyEndemic area, bilateral, or rash/tick bite
GlucoseDiabetes screening
HIVIf immunocompromise suspected
ESR, ACE levelIf sarcoidosis suspected

Electrodiagnostic Testing

  • Electroneuronography (ENoG) or EMG
  • Used to assess prognosis
  • Not performed in ED; refer if needed

Treatment

Principles

  1. Steroids within 72 hours: Improve recovery
  2. Eye protection: Prevent corneal damage
  3. Antivirals: Controversial; consider if severe
  4. Rule out other causes: Stroke, Lyme, VZV
  5. Reassurance: Most recover fully

Corticosteroids

Prednisone:

DoseDuration
60-80 mg/day7 days (can taper or stop abruptly)

Alternative: Methylprednisolone equivalent

Evidence: Strong evidence that steroids improve complete recovery rates

Contraindications: Relative (uncontrolled diabetes, active infection)—benefits usually outweigh risks

Antiviral Therapy

Controversial:

  • Studies show mixed results
  • May add modest benefit when combined with steroids
  • Consider for severe cases (House-Brackmann grade IV-VI)

If Used:

AgentDoseDuration
Valacyclovir1 g TID7 days
Acyclovir400 mg 5× daily7-10 days

Ramsay Hunt Syndrome (Herpes Zoster Oticus)

Always treat with antivirals + steroids:

AgentDose
Valacyclovir1 g TID × 7 days
+ Prednisone60-80 mg/day × 7 days

Prognosis is worse than Bell's palsy

Eye Care

Essential to Prevent Corneal Damage:

InterventionDetails
Artificial tearsq1-2h during day
Lubricating ointmentAt night
Eye patch/tapeAt night to close lid
Moisture chamber or gogglesAlternative

If incomplete eye closure: Ophthalmology referral

Lyme Disease-Associated Facial Palsy

If Lyme suspected or confirmed:

AgentDoseDuration
Doxycycline100 mg BID14-21 days
OR Amoxicillin500 mg TID14-21 days

Steroids not recommended for Lyme-associated facial palsy


Disposition

Discharge Criteria

  • Typical Bell's palsy presentation
  • No signs of stroke or other serious cause
  • Eye protection education
  • Steroids prescribed
  • Follow-up arranged

Referral

IndicationReferral
Incomplete eye closureOphthalmology
No improvement in 3-4 weeksNeurology
Atypical featuresNeurology, ENT
Ramsay HuntENT

Follow-Up

SituationFollow-Up
Typical Bell'sPCP or Neurology in 2-4 weeks
Severe or Ramsay HuntNeurology/ENT in 1-2 weeks
Eye involvementOphthalmology

Patient Education

Condition Explanation

  • "You have Bell's palsy, which is a temporary paralysis of the facial nerve."
  • "We don't know exactly what causes it, but it may be related to a viral infection."
  • "Most people recover completely within a few weeks to months."
  • "Protecting your eye is very important since you can't blink properly."

Home Care

  • Use artificial tears and ointment as directed
  • Tape or patch eye closed at night
  • Wear sunglasses outside
  • Massage facial muscles gently
  • Continue steroids as prescribed

Warning Signs to Return

  • Weakness in arm or leg
  • Difficulty speaking or understanding
  • Severe headache
  • Vision changes
  • Eye pain or redness
  • Weakness not improving after 3-4 weeks

Special Populations

Pregnancy

  • Higher incidence (3rd trimester, postpartum)
  • Steroids are generally safe
  • Avoid high-dose steroids near delivery if possible
  • Eye care is essential

Diabetes

  • Higher incidence
  • Steroids may worsen glucose control
  • Benefits usually outweigh risks—monitor glucose
  • May have worse prognosis

Children

  • Less common
  • Always consider Lyme disease
  • Similar treatment (weight-based steroids)
  • Good prognosis

Quality Metrics

Performance Indicators

MetricTargetRationale
Forehead weakness documented100%Differentiate central vs peripheral
Steroids started within 72h onset>0%Improves recovery
Eye protection education100%Prevent corneal damage
Stroke excluded if forehead spared100%Life-threatening mimic

Documentation Requirements

  • Laterality (unilateral)
  • Forehead involvement (confirms peripheral)
  • Eye closure assessment
  • Time of symptom onset
  • Treatment prescribed
  • Eye care education provided
  • Follow-up plan

Key Clinical Pearls

Diagnostic Pearls

  • Forehead involved = Peripheral (Bell's): Forehead spared = Central (Stroke)
  • Acute onset (hours to days): Typical for Bell's
  • Vesicles in ear = Ramsay Hunt: Add antivirals
  • Bilateral = Not Bell's: Think Lyme, GBS, sarcoidosis
  • Gradual onset = Tumor: MRI needed
  • Always check for other deficits: Rule out stroke

Treatment Pearls

  • Steroids work: Start within 72 hours
  • Antivirals controversial: Consider if severe
  • Eye care is critical: Corneal damage is preventable
  • Lyme disease = Antibiotics, not steroids
  • Ramsay Hunt = Both antivirals AND steroids
  • Most recover completely: Reassure patients

Disposition Pearls

  • Most can be discharged: With steroids and eye care
  • Follow-up in 2-4 weeks: For reassessment
  • Ophthalmology if eye at risk: Incomplete closure
  • Neurology if no improvement: By 3-4 weeks
  • MRI if atypical: Gradual onset, recurrent, bilateral

References
  1. Gronseth GS, et al. Practice Parameter: Steroids and antivirals for Bell palsy. Neurology. 2012;79(22):2209-2213.
  2. Sullivan FM, et al. Early treatment with prednisolone or acyclovir in Bell's palsy. N Engl J Med. 2007;357(16):1598-1607.
  3. Gagyor I, et al. Antiviral treatment for Bell's palsy (idiopathic facial paralysis). Cochrane Database Syst Rev. 2019;9:CD001869.
  4. Baugh RF, et al. Clinical Practice Guideline: Bell's Palsy. Otolaryngol Head Neck Surg. 2013;149(3 Suppl):S1-S27.
  5. Peitersen E. Bell's palsy: the spontaneous course of 2,500 peripheral facial nerve palsies of different etiologies. Acta Otolaryngol Suppl. 2002;(549):4-30.
  6. Murakami S, et al. Bell palsy and herpes simplex virus. Ann Intern Med. 1996;124(1 Pt 1):27-30.
  7. American Academy of Neurology. Practice parameter: steroids and antivirals for Bell palsy. 2012.
  8. UpToDate. Bell's palsy: Treatment and prognosis. 2024.

At a Glance

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Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines