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Hyperemesis Gravidarum

High EvidenceUpdated: 2025-12-25

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Red Flags

  • Wernicke Encephalopathy (Thiamine Deficiency - Confusion, Ataxia, Ophthalmoplegia)
  • Severe Electrolyte Disturbance (Hypokalaemia, Hyponatraemia)
  • Acute Kidney Injury (Severe Dehydration)
  • Weight Loss >5% of Pre-Pregnancy Weight
  • Ketosis
Overview

Hyperemesis Gravidarum

1. Clinical Overview

Summary

Hyperemesis Gravidarum (HG) is a severe form of nausea and vomiting in pregnancy (NVP) characterised by intractable vomiting, weight loss (>5% of pre-pregnancy weight), dehydration, and ketonuria. It affects approximately 0.3-2% of pregnancies and is the most common reason for hospitalisation in the first trimester. Unlike typical "morning sickness" (which affects 70-80% of pregnancies and is self-limiting), HG is debilitating and may persist throughout pregnancy. The exact cause is unknown but is linked to rising hCG levels (Peak at 9-12 weeks), which is why it is more severe in multiple pregnancy and molar pregnancy. Untreated, severe HG can lead to Wernicke Encephalopathy (Thiamine deficiency), electrolyte disturbances, and rarely maternal death. Management involves IV fluid resuscitation, thiamine supplementation, and antiemetics (Cyclizine, Ondansetron, Prochlorperazine). The PUQE Score is used to assess severity. Women with HG require compassionate care and may need multiple admissions. [1,2,3]

Clinical Pearls

"More Than Morning Sickness": HG is NOT just bad morning sickness. It is a distinct, severe condition causing >5% weight loss, Ketonuria, Dehydration.

Give Thiamine BEFORE Glucose: In severe HG, Thiamine (Pabrinex) must be given BEFORE IV glucose/dextrose. Glucose without Thiamine can precipitate Wernicke Encephalopathy.

"Think Molar/Twins": Very severe or early-onset HG should prompt USS to exclude molar pregnancy and multiple pregnancy (High hCG drives worse symptoms).

Ondansetron is Safe: Ondansetron 4-8mg is widely used in HG. Large studies (1.8 million pregnancies) show no increased risk of congenital malformation.


2. Epidemiology

Demographics

FactorNotes
PrevalenceHG: 0.3-2% of pregnancies. Milder NVP: 70-80%.
OnsetTypically starts 4-7 weeks gestation. Peaks 9-12 weeks (hCG peak).
DurationUsually improves by 16-20 weeks. 10% persist entire pregnancy.

Risk Factors

Risk FactorNotes
Previous HGStrongest predictor. ~15-20% recurrence risk.
Family HistoryGenetic component.
Multiple PregnancyHigher hCG.
Molar PregnancyVery high hCG. Severe early HG → Exclude molar.
Female FetusSlightly higher incidence.
Thyroid DisordersTransient gestational thyrotoxicosis (hCG cross-reacts with TSH receptor).
ObesitySome studies show increased risk.

3. Pathophysiology

Proposed Mechanisms

MechanismNotes
hCGStrong correlation. hCG peaks at 9-12 weeks = Peak symptom severity. Multiple pregnancy/Molar = Higher hCG = Worse symptoms.
OestrogenContributes to nausea.
ProgesteroneSlows gastric motility.
Helicobacter PyloriAssociation with more severe NVP (Controversial).
Altered Gastrointestinal MotilityDelayed gastric emptying.
Psychological FactorsStress may exacerbate (But NOT the cause).

Consequences of Prolonged Vomiting

ConsequenceNotes
DehydrationReduced oral intake + Vomiting.
Electrolyte DisturbanceHypokalaemia (Vomiting loses K+), Hyponatraemia, Hypochloraemic Metabolic Alkalosis.
KetonuriaStarvation ketosis. Fat breakdown for energy.
Thiamine DeficiencyIf untreated → Wernicke Encephalopathy (Confusion, Ataxia, Ophthalmoplegia). Preventable with Thiamine.
Weight Loss>5% of pre-pregnancy weight = Diagnostic criterion.

4. Differential Diagnosis
ConditionKey Features
Hyperemesis GravidarumOnset 4-12 weeks, Ketonuria, Weight loss, Severe vomiting, Dehydration.
Molar PregnancyVery early severe HG, Vaginal bleeding, Large-for-dates uterus, Snowstorm USS.
Multiple PregnancyLarger uterus, High hCG.
Urinary Tract InfectionDysuria, Frequency, Suprapubic pain, Positive urine culture.
GastroenteritisDiarrhoea, Fever, Sick contacts, Acute onset.
AppendicitisRight iliac fossa pain, Fever, Raised WCC/CRP. Can occur in pregnancy.
Bowel ObstructionColicky abdominal pain, Distension, Constipation, X-ray findings.
Diabetic Ketoacidosis (DKA)Known diabetic, High glucose, Metabolic acidosis (Not alkalosis).
ThyrotoxicosisPalpitations, Tremor, Weight loss, Suppressed TSH, Raised T4.

5. Clinical Presentation

Symptoms

SymptomNotes
Persistent VomitingMultiple times per day. Unable to keep down food/fluids.
NauseaConstant, debilitating.
Weight Loss>5% of pre-pregnancy weight is diagnostic criterion.
FatigueProfound.
Ptyalism (Excessive Salivation)Unable to swallow saliva.
Aversion to Food/SmellsStrong triggers.

Signs

SignNotes
DehydrationDry mucous membranes, Reduced skin turgor, Tachycardia, Hypotension, Concentrated urine.
KetonuriaPositive ketones on urine dipstick (++/+++).
WeightDocument at each visit. Compare to pre-pregnancy weight.
Epigastric TendernessMay have from retching.

PUQE Score (Pregnancy-Unique Quantification of Emesis)

Used to assess severity (Score out of 15):

ScoreSeverity
less than 6Mild
7-12Moderate
≥13Severe

Nausea duration
Common presentation.
Number of vomiting episodes
Common presentation.
Number of retching episodes
Common presentation.
6. Investigations

Bedside

TestFindings / Rationale
Urine DipstickKetones (Starvation), Specific gravity (Dehydration), Exclude UTI.
WeightCompare to pre-pregnancy. >5% loss = HG.
Blood Pressure/PulseTachycardia, Postural hypotension (Dehydration).
PUQE ScoreQuantify severity.

Blood Tests

TestFindings / Rationale
U&EsHypokalaemia, Hyponatraemia, Raised Urea (Dehydration). Metabolic alkalosis (From vomiting HCl).
FBCRaised Hct (Haemoconcentration).
LFTsElevated transaminases in ~50% (Transient).
TFTsSuppressed TSH, Raised T4 in hyperthyroid HG (Due to hCG cross-reactivity with TSH receptor). Usually transient.
Blood GlucoseExclude DKA.

Imaging

ImagingIndication
Pelvic USSConfirm viable intrauterine pregnancy. Exclude molar pregnancy, Multiple pregnancy.

7. Management

Management Algorithm

       HYPEREMESIS GRAVIDARUM
       (Severe NVP + Weight loss >5% + Ketonuria)
                     ↓
       INITIAL ASSESSMENT
       - PUQE Score
       - Weight (Compare to pre-pregnancy)
       - Urine ketones, Dipstick
       - U&Es, LFTs, TFTs
       - USS to confirm viable pregnancy / Exclude molar/twins
                     ↓
       ADMISSION CRITERIA
       - Unable to tolerate oral fluids/medications
       - Ketonuria ≥2+
       - Weight loss >5%
       - Abnormal electrolytes
       - Unable to manage at home
                     ↓
       INPATIENT MANAGEMENT
    ┌──────────────────────────────────────────────────────────┐
    │  IV FLUIDS:                                              │
    │  - Normal Saline (0.9% NaCl) with KCl as required        │
    │  - Avoid Dextrose initially (Risk of Wernicke if         │
    │    Thiamine deficient)                                   │
    │                                                          │
    │  THIAMINE (Pabrinex):                                    │
    │  - Give Pabrinex IV (1 pair vials) BEFORE any dextrose   │
    │  - Continue oral Thiamine 50mg TDS during admission       │
    │  - Prevents Wernicke Encephalopathy                      │
    │                                                          │
    │  ANTIEMETICS (Stepwise):                                 │
    │  First-Line: Cyclizine 50mg IV/IM/PO TDS                 │
    │        OR    Prochlorperazine 12.5mg IM / 5mg PO TDS     │
    │  Second-Line: Ondansetron 4-8mg IV/PO BD-TDS             │
    │  Third-Line: Metoclopramide 10mg IV/PO TDS (Max 5 days)  │
    │  Refractory: Steroids (Hydrocortisone → Prednisolone)    │
    │              - Reserved for severe cases unresponsive    │
    │                to standard antiemetics                   │
    │                                                          │
    │  THROMBOPROPHYLAXIS:                                     │
    │  - TED stockings                                         │
    │  - Consider LMWH if prolonged admission/severe           │
    │    dehydration (HG increases VTE risk)                   │
    └──────────────────────────────────────────────────────────┘
                     ↓
       OUTPATIENT/DISCHARGE
       - Oral antiemetics to take home
       - Oral Thiamine
       - Safety net: Return if unable to keep down fluids
       - Day Unit option for IV fluids if recurrence

Antiemetic Options

DrugDoseNotes
Cyclizine50mg TDS PO/IV/IMFirst-line. Antihistamine. May cause drowsiness.
Prochlorperazine (Stemetil)5-10mg TDS PO or 12.5mg IMDopamine antagonist. Can cause dystonia.
Ondansetron4-8mg BD-TDS PO/IV5-HT3 antagonist. Very effective. Safe in pregnancy (Large studies). May cause constipation.
Metoclopramide10mg TDS PO/IVLimit to 5 days (MHRA – Dystonia risk).
Promethazine25mg PO/IMUseful at night (Sedating).
Steroids (Hydrocortisone/Prednisolone)Specialist useFor refractory HG. Avoid if possible less than 10 weeks (Cleft lip risk).

Non-Pharmacological

InterventionNotes
GingerMay help mild NVP. Limited evidence.
Acupressure (P6 Point)Bands (Sea-Bands). Some evidence.
Dietary AdviceSmall frequent meals, Avoid triggers, Dry crackers before rising.

8. Complications

Maternal

ComplicationNotes
Wernicke EncephalopathyPREVENTABLE. Thiamine deficiency → Confusion, Ataxia, Ophthalmoplegia. Give Thiamine BEFORE glucose.
Mallory-Weiss TearOesophageal mucosal tear from retching → Haematemesis.
Electrolyte DisturbanceHypokalaemia → Arrhythmias. Hyponatraemia → Confusion.
Acute Kidney InjurySevere dehydration.
Venous ThromboembolismDehydration + Pregnancy → Increased VTE risk.
Psychological MorbidityDepression, Anxiety, PTSD. HG significantly impacts mental health. May affect bonding.
Termination of PregnancySome women request TOP due to severity.

Fetal

ComplicationNotes
Low Birth WeightAssociated with severe prolonged HG with >5% weight loss.
Preterm BirthSlightly increased risk.
SGASome studies.

9. Prognosis and Outcomes
FactorNotes
Typical CourseSymptoms usually improve by 16-20 weeks (After hCG falls). 10% persist throughout pregnancy.
Recurrence15-20% recurrence risk in future pregnancies.
Long-TermNo long-term maternal effects. Fetus usually unaffected if adequately treated.
Psychological ImpactHigh rates of depression, Anxiety, PTSD. Support essential.

10. Evidence and Guidelines

Key Guidelines

GuidelineOrganisationKey Recommendations
Management of Nausea and Vomiting in Pregnancy and Hyperemesis GravidarumRCOG GTG 69 (2016)Thiamine before dextrose, Antiemetic ladder, VTE prophylaxis.
HG Care PathwayNICE QSAmbulatory care options, Day units.

Key Points

  • Ondansetron Safety: Large cohort studies (>1.8 million pregnancies) show no significant increase in congenital malformations.
  • Steroids: Reserved for refractory cases. Some evidence of benefit. Risk of cleft lip if used less than 10 weeks.

11. Patient and Layperson Explanation

What is Hyperemesis Gravidarum?

It is a severe form of pregnancy sickness – much worse than ordinary "morning sickness." It causes persistent vomiting that leads to dehydration, weight loss, and difficulty keeping down food or fluids.

Is it just bad morning sickness?

No. Morning sickness is usually manageable and settles by 12-14 weeks. Hyperemesis is debilitating – you may be unable to work, care for yourself, or even keep water down. It often requires hospital treatment.

What causes it?

We don't fully understand the cause, but it is linked to pregnancy hormones (hCG), which peak around 9-12 weeks. It is more common in twin pregnancies (Higher hCG).

Is my baby okay?

Yes, in most cases. If you receive treatment (Fluids, Vitamins, Anti-sickness medication), the baby is usually unaffected.

What is the treatment?

  • Fluids through a drip (If you cannot keep fluids down).
  • Anti-sickness tablets or injections (Safe in pregnancy).
  • Vitamin B1 (Thiamine) – Very important to prevent a rare but serious complication.

Will it happen again?

There is about a 15-20% chance of it recurring in a future pregnancy. But knowing this means we can sometimes prevent it with early anti-sickness medication before symptoms start.


12. References

Primary Sources

  1. Royal College of Obstetricians and Gynaecologists. The Management of Nausea and Vomiting of Pregnancy and Hyperemesis Gravidarum (GTG 69). 2016.
  2. Huybrechts KF, et al. Association of Maternal First-Trimester Ondansetron Use With Cardiac Malformations and Oral Clefts in Offspring. JAMA. 2018;320(23):2429-2437. PMID: 30561481.
  3. London V, et al. Hyperemesis Gravidarum: A Review of Recent Literature. Pharmacology. 2017;100(3-4):161-171. PMID: 28614831.

13. Examination Focus

Common Exam Questions

  1. Diagnostic Criteria: "What defines HG vs Normal NVP?"
    • Answer: Weight loss >5% pre-pregnancy weight + Ketonuria + Dehydration.
  2. Thiamine: "Why must Thiamine be given before IV glucose?"
    • Answer: Glucose metabolism consumes Thiamine. In deficient patients, giving glucose without Thiamine precipitates Wernicke Encephalopathy.
  3. Investigation to Exclude: "What must be excluded in very severe/early HG?"
    • Answer: Molar Pregnancy (Very high hCG) and Multiple Pregnancy.
  4. First-Line Antiemetic: "What is first-line antiemetic in HG?"
    • Answer: Cyclizine 50mg TDS (Or Prochlorperazine).

Viva Points

  • Wernicke Triad: Confusion, Ataxia, Ophthalmoplegia. Often incomplete. IRREVERSIBLE if severe.
  • PUQE Score: Pregnancy-Unique Quantification of Emesis. Quantifies severity.
  • VTE Risk: Dehydration + Pregnancy = High VTE risk. Consider TED stockings and LMWH.
  • Ondansetron is Safe: Large studies show no increased teratogenicity.

Medical Disclaimer: MedVellum content is for educational purposes and clinical reference. Clinical decisions should account for individual patient circumstances. Always consult appropriate specialists.

Last updated: 2025-12-25

At a Glance

EvidenceHigh
Last Updated2025-12-25

Red Flags

  • Wernicke Encephalopathy (Thiamine Deficiency - Confusion, Ataxia, Ophthalmoplegia)
  • Severe Electrolyte Disturbance (Hypokalaemia, Hyponatraemia)
  • Acute Kidney Injury (Severe Dehydration)
  • Weight Loss >5% of Pre-Pregnancy Weight
  • Ketosis

Clinical Pearls

  • **"More Than Morning Sickness"**: HG is NOT just bad morning sickness. It is a distinct, severe condition causing >5% weight loss, Ketonuria, Dehydration.
  • **Give Thiamine BEFORE Glucose**: In severe HG, Thiamine (Pabrinex) must be given BEFORE IV glucose/dextrose. Glucose without Thiamine can precipitate Wernicke Encephalopathy.
  • **"Think Molar/Twins"**: Very severe or early-onset HG should prompt USS to exclude molar pregnancy and multiple pregnancy (High hCG drives worse symptoms).
  • **Ondansetron is Safe**: Ondansetron 4-8mg is widely used in HG. Large studies (1.8 million pregnancies) show no increased risk of congenital malformation.

Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines