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Gastroenterology
Emergency Medicine
General Surgery
EMERGENCY

Mallory-Weiss Tear

High EvidenceUpdated: 2025-12-22

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Red Flags

  • Boerhaave syndrome (full thickness rupture - surgical emergency)
  • Haemodynamic instability (massive haematemesis)
  • Chest pain with subcutaneous emphysema (perforation)
Overview

Mallory-Weiss Tear

1. Clinical Overview

Summary

A Mallory-Weiss tear is a longitudinal mucosal laceration at or near the gastro-oesophageal junction (GOJ), caused by a sudden rise in intra-abdominal pressure, classically from forceful vomiting or retching. It typically presents with haematemesis following an episode of non-bloody vomiting or retching. The tear is mucosal/submucosal (NOT full thickness). It accounts for 5-15% of acute upper GI bleeds. Most tears stop bleeding spontaneously (90%), but endoscopic intervention may be required for ongoing haemorrhage. Prognosis is excellent. Mallory-Weiss must be distinguished from Boerhaave syndrome, which is a full-thickness oesophageal rupture and a surgical emergency.

Key Facts

  • Location: Gastro-oesophageal junction (GOJ); may extend into gastric cardia
  • Cause: Forceful vomiting, retching, coughing, straining
  • Depth: Mucosal/submucosal (partial thickness)
  • Classic History: Repeated retching → then haematemesis
  • Diagnosis: Upper GI endoscopy (OGD)
  • Treatment: 90% stop spontaneously; Endoscopic therapy if persistent
  • Key Differential: Boerhaave syndrome (full thickness perforation)

Clinical Pearls

"Retching First, Blood Later": The classic history is initial non-bloody vomiting followed by haematemesis. This distinguishes Mallory-Weiss from variceal or ulcer bleeding.

"Mallory-Weiss Bleeds, Boerhaave Perforates": Mallory-Weiss is a mucosal tear (bleeding). Boerhaave is a full-thickness rupture (chest pain, mediastinitis, surgical emergency). Know the difference.

"Alcohol is the Classic Precipitant": Binge drinking followed by prolonged vomiting is the classic scenario. But any forceful vomiting can cause it.

"Most Stop Spontaneously": 90% of Mallory-Weiss tears stop bleeding without intervention. Only 10% need endoscopic or surgical treatment.


2. Epidemiology

Incidence

  • 5-15% of acute upper GI bleeds
  • More common in young-middle aged adults

Demographics

  • Peak age: 30-50 years
  • M:F = 2-3:1
  • Associated with alcohol use

Risk Factors/Precipitants

FactorNotes
Alcohol intoxicationMost common precipitant
Forceful vomitingAny cause
Prolonged retchingBulimia, pregnancy (hyperemesis)
Violent coughingPertussis, COPD exacerbation
StrainingChildbirth, heavy lifting
Hiatus herniaIncreases risk (found in 35-100% of cases)
AnticoagulationMay increase bleeding severity

3. Pathophysiology

Anatomy

  • Tear occurs at or just below the gastro-oesophageal junction
  • Usually on the lesser curve/cardia of stomach (85%)
  • May extend into distal oesophagus

Mechanism

  1. Sudden rise in intra-abdominal pressure (retching, vomiting, coughing)
  2. Transmitted to gastric fundus
  3. Rapid distension of GOJ
  4. Shear stress exceeds mucosal tensile strength
  5. Longitudinal mucosal laceration
  6. Submucosal arterial bleeding

Depth of Injury

EntityDepthOutcome
Mallory-Weiss tearMucosa/submucosaBleeding
Boerhaave syndromeFull thicknessPerforation → Mediastinitis

Association with Hiatus Hernia

  • Hiatus hernia present in 35-100% of cases
  • Herniated gastric pouch may increase shear stress at GOJ

4. Clinical Presentation

Classic History

  1. Prodrome: Repeated episodes of non-bloody vomiting or retching
  2. Then: Fresh haematemesis (bright red blood)
  3. Context: Often alcohol-related; may follow food poisoning, gastroenteritis

Symptoms

FeatureNotes
HaematemesisBright red blood (after initial non-bloody vomiting)
Retching historyKey history; ask specifically
Epigastric painMild
MelaenaIf significant blood loss
Dizziness/syncopeIf haemodynamically significant

Red Flags (Consider Boerhaave)

FeatureSuggests Boerhaave
Severe chest/back painFull thickness rupture
Subcutaneous emphysemaAir leaking into tissues
Mackler's triadVomiting + Chest pain + Subcutaneous emphysema
Hamman's signMediastinal crunch
Sepsis/shockMediastinitis

5. Clinical Examination

General

  • May appear well if self-limiting
  • Signs of hypovolaemia if significant bleed (tachycardia, hypotension, pallor)

Abdominal

  • Mild epigastric tenderness
  • No peritonism (unless perforation)

Look for

  • Signs of chronic liver disease (if alcohol-related)
  • Subcutaneous emphysema (neck/chest) - suggests Boerhaave

Risk Stratification

  • Use Glasgow-Blatchford Score or Rockall Score
  • Assess haemodynamic status

6. Investigations

First-Line

TestPurpose
FBCHb (blood loss), Platelets
U&EUrea raised (digested blood), Renal function
CoagulationINR, APTT (especially if anticoagulated)
Group & Save / CrossmatchIn case transfusion needed
LFTsLiver function (alcohol, varices)

Diagnostic

  • Upper GI Endoscopy (OGD): Gold standard; visualises tear, allows treatment
  • Usually shows single longitudinal tear at GOJ/cardia

Imaging (If Boerhaave Suspected)

  • Chest X-ray: Pneumomediastinum, pleural effusion
  • CT Chest with Contrast: Oesophageal perforation, mediastinal air/fluid

7. Management

Initial Resuscitation

┌──────────────────────────────────────────────────────────┐
│   MALLORY-WEISS TEAR - MANAGEMENT                        │
├──────────────────────────────────────────────────────────┤
│                                                          │
│  RESUSCITATION:                                           │
│  • IV access (large bore)                                │
│  • IV fluids                                             │
│  • Crossmatch blood if significant bleed                 │
│  • Correct coagulopathy if present                       │
│  • PPI (e.g. Omeprazole 40mg IV)                         │
│  • Antiemetics                                           │
│                                                          │
│  ENDOSCOPY (OGD):                                         │
│  • Timing: Urgent if haemodynamically unstable           │
│  • Most cases: Within 24 hours                           │
│  • Findings: Longitudinal mucosal tear at GOJ            │
│                                                          │
│  ENDOSCOPIC THERAPY (if active bleeding):                 │
│  • Adrenaline injection (1:10,000)                       │
│  • Haemoclips                                            │
│  • Thermal coagulation                                   │
│  • Band ligation (less common)                           │
│                                                          │
│  MOST (90%) STOP SPONTANEOUSLY:                           │
│  • Supportive care only                                  │
│  • PPI                                                   │
│  • Antiemetics                                           │
│                                                          │
│  SURGERY (rare):                                          │
│  • If endoscopic haemostasis fails                       │
│  • Oversewing of tear                                    │
│                                                          │
└──────────────────────────────────────────────────────────┘

Post-Procedure

  • Continue PPI
  • Antiemetics to prevent re-tearing
  • Avoid alcohol
  • Treat underlying cause (e.g., gastroenteritis, alcohol withdrawal)

8. Complications

Immediate

  • Significant haemorrhage (10% need intervention)
  • Hypovolaemic shock (rare)
  • Aspiration

Late

  • Recurrent tear (if ongoing vomiting)
  • Rare: Perforation (if tear deepens)

Mortality

  • Very low (<5%) unless severe comorbidity or massive bleed

9. Prognosis & Outcomes

Natural History

  • 90% of tears stop bleeding spontaneously
  • Healing occurs within 48-72 hours

With Treatment

  • Endoscopic therapy: >95% success rate
  • Surgical intervention: Rarely needed (<5%)

Recurrence

  • Uncommon unless precipitating cause persists (e.g., ongoing vomiting)

Factors Affecting Outcome

GoodPoor
Young ageCoagulopathy
Self-limiting bleedAnticoagulation
No comorbidityLiver disease

10. Evidence & Guidelines

Key Guidelines

  1. BSG Guidelines: Management of Acute Upper GI Bleeding
  2. NICE NG141: Acute Upper GI Bleeding

Key Evidence

Natural History

  • Studies show 80-90% spontaneous haemostasis
  • Endoscopic therapy effective for ongoing bleed

Endoscopic Therapy

  • Haemoclips and injection are effective
  • Combination therapy may be superior to monotherapy

11. Patient/Layperson Explanation

What is a Mallory-Weiss Tear?

A Mallory-Weiss tear is a small tear in the lining of the food pipe (oesophagus) just where it joins the stomach. It happens when you vomit or retch very forcefully, which puts pressure on this area and causes it to tear slightly.

What Are the Symptoms?

  • Vomiting blood (bright red) after repeated retching
  • Usually starts with normal vomiting, then blood appears
  • Sometimes mild pain in the upper tummy

Is it Serious?

Most of the time, no. About 9 out of 10 people stop bleeding on their own without needing any specific treatment. Occasionally, a small procedure is needed to stop the bleeding.

A more serious condition called Boerhaave syndrome can also happen if the tear goes all the way through - but this is rare and would cause severe chest pain.

How is it Diagnosed and Treated?

  • Diagnosis: A camera test (endoscopy) looks at the tear
  • Treatment: Usually just supportive care (fluids, anti-sickness medication)
  • If still bleeding, clips or injections can be used through the camera

How Long to Recover?

The tear usually heals within a couple of days. You'll be advised to avoid alcohol and take medication to reduce stomach acid.


12. References

Primary Guidelines

  1. NICE Guideline [NG141]. Acute Upper Gastrointestinal Bleeding in Over 16s: Management. 2016, updated 2020.
  2. British Society of Gastroenterology. UK Guidelines on the Management of Variceal Haemorrhage. Gut. 2015.

Key Studies

  1. Kortas DY, et al. Mallory-Weiss tear: predisposing factors and predictors of a complicated course. Am J Gastroenterol. 2001;96(10):2863-2865. PMID: 11693319
  2. Younes Z, Johnson DA. The spectrum of spontaneous and iatrogenic esophageal injury: perforations, Mallory-Weiss tears, and hematomas. J Clin Gastroenterol. 1999;29(4):306-317. PMID: 10599628

Last updated: 2025-12-22

At a Glance

EvidenceHigh
Last Updated2025-12-22
Emergency Protocol

Red Flags

  • Boerhaave syndrome (full thickness rupture - surgical emergency)
  • Haemodynamic instability (massive haematemesis)
  • Chest pain with subcutaneous emphysema (perforation)

Clinical Pearls

  • **"Retching First, Blood Later"**: The classic history is initial non-bloody vomiting followed by haematemesis. This distinguishes Mallory-Weiss from variceal or ulcer bleeding.
  • **"Alcohol is the Classic Precipitant"**: Binge drinking followed by prolonged vomiting is the classic scenario. But any forceful vomiting can cause it.
  • **"Most Stop Spontaneously"**: 90% of Mallory-Weiss tears stop bleeding without intervention. Only 10% need endoscopic or surgical treatment.

Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines